UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Novel quantitative autophagy analysis by organelle flow cytometry after cell sonication

PloS one, 2014-01, Vol.9 (1), p.e87707-e87707

2014

Volltextzugriff (PDF)

Akt inhibition promotes autophagy and sensitizes PTEN-null tumors to lysosomotropic agents

The Journal of cell biology, 2008-10, Vol.183 (1), p.101-116

2008

Volltextzugriff (PDF)

Conditional Expression of a Gi-Coupled Receptor Causes Ventricular Conduction Delay and a Lethal Cardiomyopathy

Proceedings of the National Academy of Sciences - PNAS, 2000-04, Vol.97 (9), p.4826-4831

2000

Volltextzugriff (PDF)

Autophagy, an Achilles' heel AKTing against cancer?

Autophagy, 2009-04, Vol.5 (3), p.415-418

2009

Volltextzugriff (PDF)

Evolution of microstructure and microtexture upon recrystallization of submicrocrystalline niobium

International journal of refractory metals & hard materials, 2020-01, Vol.86, p.105117, Article 105117

2020

Volltextzugriff (PDF)

Targeting Activated Akt with GDC-0068, a Novel Selective Akt Inhibitor That Is Efficacious in Multiple Tumor Models

Clinical cancer research, 2013-04, Vol.19 (7), p.1760-1772

2013

Volltextzugriff (PDF)

Abstract 328: Novel quantitative autophagy analysis by organelle flow cytometry after cell sonication

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.328-328

2014

Volltextzugriff (PDF)

Novel Quantitative Autophagy Analysis by Organelle Flow Cytometry after Cell Sonication: e87707

PloS one, 2014-01, Vol.9 (1)

2014

Volltextzugriff (PDF)

Abstract 873: Combined targeting of Akt and MEK with the Akt inhibitor GDC-0068 and MEK inhibitor GDC-0973 demonstrates synergistic anti-tumor effects

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.873-873

2012

Volltextzugriff (PDF)

mTOR kinase inhibitor potentiates apoptosis of PI3K and MEK inhibitors in diagnostically defined subpopulations

Cancer letters, 2012-12, Vol.326 (2), p.168-175

2012

Volltextzugriff (PDF)

Increased palmitoylation of the Gs protein alpha subunit after activation by the beta-adrenergic receptor or cholera toxin

The Journal of biological chemistry, 1993-11, Vol.268 (32), p.23769-23772

1993

Volltextzugriff (PDF)

The Stoichiometry of Gαs Palmitoylation in Its Basal and Activated States

Biochemistry (Easton), 1997-06, Vol.36 (23), p.7185-7191

1997

Volltextzugriff (PDF)

Palmitoylation of a G protein alpha i subunit requires membrane localization not myristoylation

The Journal of biological chemistry, 1994-12, Vol.269 (49), p.30898-30903

1994

Volltextzugriff (PDF)

Conditional expression and signaling of a specifically designed Gi-coupled receptor in transgenic mice

Nature biotechnology, 1999-02, Vol.17 (2), p.165-169

1999

Volltextzugriff (PDF)

The G protein .alpha.s subunit incorporates [3H]palmitic acid and mutation of cysteine-3 prevents this modification

Biochemistry (Easton), 1993-08, Vol.32 (32), p.8057-8061

1993

Volltextzugriff (PDF)

The G protein αs subunit incorporates [3H]palmitic acid and mutation of cysteine-3 prevents this modification

Biochemistry (Easton), 1993-08, Vol.32 (32), p.8057-8061

1993

Volltextzugriff (PDF)

The stoichiometry of G alpha(s) palmitoylation in its basal and activated states

Biochemistry (Easton), 1997-06, Vol.36 (23), p.7185-7191

1997

Volltextzugriff (PDF)

The N-terminal Extension of Gαq Is Critical for Constraining the Selectivity of Receptor Coupling

The Journal of biological chemistry, 1997-08, Vol.272 (31), p.19107-19110

1997

Volltextzugriff (PDF)

Conditional expression and signaling of a specifically designed G i -coupled receptor in transgenic mice

Nature biotechnology, 1999-02, Vol.17 (2), p.165-169

1999

Volltextzugriff (PDF)

The membrane localization of the G protein αs subunit is not dependent on its tenir sequence or effector domain

Cellular signalling, 1994, Vol.6 (1), p.25-33

1994

Volltextzugriff (PDF)

Conditional expression of a G i -coupled receptor causes ventricular conduction delay and a lethal cardiomyopathy

Proceedings of the National Academy of Sciences - PNAS, 2000-04, Vol.97 (9), p.4826-4831

2000

Volltextzugriff (PDF)

Genomic analysis of an inducible and reversible model of dilated cardiomyopathy

Journal of cardiac failure, 1999-09, Vol.5 (3), p.14-14

1999

Volltextzugriff (PDF)

A critical period of vulnerability to hyperactive Gq signaling revealed by inducible and heart-specific expression of Gq∗ in transgenic mice

Journal of cardiac failure, 1999-09, Vol.5 (3), p.12-12

1999

Volltextzugriff (PDF)

The N-terminal Extension of GÎ±q Is Critical for Constraining the Selectivity of Receptor Coupling

The Journal of biological chemistry, 1997-08, Vol.272 (31), p.19107

1997

Volltextzugriff (PDF)

Second-Line Tisagenlecleucel or Standard Care in Aggressive B-Cell Lymphoma

The New England journal of medicine, 2022-02, Vol.386 (7), p.629-639

2022

Volltextzugriff (PDF)

1 – 25 von 46

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(44)
Mit Volltextzugriff
(44)

Publikationsform

Sprache

Englisch
(46)

Erscheinungsjahr

n.n

Thema

Science & Technology
(19)
Animals
(11)
Life Sciences & Biomedicine
(9)
Exact Sciences And Technology
(8)
Humans
(8)
Technology
(8)
Applied Sciences
(7)
Computer Science
(7)
Computer Science; Control Theory; Systems
(7)
Artificial Intelligence
(5)
Biological And Medical Sciences
(5)
Cell Line, Tumor
(5)
Computer Science, Artificial Intelligence
(5)
Gene Expression
(5)
Mice
(5)
Proteins
(5)
Temporal Logic
(5)
Theoretical Computing
(5)
Base Sequence
(4)
Computer Science, Theory & Methods
(4)

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Novel quantitative autophagy analysis by organelle flow cytometry after cell sonication

Akt inhibition promotes autophagy and sensitizes PTEN-null tumors to lysosomotropic agents

Conditional Expression of a Gi-Coupled Receptor Causes Ventricular Conduction Delay and a Lethal Cardiomyopathy

Autophagy, an Achilles' heel AKTing against cancer?

Evolution of microstructure and microtexture upon recrystallization of submicrocrystalline niobium

Targeting Activated Akt with GDC-0068, a Novel Selective Akt Inhibitor That Is Efficacious in Multiple Tumor Models

Abstract 328: Novel quantitative autophagy analysis by organelle flow cytometry after cell sonication

Novel Quantitative Autophagy Analysis by Organelle Flow Cytometry after Cell Sonication: e87707

Abstract 873: Combined targeting of Akt and MEK with the Akt inhibitor GDC-0068 and MEK inhibitor GDC-0973 demonstrates synergistic anti-tumor effects

mTOR kinase inhibitor potentiates apoptosis of PI3K and MEK inhibitors in diagnostically defined subpopulations

Increased palmitoylation of the Gs protein alpha subunit after activation by the beta-adrenergic receptor or cholera toxin

The Stoichiometry of Gαs Palmitoylation in Its Basal and Activated States

Palmitoylation of a G protein alpha i subunit requires membrane localization not myristoylation

Conditional expression and signaling of a specifically designed Gi-coupled receptor in transgenic mice

The G protein .alpha.s subunit incorporates [3H]palmitic acid and mutation of cysteine-3 prevents this modification

The G protein αs subunit incorporates [3H]palmitic acid and mutation of cysteine-3 prevents this modification

The stoichiometry of G alpha(s) palmitoylation in its basal and activated states

The N-terminal Extension of Gαq Is Critical for Constraining the Selectivity of Receptor Coupling

Conditional expression and signaling of a specifically designed G i -coupled receptor in transgenic mice

The membrane localization of the G protein αs subunit is not dependent on its tenir sequence or effector domain

Conditional expression of a G i -coupled receptor causes ventricular conduction delay and a lethal cardiomyopathy

Genomic analysis of an inducible and reversible model of dilated cardiomyopathy

A critical period of vulnerability to hyperactive Gq signaling revealed by inducible and heart-specific expression of Gq∗ in transgenic mice

The N-terminal Extension of GÎ±q Is Critical for Constraining the Selectivity of Receptor Coupling

Second-Line Tisagenlecleucel or Standard Care in Aggressive B-Cell Lymphoma

Suchergebnisse filtern