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A technology platform to assess multiple cancer agents simultaneously within a patient's tumor

Science translational medicine, 2015-04, Vol.7 (284), p.284ra58

2015

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Discovery of Potent Antagonists of the Antiapoptotic Protein XIAP for the Treatment of Cancer

Journal of medicinal chemistry, 2004-08, Vol.47 (18), p.4417-4426

2004

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BAX Is Required for Neuronal Death after Trophic Factor Deprivation and during Development

Neuron (Cambridge, Mass.), 1996-09, Vol.17 (3), p.401-411

1996

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A monobromobimane‐based assay to measure the pharmacokinetic profile of reactive sulphide species in blood

British journal of pharmacology, 2010-06, Vol.160 (4), p.941-957

2010

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Voruciclib, a clinical stage oral CDK9 inhibitor, represses MCL-1 and sensitizes high-risk Diffuse Large B-cell Lymphoma to BCL2 inhibition

Scientific reports, 2017-12, Vol.7 (1), p.18007-18007, Article 18007

2017

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Detection of exhaled hydrogen sulphide gas in healthy human volunteers during intravenous administration of sodium sulphide

British journal of clinical pharmacology, 2010-06, Vol.69 (6), p.626-636

2010

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Detection of exhaled hydrogen sulphide gas in rats exposed to intravenous sodium sulphide

British journal of pharmacology, 2009-07, Vol.157 (6), p.944-951

2009

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Endoplasmic Reticulum Stress in PLP-Overexpressing Transgenic Rats: Gray Matter Oligodendrocytes Are More Vulnerable than White Matter Oligodendrocytes

Journal of neuropathology and experimental neurology, 2002-01, Vol.61 (1), p.12-22

2002

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Activation of caspase-3 in single neurons and autophagic granules of granulovacuolar degeneration in Alzheimer's disease : Evidence for apoptotic cell death

The American journal of pathology, 1999-11, Vol.155 (5), p.1459-1466

1999

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Destruction of neurons by cytotoxic T cells: A new pathogenic mechanism in rasmussen's encephalitis

Annals of neurology, 2002-03, Vol.51 (3), p.311-318

2002

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Superoxide dismutase delays neuronal apoptosis: A role for reactive oxygen species in programmed neuronal death

Neuron (Cambridge, Mass.), 1995-02, Vol.14 (2), p.303-315

1995

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Temporal Analysis of Events Associated with Programmed Cell Death (Apoptosis) of Sympathetic Neurons Deprived of Nerve Growth Factor

The Journal of cell biology, 1993-12, Vol.123 (5), p.1207-1222

1993

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Analysis of the Mechanism of Loss of Trophic Factor Dependence Associated with Neuronal Maturation: A Phenotype Indistinguishable from Bax Deletion

The Journal of neuroscience, 1997-12, Vol.17 (24), p.9656-9666

1997

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Neurites Can Remain Viable after Destruction of the Neuronal Soma by Programmed Cell Death (Apoptosis)

Developmental biology, 1994-09, Vol.165 (1), p.63-72

1994

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Placement of the BCL2 Family Member BAX in the Death Pathway of Sympathetic Neurons Activated by Trophic Factor Deprivation

Experimental neurology, 1998-07, Vol.152 (1), p.150-162

1998

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Oxamyl dipeptide caspase inhibitors developed for the treatment of stroke

Bioorganic & medicinal chemistry letters, 2004-05, Vol.14 (10), p.2685-2691

2004

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Structure-activity relationships within a series of caspase inhibitors: Effect of leaving group modifications

Bioorganic & medicinal chemistry letters, 2003-10, Vol.13 (20), p.3623-3626

2003

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Neuronal Death in Developmental Models: Possible Implications in Neuropathology

Brain pathology (Zurich, Switzerland), 1996-10, Vol.6 (4), p.397-409

1996

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Activation of Caspase-3 in Single Neurons and Autophagic Granules of Granulovacuolar Degeneration in Alzheimer's Disease

The American journal of pathology, 1999-11, Vol.155 (5), p.1459-1466

1999

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Suppression of developmental retinal cell death but not of photoreceptor degeneration in Bax-deficient mice [published erratum appears in Invest Ophthalmol Vis Sci 1998 Nov;39(12):2244]

Investigative ophthalmology & visual science, 1998-08, Vol.39 (9), p.1713-1720

1998

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Expression of Cell Death-Associated Proteins in Neuronal Apoptosis Associated with Pontosubicular Neuron Necrosis

Brain pathology (Zurich, Switzerland), 2001-07, Vol.11 (3), p.273-281

2001

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Small‐molecule Inhibitors of the Inhibitor of Apoptosis Proteins

Apoptosis and Cancer Therapy, 2006, p.913-932

2006

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Neurotrophic Factor Deprivation-induced Death

Annals of the New York Academy of Sciences, 1993-05, Vol.679 (1), p.121-131

1993

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Long-term protection of brain tissue from cerebral ischemia by peripherally administered peptidomimetic caspase inhibitors

Drug development research, 2001-04, Vol.52 (4), p.579-586

2001

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A technology platform to assess multiple cancer agents simultaneously within a patient's tumor

Discovery of Potent Antagonists of the Antiapoptotic Protein XIAP for the Treatment of Cancer

BAX Is Required for Neuronal Death after Trophic Factor Deprivation and during Development

A monobromobimane‐based assay to measure the pharmacokinetic profile of reactive sulphide species in blood

Voruciclib, a clinical stage oral CDK9 inhibitor, represses MCL-1 and sensitizes high-risk Diffuse Large B-cell Lymphoma to BCL2 inhibition

An inhibitor of Bcl-2 family proteins induces regression of solid tumours

Detection of exhaled hydrogen sulphide gas in healthy human volunteers during intravenous administration of sodium sulphide

Detection of exhaled hydrogen sulphide gas in rats exposed to intravenous sodium sulphide

Endoplasmic Reticulum Stress in PLP-Overexpressing Transgenic Rats: Gray Matter Oligodendrocytes Are More Vulnerable than White Matter Oligodendrocytes

Activation of caspase-3 in single neurons and autophagic granules of granulovacuolar degeneration in Alzheimer's disease : Evidence for apoptotic cell death

Destruction of neurons by cytotoxic T cells: A new pathogenic mechanism in rasmussen's encephalitis

Superoxide dismutase delays neuronal apoptosis: A role for reactive oxygen species in programmed neuronal death

Temporal Analysis of Events Associated with Programmed Cell Death (Apoptosis) of Sympathetic Neurons Deprived of Nerve Growth Factor

Analysis of the Mechanism of Loss of Trophic Factor Dependence Associated with Neuronal Maturation: A Phenotype Indistinguishable from Bax Deletion

Neurites Can Remain Viable after Destruction of the Neuronal Soma by Programmed Cell Death (Apoptosis)

Placement of the BCL2 Family Member BAX in the Death Pathway of Sympathetic Neurons Activated by Trophic Factor Deprivation

Oxamyl dipeptide caspase inhibitors developed for the treatment of stroke

Structure-activity relationships within a series of caspase inhibitors: Effect of leaving group modifications

Neuronal Death in Developmental Models: Possible Implications in Neuropathology

Activation of Caspase-3 in Single Neurons and Autophagic Granules of Granulovacuolar Degeneration in Alzheimer's Disease

Suppression of developmental retinal cell death but not of photoreceptor degeneration in Bax-deficient mice [published erratum appears in Invest Ophthalmol Vis Sci 1998 Nov;39(12):2244]

Expression of Cell Death-Associated Proteins in Neuronal Apoptosis Associated with Pontosubicular Neuron Necrosis

Small‐molecule Inhibitors of the Inhibitor of Apoptosis Proteins

Neurotrophic Factor Deprivation-induced Death

Long-term protection of brain tissue from cerebral ischemia by peripherally administered peptidomimetic caspase inhibitors

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