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Immune Surveillance and Therapy of Lymphomas Driven by Epstein-Barr Virus Protein LMP1 in a Mouse Model

Cell, 2012-02, Vol.148 (4), p.739-751

2012

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The B-cell antigen receptor integrates adaptive and innate immune signals

Proceedings of the National Academy of Sciences - PNAS, 2015-09, Vol.112 (39), p.12145-12150

2015

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A role for the nuclear receptor NR2F6 in peritoneal B cell homeostasis

Frontiers in immunology, 2022-08, Vol.13, p.845235-845235

2022

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Inflammation and the Alteration of B-Cell Physiology in Aging

Gerontology (Basel), 2020-03, Vol.66 (2), p.105-113

2020

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miR-142 favors naïve B cell residence in peripheral lymph nodes

Frontiers in immunology, 2022-11, Vol.13, p.847415-847415

2022

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Canonical NF-κB signaling is uniquely required for the long-term persistence of functional mature B cells

Proceedings of the National Academy of Sciences - PNAS, 2016-05, Vol.113 (18), p.5065-5070

2016

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IKK phosphorylates RelB to modulate its promoter specificity and promote fibroblast migration downstream of TNF receptors

Proceedings of the National Academy of Sciences - PNAS, 2014-10, Vol.111 (41), p.14794-14799

2014

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Cell signaling and the aging of B cells

Experimental gerontology, 2020-09, Vol.138, p.110985-110985, Article 110985

2020

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NIK overexpression amplifies, whereas ablation of its TRAF3-binding domain replaces BAFF:BAFF-R-mediated survival signals in B cells

Proceedings of the National Academy of Sciences - PNAS, 2008-08, Vol.105 (31), p.10883-10888

2008

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The miR-142 miRNAs: Shaping the naïve immune system

Immunology letters, 2023-09, Vol.261, p.37-46

2023

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TET enzymes control antibody production and shape the mutational landscape in germinal centre B cells

The FEBS journal, 2019-09, Vol.286 (18), p.3566-3581

2019

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Canonical NF-κB Activity, Dispensable for B Cell Development, Replaces BAFF-Receptor Signals and Promotes B Cell Proliferation upon Activation

Immunity (Cambridge, Mass.), 2006-06, Vol.24 (6), p.729-739

2006

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T cell-derived IL-17 mediates epithelial changes in the airway and drives pulmonary neutrophilia

The Journal of immunology (1950), 2013-09, Vol.191 (6), p.3100-3111

2013

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Control of NF-κB activity by proteolysis

Current topics in microbiology and immunology, 2011-01, Vol.349, p.97

2011

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Context-specific regulation of cell survival by a miRNA-controlled BIM rheostat

Genes & development, 2019-12, Vol.33 (23-24), p.1673-1687

2019

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Canonical NF-[kappa]B signaling is uniquely required for the long-term persistence of functional mature B cells

Proceedings of the National Academy of Sciences - PNAS, 2016-05, Vol.113 (18), p.5065

2016

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Development of immunoglobulin λ-chain-positive B cells, but not editing of immunoglobulin κ-chain, depends on NF-κB signals

Nature immunology, 2009-06, Vol.10 (6), p.647-654

2009

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RelB/p50 dimers are differentially regulated by tumor necrosis factor-alpha and lymphotoxin-beta receptor activation: critical roles for p100

The Journal of biological chemistry, 2003-06, Vol.278 (26), p.23278-23284

2003

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T cell-derived IL-17 mediates epithelial changes in the airway and drives pulmonary neutrophilia1

The Journal of immunology (1950), 2013-08, Vol.191 (6)

2013

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Correction: T cell–derived IL-17 mediates epithelial changes in the airway and drives pulmonary neutrophilia

The Journal of immunology (1950), 2013-11, Vol.191 (10), p.5318-5318

2013

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Role of NFkappaB signaling in normal and malignant B cell development

Advances in experimental medicine and biology, 2007, Vol.596, p.149

2007

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Development of immunoglobulin [lambda]-chain-positive B cells, but not editing of immunoglobulin [kappa]-chain, depends on NF-[kappa]B signals

Nature immunology, 2009-06, Vol.10 (6), p.647-654

2009

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Igλ+ B cell development but not Igκ editing depends on NF-κB signals

Nature immunology, 2009-05, Vol.10 (6), p.647-654

2009

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Canonical NF-kappaB activity, dispensable for B cell development, replaces BAFF-receptor signals and promotes B cell proliferation upon activation

Immunity (Cambridge, Mass.), 2006-06, Vol.24 (6), p.729-739

2006

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Canonical NF-[kappa]B Activity, Dispensable for B Cell Development, Replaces BAFF-Receptor Signals and Promotes B Cell Proliferation upon Activation

Immunity (Cambridge, Mass.), 2006-06, Vol.24 (6), p.729

2006

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Immune Surveillance and Therapy of Lymphomas Driven by Epstein-Barr Virus Protein LMP1 in a Mouse Model

The B-cell antigen receptor integrates adaptive and innate immune signals

A role for the nuclear receptor NR2F6 in peritoneal B cell homeostasis

Inflammation and the Alteration of B-Cell Physiology in Aging

miR-142 favors naïve B cell residence in peripheral lymph nodes

Canonical NF-κB signaling is uniquely required for the long-term persistence of functional mature B cells

IKK phosphorylates RelB to modulate its promoter specificity and promote fibroblast migration downstream of TNF receptors

Cell signaling and the aging of B cells

NIK overexpression amplifies, whereas ablation of its TRAF3-binding domain replaces BAFF:BAFF-R-mediated survival signals in B cells

The miR-142 miRNAs: Shaping the naïve immune system

TET enzymes control antibody production and shape the mutational landscape in germinal centre B cells

Canonical NF-κB Activity, Dispensable for B Cell Development, Replaces BAFF-Receptor Signals and Promotes B Cell Proliferation upon Activation

T cell-derived IL-17 mediates epithelial changes in the airway and drives pulmonary neutrophilia

Control of NF-κB activity by proteolysis

Context-specific regulation of cell survival by a miRNA-controlled BIM rheostat

Canonical NF-[kappa]B signaling is uniquely required for the long-term persistence of functional mature B cells

Development of immunoglobulin λ-chain-positive B cells, but not editing of immunoglobulin κ-chain, depends on NF-κB signals

RelB/p50 dimers are differentially regulated by tumor necrosis factor-alpha and lymphotoxin-beta receptor activation: critical roles for p100

T cell-derived IL-17 mediates epithelial changes in the airway and drives pulmonary neutrophilia1

Correction: T cell–derived IL-17 mediates epithelial changes in the airway and drives pulmonary neutrophilia

Role of NFkappaB signaling in normal and malignant B cell development

Development of immunoglobulin [lambda]-chain-positive B cells, but not editing of immunoglobulin [kappa]-chain, depends on NF-[kappa]B signals

Igλ+ B cell development but not Igκ editing depends on NF-κB signals

Canonical NF-kappaB activity, dispensable for B cell development, replaces BAFF-receptor signals and promotes B cell proliferation upon activation

Canonical NF-[kappa]B Activity, Dispensable for B Cell Development, Replaces BAFF-Receptor Signals and Promotes B Cell Proliferation upon Activation

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