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Neuregulin-1/ErbB4 Signaling Regulates Visual Cortical Plasticity

Neuron (Cambridge, Mass.), 2016-10, Vol.92 (1), p.160-173

2016

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The Antinociceptive Properties of the Corydalis yanhusuo Extract

PloS one, 2016-09, Vol.11 (9), p.e0162875-e0162875

2016

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Epileptiform activity and behavioral arrests in mice overexpressing the calcium channel subunit α2δ-1

Neurobiology of disease, 2017-06, Vol.102, p.70-80

2017

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Calcium channel alpha-2-delta-1 protein upregulation in dorsal spinal cord mediates spinal cord injury-induced neuropathic pain states

Pain (Amsterdam), 2011-03, Vol.152 (3), p.649-655

2011

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A Novel Analgesic Isolated from a Traditional Chinese Medicine

Current biology, 2014-01, Vol.24 (2), p.117-123

2014

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Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states

The Journal of neuroscience, 2012-06, Vol.32 (26), p.8977-8987

2012

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Profiling of dynamically changed gene expression in dorsal root ganglia post peripheral nerve injury and a critical role of injury-induced glial fibrillary acetic protein in maintenance of pain behaviors

Pain (Amsterdam), 2009-05, Vol.143 (1), p.114-122

2009

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Central Mechanisms Mediating Thrombospondin-4-induced Pain States

The Journal of biological chemistry, 2016-06, Vol.291 (25), p.13335-13348

2016

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The EGF-LIKE domain of thrombospondin-4 is a key determinant in the development of pain states due to increased excitatory synaptogenesis

The Journal of biological chemistry, 2018-10, Vol.293 (42), p.16453-16463

2018

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Calcium channel functions in pain processing

Channels (Austin, Tex.), 2010-11, Vol.4 (6), p.510-517

2010

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Thrombospondin-4 divergently regulates voltage-gated Ca2+ channel subtypes in sensory neurons after nerve injury

Pain (Amsterdam), 2016-09, Vol.157 (9), p.2068-2080

2016

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Pathophysiological roles of thrombospondin-4 in disease development

Seminars in cell & developmental biology, 2024-03, Vol.155 (Pt B), p.66-73

2024

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Persistent inflammation alters the density and distribution of voltage-activated calcium channels in subpopulations of rat cutaneous DRG neurons

Pain (Amsterdam), 2010-12, Vol.151 (3), p.633-643

2010

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Injury-induced maladaptation and dysregulation of calcium channel α 2 δ subunit proteins and its contribution to neuropathic pain development

British journal of pharmacology, 2018-06, Vol.175 (12), p.2231-2243

2018

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Injury discharges regulate calcium channel alpha-2-delta-1 subunit upregulation in the dorsal horn that contributes to initiation of neuropathic pain

Pain (Amsterdam), 2008-10, Vol.139 (2), p.358-366

2008

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Enhanced Pre-Synaptic Glutamate Release in Deep-Dorsal Horn Contributes to Calcium Channel Alpha-2-Delta-1 Protein-Mediated Spinal Sensitization and Behavioral Hypersensitivity

Molecular pain, 2009-02, Vol.5 (1), p.6-6

2009

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Impaired Pain Sensation in Mice Lacking Prokineticin 2

Molecular pain, 2006-11, Vol.2 (1), p.35-35

2006

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Targeting Voltage-Gated Calcium Channels for Neuropathic Pain Management

Neurotherapeutics, 2009-10, Vol.6 (4), p.679-692

2009

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Painful nerve injury upregulates thrombospondin-4 expression in dorsal root ganglia

Journal of neuroscience research, 2015-03, Vol.93 (3), p.443-453

2015

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Thrombospondin-4 and excitatory synaptogenesis promote spinal sensitization after painful mechanical joint injury

Experimental neurology, 2015-02, Vol.264, p.111-120

2015

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Increased thrombospondin-4 after nerve injury mediates disruption of intracellular calcium signaling in primary sensory neurons

Neuropharmacology, 2017-05, Vol.117, p.292-304

2017

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Calcium Channel α2δ1 Proteins Mediate Trigeminal Neuropathic Pain States Associated with Aberrant Excitatory Synaptogenesis

The Journal of biological chemistry, 2014-03, Vol.289 (10), p.7025-7037

2014

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Spinal Dorsal Horn Calcium Channel α2δ-1 Subunit Upregulation Contributes to Peripheral Nerve Injury-Induced Tactile Allodynia

The Journal of neuroscience, 2004-09, Vol.24 (39), p.8494-8499

2004

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Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury

Journal of comparative neurology (1911), 2016-02, Vol.524 (2), p.309-322

2016

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Neuregulin-1/ErbB4 Signaling Regulates Visual Cortical Plasticity

The Antinociceptive Properties of the Corydalis yanhusuo Extract

Epileptiform activity and behavioral arrests in mice overexpressing the calcium channel subunit α2δ-1

Calcium channel alpha-2-delta-1 protein upregulation in dorsal spinal cord mediates spinal cord injury-induced neuropathic pain states

A Novel Analgesic Isolated from a Traditional Chinese Medicine

Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states

Profiling of dynamically changed gene expression in dorsal root ganglia post peripheral nerve injury and a critical role of injury-induced glial fibrillary acetic protein in maintenance of pain behaviors

Central Mechanisms Mediating Thrombospondin-4-induced Pain States

The EGF-LIKE domain of thrombospondin-4 is a key determinant in the development of pain states due to increased excitatory synaptogenesis

Calcium channel functions in pain processing

Thrombospondin-4 divergently regulates voltage-gated Ca2+ channel subtypes in sensory neurons after nerve injury

Pathophysiological roles of thrombospondin-4 in disease development

Persistent inflammation alters the density and distribution of voltage-activated calcium channels in subpopulations of rat cutaneous DRG neurons

Injury-induced maladaptation and dysregulation of calcium channel α 2 δ subunit proteins and its contribution to neuropathic pain development

Injury discharges regulate calcium channel alpha-2-delta-1 subunit upregulation in the dorsal horn that contributes to initiation of neuropathic pain

Gabapentin Receptor α2δ-1 Is a Neuronal Thrombospondin Receptor Responsible for Excitatory CNS Synaptogenesis

Enhanced Pre-Synaptic Glutamate Release in Deep-Dorsal Horn Contributes to Calcium Channel Alpha-2-Delta-1 Protein-Mediated Spinal Sensitization and Behavioral Hypersensitivity

Impaired Pain Sensation in Mice Lacking Prokineticin 2

Targeting Voltage-Gated Calcium Channels for Neuropathic Pain Management

Painful nerve injury upregulates thrombospondin-4 expression in dorsal root ganglia

Thrombospondin-4 and excitatory synaptogenesis promote spinal sensitization after painful mechanical joint injury

Increased thrombospondin-4 after nerve injury mediates disruption of intracellular calcium signaling in primary sensory neurons

Calcium Channel α2δ1 Proteins Mediate Trigeminal Neuropathic Pain States Associated with Aberrant Excitatory Synaptogenesis

Spinal Dorsal Horn Calcium Channel α2δ-1 Subunit Upregulation Contributes to Peripheral Nerve Injury-Induced Tactile Allodynia

Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury

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