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Sleep deprivation impairs memory, tau metabolism, and synaptic integrity of a mouse model of Alzheimer's disease with plaques and tangles

Neurobiology of aging, 2014-08, Vol.35 (8), p.1813-1820

2014

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Early detection and personalized medicine: Future strategies against Alzheimer's disease

Progress in molecular biology and translational science, 2021-01, Vol.177, p.157-173

2021

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Early‐life exposure to high‐fat diet influences brain health in aging mice

Aging cell, 2019-12, Vol.18 (6), p.e13040-n/a

2019

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Impaired mitochondrial calcium efflux contributes to disease progression in models of Alzheimer's disease

Nature communications, 2019-08, Vol.10 (1), p.3885-14, Article 3885

2019

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Elevated levels of brain homocysteine directly modulate the pathological phenotype of a mouse model of tauopathy

Molecular psychiatry, 2019-11, Vol.24 (11), p.1696-1706

2019

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Gestational high fat diet protects 3xTg offspring from memory impairments, synaptic dysfunction, and brain pathology

Molecular psychiatry, 2021-11, Vol.26 (11), p.7006-7019

2021

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Dissecting the Role of 5-Lipoxygenase in the Homocysteine-Induced Alzheimer's Disease Pathology

Journal of Alzheimer's disease, 2018-01, Vol.62 (3), p.1337-1344

2018

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The gut microbiome regulates astrocyte reaction to Aβ amyloidosis through microglial dependent and independent mechanisms

Molecular neurodegeneration, 2023-07, Vol.18 (1), p.45-45, Article 45

2023

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Modulation of AD neuropathology and memory impairments by the isoprostane F2α is mediated by the thromboxane receptor

Neurobiology of aging, 2015-02, Vol.36 (2), p.812-820

2015

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Poloxamer-188 Exacerbates Brain Amyloidosis, Presynaptic Dystrophies, and Pathogenic Microglial Activation in 5XFAD Mice

Current Alzheimer research, 2022-01, Vol.19 (4), p.317-329

2022

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Autophagy Dysfunction in Alzheimer’s Disease: Mechanistic Insights and New Therapeutic Opportunities

Biological psychiatry (1969), 2020-05, Vol.87 (9), p.797-807

2020

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GESTATIONAL HIGH SUGAR DIET PROMOTES ALZHEIMER’S DISEASE PHENOTYPE, TRIGGERS METABOLIC DYSFUNCTION AND SHORTENS SURVIVAL IN 3XTG OFFSPRING LATER IN LIFE

Alzheimer's & dementia, 2018-07, Vol.14 (7), p.P308-P308

2018

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P1‐097: GESTATIONAL HIGH SUGAR DIET PROMOTES ALZHEIMER'S DISEASE PHENOTYPE, TRIGGERS METABOLIC DYSFUNCTION AND SHORTENS SURVIVAL IN 3XTG OFFSPRING LATER IN LIFE

Alzheimer's & dementia, 2018-07, Vol.14 (7S_Part_5)

2018

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12/15-Lipoxygenase Inhibition Reverses Cognitive Impairment, Brain Amyloidosis, and Tau Pathology by Stimulating Autophagy in Aged Triple Transgenic Mice

Biological psychiatry (1969), 2017-01, Vol.81 (2), p.92-100

2017

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Zileuton restores memory impairments and reverses amyloid and tau pathology in aged Alzheimer's disease mice

Neurobiology of aging, 2014-11, Vol.35 (11), p.2458-2464

2014

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MicroRNAs as Therapeutic Targets for Alzheimer's Disease

Journal of Alzheimer's disease, 2016-07, Vol.53 (2), p.367-372

2016

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The gut microbiome regulates astrocyte reaction to A[beta] amyloidosis through microglial dependent and independent mechanisms

Molecular neurodegeneration, 2023-07, Vol.18 (1)

2023

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Elevated levels of brain homocysteine directly modulates the pathological phenotype of a mouse model of tauopathy

Molecular psychiatry, 2018-05, Vol.24 (11), p.1696-1706

2018

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High cholesterol diet during pregnancy attenuates amyloid pathology, cognitive deficit, and synaptic dysfunction in the offspring of 3xTg Alzheimer's disease mice

Alzheimer's & dementia, 2015-07, Vol.11 (7), p.P758-P758

2015

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P3‐316: High cholesterol diet during pregnancy attenuates amyloid pathology, cognitive deficit, and synaptic dysfunction in the offspring of 3xTg Alzheimer's disease mice

Alzheimer's & dementia, 2015-07, Vol.11 (7S_Part_16), p.P758-P758

2015

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12/15-LIPOXYGENASE INHIBITION REVERSES COGNITIVE IMPAIRMENT, AMYLOIDOSIS, TAU AND SYNAPTIC PATHOLOGY BY STIMULATING AUTOPHAGY IN THE BRAIN OF AGED 3XTG MICE

Alzheimer's & dementia, 2016-07, Vol.12 (7), p.P405-P406

2016

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P1‐017: 12/15‐Lipoxygenase Inhibition Reverses Cognitive Impairment, Amyloidosis, TAU and Synaptic Pathology by Stimulating Autophagy in the Brain of Aged 3XTG Mice

Alzheimer's & dementia, 2016-07, Vol.12 (7S_Part_8), p.P405-P406

2016

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P1‐097: GESTATIONAL HIGH SUGAR DIET PROMOTES ALZHEIMER'S DISEASE PHENOTYPE, TRIGGERS METABOLIC DYSFUNCTION AND SHORTENS SURVIVAL IN 3XTG OFFSPRING LATER IN LIFE

Alzheimer's & dementia, 2018-07, Vol.14 (7S_Part_5), p.P308-P308

2018

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Zileuton restores memory impairments and reverses amyloid and tau pathology in aged AD mice

Neurobiology of aging, 2014-05, Vol.35 (11), p.2458-2464

2014

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12/15-Lipoxygenase Inhibition Reverses Cognitive Impairment, Brain Amyloidosis and Tau Pathology by Stimulating Autophagy in Aged 3xTg Mice

Biological psychiatry (1969), 2016

2016

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Sleep deprivation impairs memory, tau metabolism, and synaptic integrity of a mouse model of Alzheimer's disease with plaques and tangles

Early detection and personalized medicine: Future strategies against Alzheimer's disease

Early‐life exposure to high‐fat diet influences brain health in aging mice

Impaired mitochondrial calcium efflux contributes to disease progression in models of Alzheimer's disease

Elevated levels of brain homocysteine directly modulate the pathological phenotype of a mouse model of tauopathy

Gestational high fat diet protects 3xTg offspring from memory impairments, synaptic dysfunction, and brain pathology

Dissecting the Role of 5-Lipoxygenase in the Homocysteine-Induced Alzheimer's Disease Pathology

The gut microbiome regulates astrocyte reaction to Aβ amyloidosis through microglial dependent and independent mechanisms

Modulation of AD neuropathology and memory impairments by the isoprostane F2α is mediated by the thromboxane receptor

Poloxamer-188 Exacerbates Brain Amyloidosis, Presynaptic Dystrophies, and Pathogenic Microglial Activation in 5XFAD Mice

Autophagy Dysfunction in Alzheimer’s Disease: Mechanistic Insights and New Therapeutic Opportunities

GESTATIONAL HIGH SUGAR DIET PROMOTES ALZHEIMER’S DISEASE PHENOTYPE, TRIGGERS METABOLIC DYSFUNCTION AND SHORTENS SURVIVAL IN 3XTG OFFSPRING LATER IN LIFE

P1‐097: GESTATIONAL HIGH SUGAR DIET PROMOTES ALZHEIMER'S DISEASE PHENOTYPE, TRIGGERS METABOLIC DYSFUNCTION AND SHORTENS SURVIVAL IN 3XTG OFFSPRING LATER IN LIFE

12/15-Lipoxygenase Inhibition Reverses Cognitive Impairment, Brain Amyloidosis, and Tau Pathology by Stimulating Autophagy in Aged Triple Transgenic Mice

Zileuton restores memory impairments and reverses amyloid and tau pathology in aged Alzheimer's disease mice

MicroRNAs as Therapeutic Targets for Alzheimer's Disease

The gut microbiome regulates astrocyte reaction to A[beta] amyloidosis through microglial dependent and independent mechanisms

Elevated levels of brain homocysteine directly modulates the pathological phenotype of a mouse model of tauopathy

High cholesterol diet during pregnancy attenuates amyloid pathology, cognitive deficit, and synaptic dysfunction in the offspring of 3xTg Alzheimer's disease mice

P3‐316: High cholesterol diet during pregnancy attenuates amyloid pathology, cognitive deficit, and synaptic dysfunction in the offspring of 3xTg Alzheimer's disease mice

12/15-LIPOXYGENASE INHIBITION REVERSES COGNITIVE IMPAIRMENT, AMYLOIDOSIS, TAU AND SYNAPTIC PATHOLOGY BY STIMULATING AUTOPHAGY IN THE BRAIN OF AGED 3XTG MICE

P1‐017: 12/15‐Lipoxygenase Inhibition Reverses Cognitive Impairment, Amyloidosis, TAU and Synaptic Pathology by Stimulating Autophagy in the Brain of Aged 3XTG Mice

P1‐097: GESTATIONAL HIGH SUGAR DIET PROMOTES ALZHEIMER'S DISEASE PHENOTYPE, TRIGGERS METABOLIC DYSFUNCTION AND SHORTENS SURVIVAL IN 3XTG OFFSPRING LATER IN LIFE

Zileuton restores memory impairments and reverses amyloid and tau pathology in aged AD mice

12/15-Lipoxygenase Inhibition Reverses Cognitive Impairment, Brain Amyloidosis and Tau Pathology by Stimulating Autophagy in Aged 3xTg Mice

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