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Naturally Occurring Off-Switches for CRISPR-Cas9

Cell, 2016-12, Vol.167 (7), p.1829-1838.e9

2016

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Prophages mediate defense against phage infection through diverse mechanisms

The ISME Journal, 2016-12, Vol.10 (12), p.2854-2866

2016

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When a virus is not a parasite: the beneficial effects of prophages on bacterial fitness

The Journal of Microbiology, 2014, 52(3), , pp.235-242

2014

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The Discovery, Mechanisms, and Evolutionary Impact of Anti-CRISPRs

Annual review of virology, 2017-09, Vol.4 (1), p.37-59

2017

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A contractile injection system is required for developmentally regulated cell death in Streptomyces coelicolor

Nature communications, 2023-03, Vol.14 (1), p.1469-1469, Article 1469

2023

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Foreign DNA acquisition by the I-F CRISPR-Cas system requires all components of the interference machinery

Nucleic acids research, 2015-12, Vol.43 (22), p.10848-10860

2015

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Anti-CRISPR AcrIF9 functions by inducing the CRISPR-Cas complex to bind DNA non-specifically

Nucleic acids research, 2021-04, Vol.49 (6), p.3381-3393

2021

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CRISPR/Cas Adaptive Immune System of Pseudomonas aeruginosa Mediates Resistance to Naturally Occurring and Engineered Phages

Journal of Bacteriology, 2012-11, Vol.194 (21), p.5728-5738

2012

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The phage tail tape measure protein, an inner membrane protein and a periplasmic chaperone play connected roles in the genome injection process of E. coli phage HK97

Molecular microbiology, 2015-05, Vol.96 (3), p.437-447

2015

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Complete genomes and comparative analyses of Streptomyces phages that influence secondary metabolism and sporulation

Scientific reports, 2023-06, Vol.13 (1), p.9820-9820, Article 9820

2023

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A new group of phage anti-CRISPR genes inhibits the type I-E CRISPR-Cas system of Pseudomonas aeruginosa

mBio, 2014-04, Vol.5 (2), p.e00896-e00896

2014

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Anti-phage defence through inhibition of virion assembly

Nature communications, 2024-02, Vol.15 (1), p.1644-1644, Article 1644

2024

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Potent Cas9 Inhibition in Bacterial and Human Cells by AcrIIC4 and AcrIIC5 Anti-CRISPR Proteins

mBio, 2018-12, Vol.9 (6)

2018

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The solution structure of an anti-CRISPR protein

Nature communications, 2016-10, Vol.7 (1), p.13134-13134, Article 13134

2016

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AcrIF9 tethers non-sequence specific dsDNA to the CRISPR RNA-guided surveillance complex

Nature communications, 2020-06, Vol.11 (1), p.2730-2730, Article 2730

2020

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HNH proteins are a widespread component of phage DNA packaging machines

Proceedings of the National Academy of Sciences - PNAS, 2014-04, Vol.111 (16), p.6022-6027

2014

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Inhibition of CRISPR-Cas9 ribonucleoprotein complex assembly by anti-CRISPR AcrIIC2

Nature communications, 2019-06, Vol.10 (1), p.2806-11, Article 2806

2019

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phage λ major tail protein structure reveals a common evolution for long-tailed phages and the type VI bacterial secretion system

Proceedings of the National Academy of Sciences - PNAS, 2009-03, Vol.106 (11), p.4160-4165

2009

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Disabling a Type I-E CRISPR-Cas Nuclease with a Bacteriophage-Encoded Anti-CRISPR Protein

mBio, 2017-12, Vol.8 (6)

2017

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Anti-CRISPR: discovery, mechanism and function

Nature reviews. Microbiology, 2018-01, Vol.16 (1), p.12-17

2018

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Bacteriophage HK97 gp15 Moron Element Encodes a Novel Superinfection Exclusion Protein

Journal of Bacteriology, 2012-09, Vol.194 (18), p.5012-5019

2012

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Proteome-wide discovery of evolutionary conserved sequences in disordered regions

Science signaling, 2012-03, Vol.5 (215), p.rs1

2012

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A Comprehensive Analysis of Structural and Sequence Conservation in the TetR Family Transcriptional Regulators

Journal of molecular biology, 2010-07, Vol.400 (4), p.847-864

2010

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The Induction of Folding Cooperativity by Ligand Binding Drives the Allosteric Response of Tetracycline Repressor

Proceedings of the National Academy of Sciences - PNAS, 2009-12, Vol.106 (52), p.22263-22268

2009

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Anti-CRISPR AcrIIA5 Potently Inhibits All Cas9 Homologs Used for Genome Editing

Cell reports (Cambridge), 2019-11, Vol.29 (7), p.1739-1746.e5

2019

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Naturally Occurring Off-Switches for CRISPR-Cas9

Prophages mediate defense against phage infection through diverse mechanisms

When a virus is not a parasite: the beneficial effects of prophages on bacterial fitness

The Discovery, Mechanisms, and Evolutionary Impact of Anti-CRISPRs

A contractile injection system is required for developmentally regulated cell death in Streptomyces coelicolor

Foreign DNA acquisition by the I-F CRISPR-Cas system requires all components of the interference machinery

Anti-CRISPR AcrIF9 functions by inducing the CRISPR-Cas complex to bind DNA non-specifically

CRISPR/Cas Adaptive Immune System of Pseudomonas aeruginosa Mediates Resistance to Naturally Occurring and Engineered Phages

The phage tail tape measure protein, an inner membrane protein and a periplasmic chaperone play connected roles in the genome injection process of E. coli phage HK97

Complete genomes and comparative analyses of Streptomyces phages that influence secondary metabolism and sporulation

A new group of phage anti-CRISPR genes inhibits the type I-E CRISPR-Cas system of Pseudomonas aeruginosa

Anti-phage defence through inhibition of virion assembly

Potent Cas9 Inhibition in Bacterial and Human Cells by AcrIIC4 and AcrIIC5 Anti-CRISPR Proteins

The solution structure of an anti-CRISPR protein

AcrIF9 tethers non-sequence specific dsDNA to the CRISPR RNA-guided surveillance complex

HNH proteins are a widespread component of phage DNA packaging machines

Inhibition of CRISPR-Cas9 ribonucleoprotein complex assembly by anti-CRISPR AcrIIC2

phage λ major tail protein structure reveals a common evolution for long-tailed phages and the type VI bacterial secretion system

Disabling a Type I-E CRISPR-Cas Nuclease with a Bacteriophage-Encoded Anti-CRISPR Protein

Anti-CRISPR: discovery, mechanism and function

Bacteriophage HK97 gp15 Moron Element Encodes a Novel Superinfection Exclusion Protein

Proteome-wide discovery of evolutionary conserved sequences in disordered regions

A Comprehensive Analysis of Structural and Sequence Conservation in the TetR Family Transcriptional Regulators

The Induction of Folding Cooperativity by Ligand Binding Drives the Allosteric Response of Tetracycline Repressor

Anti-CRISPR AcrIIA5 Potently Inhibits All Cas9 Homologs Used for Genome Editing

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