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Epigenetic Regulation by Z-DNA Silencer Function Controls Cancer-Associated ADAM-12 Expression in Breast Cancer: Cross-talk between MeCP2 and NF1 Transcription Factor Family

Cancer research (Chicago, Ill.), 2013-01, Vol.73 (2), p.736-744

2013

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Z-DNA-forming silencer in the first exon regulates human ADAM-12 gene expression

Proceedings of the National Academy of Sciences - PNAS, 2011-01, Vol.108 (1), p.103-108

2011

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DNA helicases and their roles in cancer

DNA repair, 2020-12, Vol.96, p.102994-102994, Article 102994

2020

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Abstract 2680: RECON Syndrome Helicase is Potently Blocked Upon Encountering a Single Mono-ribonucleotide in the Translocating Strand of a Forked Duplex DNA Substrate

The Journal of biological chemistry, 2023, Vol.299 (3), p.103684, Article 103684

2023

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BLM's balancing act and the involvement of FANCJ in DNA repair

Cell cycle (Georgetown, Tex.), 2018-09, Vol.17 (18), p.2207-2220

2018

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Synthetic Lethal Interactions of RECQ Helicases

Trends in cancer, 2021-02, Vol.7 (2), p.146-161

2021

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Nonenzymatic glycation interferes with fibronectin‐integrin interactions in vascular smooth muscle cells

Microcirculation (New York, N.Y. 1994), 2017-04, Vol.24 (3), p.n/a

2017

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Single-Molecule DNA Fiber Analyses to Characterize Replication Fork Dynamics in Living Cells

Methods in molecular biology (Clifton, N.J.), 2019, Vol.1999, p.307

2019

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NON-ENZYMATIC GLYCATION INTERFERES WITH FIBRONECTIN-INTEGRIN INTERACTIONS IN VASCULAR SMOOTH MUSCLE CELLS

Microcirculation (New York, N.Y. 1994), 2017-04, Vol.24 (3)

2017

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Transforming Growth Factor-β1–Mediated Activation of NF-κB Contributes to Enhanced ADAM-12 Expression in Mammary Carcinoma Cells

Molecular cancer research, 2010-09, Vol.8 (9), p.1261-1270

2010

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RECON syndrome is a genome instability disorder caused by mutations in the DNA helicase RECQL1

The Journal of clinical investigation, 2022-03, Vol.132 (5), p.1-17

2022

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Control of VEGF expression in triple-negative breast carcinoma cells by suppression of SAF-1 transcription factor activity

Molecular cancer research, 2011-08, Vol.9 (8), p.1030-1041

2011

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Abstract 5278: Suppression of angiogenic potential of breast cancer by the inhibition of SAF-1 transcription factor activity

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.5278-5278

2012

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Cellular Assays to Study the Functional Importance of Human DNA Repair Helicases

Methods in molecular biology (Clifton, N.J.), 2019, Vol.1999, p.185

2019

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Abstract 5973: The brain penetrant CDK4/6 Inhibitor, PRT3645, is highly effective in combination with other targeted therapies in preclinical models of NSCLC, CRC, and HER2-positive breast cancer

Cancer research (Chicago, Ill.), 2023-04, Vol.83 (7_Supplement), p.5973-5973

2023

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Increased Adhesion of Glycated Proteins to Arteriolar Vascular Smooth Muscle Cells as Determined by Atomic Force Microscopy

Biophysical journal, 2012-01, Vol.102 (3), p.587a-587a

2012

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Development of an IoT-Based Pipe Water Quality Monitoring and Control System for Smart City

2024 3rd International Conference on Advancement in Electrical and Electronic Engineering (ICAEEE), 2024, p.1-6

2024

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SAF-3, a novel splice variant of the SAF-1/MAZ/Pur-1 family, is expressed during inflammation

The FEBS journal, 2009-08, Vol.276 (15), p.4276-4286

2009

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Transforming growth factor-beta1-mediated activation of NF-kappaB contributes to enhanced ADAM-12 expression in mammary carcinoma cells

Molecular cancer research, 2010-09, Vol.8 (9), p.1261

2010

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Epigenetic Regulation by Z-DNA Silencer Function Controls Cancer-Associated ADAM-12 Expression in Breast Cancer: Cross-talk between MeCP2 and NF1 Transcription Factor Family

Z-DNA-forming silencer in the first exon regulates human ADAM-12 gene expression

DNA helicases and their roles in cancer

Abstract 2680: RECON Syndrome Helicase is Potently Blocked Upon Encountering a Single Mono-ribonucleotide in the Translocating Strand of a Forked Duplex DNA Substrate

BLM's balancing act and the involvement of FANCJ in DNA repair

Synthetic Lethal Interactions of RECQ Helicases

Nonenzymatic glycation interferes with fibronectin‐integrin interactions in vascular smooth muscle cells

Single-Molecule DNA Fiber Analyses to Characterize Replication Fork Dynamics in Living Cells

NON-ENZYMATIC GLYCATION INTERFERES WITH FIBRONECTIN-INTEGRIN INTERACTIONS IN VASCULAR SMOOTH MUSCLE CELLS

Transforming Growth Factor-β1–Mediated Activation of NF-κB Contributes to Enhanced ADAM-12 Expression in Mammary Carcinoma Cells

RECON syndrome is a genome instability disorder caused by mutations in the DNA helicase RECQL1

Control of VEGF expression in triple-negative breast carcinoma cells by suppression of SAF-1 transcription factor activity

Abstract 5278: Suppression of angiogenic potential of breast cancer by the inhibition of SAF-1 transcription factor activity

Cellular Assays to Study the Functional Importance of Human DNA Repair Helicases

Abstract 5973: The brain penetrant CDK4/6 Inhibitor, PRT3645, is highly effective in combination with other targeted therapies in preclinical models of NSCLC, CRC, and HER2-positive breast cancer

Increased Adhesion of Glycated Proteins to Arteriolar Vascular Smooth Muscle Cells as Determined by Atomic Force Microscopy

Development of an IoT-Based Pipe Water Quality Monitoring and Control System for Smart City

SAF-3, a novel splice variant of the SAF-1/MAZ/Pur-1 family, is expressed during inflammation

Transforming growth factor-beta1-mediated activation of NF-kappaB contributes to enhanced ADAM-12 expression in mammary carcinoma cells

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