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Helicobacter pylori associated chronic gastritis,clinical syndromes,precancerous lesions,and pathogenesis of gastric cancer development

World journal of gastroenterology : WJG, 2014-05, Vol.20 (18), p.5461-5473

2014

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mAb Das-1 recognizes 3'-Sulfated Lewis A/C, which is aberrantly expressed during metaplastic and oncogenic transformation of several gastrointestinal Epithelia

PloS one, 2021-12, Vol.16 (12), p.e0261082

2021

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Long-term effects of H. pylori eradication on epigenetic alterations related to gastric carcinogenesis

Scientific reports, 2018-09, Vol.8 (1), p.14369-13, Article 14369

2018

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mAb Das-1 is specific for high-risk and malignant intraductal papillary mucinous neoplasm (IPMN)

Gut, 2014-10, Vol.63 (10), p.1626-1634

2014

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Intestinal Differentiation in Metaplastic, Nongoblet Columnar Epithelium in the Esophagus

The American journal of surgical pathology, 2009-07, Vol.33 (7), p.1006-1015

2009

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Recurring Translocations in Barrett’s Esophageal Adenocarcinoma

Frontiers in genetics, 2021-06, Vol.12, p.674741-674741

2021

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Molecular alterations and PD-L1 expression in non-ampullary duodenal adenocarcinoma: Associations among clinicopathological, immunophenotypic and molecular features

Scientific reports, 2019-07, Vol.9 (1), p.10526-10, Article 10526

2019

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368 3’-SULFATED LEWIS A IS A BIOMARKER FOR METAPLASTIC AND CARCINOMATOUS TRANSFORMATION OF SEVERAL GASTROINTESTINAL EPITHELIA

Gastroenterology (New York, N.Y. 1943), 2021-05, Vol.160 (6), p.S-71-S-71

2021

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Cellular origins and molecular mechanisms of Barrett's esophagus and esophageal adenocarcinoma

Annals of the New York Academy of Sciences, 2013-10, Vol.1300 (1), p.187-199

2013

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Localization of specialized intestinal metaplasia and the molecular alterations in Barrett esophagus in a Japanese population: an analysis of biopsy samples based on the “Seattle” biopsy protocol

Human pathology, 2016-05, Vol.51, p.32-40

2016

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Management of Musculoskeletal Manifestations in Inflammatory Bowel Disease

Gastroenterology research and practice, 2015-01, Vol.2015, p.387891-11

2015

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High resolution integrative analysis reveals widespread genetic and epigenetic changes after chronic in-vitro acid and bile exposure in barrett's epithelium cells

Genes chromosomes & cancer, 2013-12, Vol.52 (12), p.1123-1132

2013

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Pathogenesis and clinical spectrum of primary sclerosing cholangitis

World journal of gastroenterology : WJG, 2017-04, Vol.23 (14), p.2459-2469

2017

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Biomarkers predicting development of metachronous gastric cancer after endoscopic resection: an analysis of molecular pathology of Helicobacter pylori eradication

International journal of cancer, 2012-05, Vol.130 (10), p.2349-2358

2012

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543 Support for Wound Healing As the Mechanism for Columnar Metaplasia of the Esophagus in a Rodent Model of Barrett's Esophagus

Gastroenterology (New York, N.Y. 1943), 2015-04, Vol.148 (4), p.S-109-S-109

2015

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Prolonged exposure to acid and bile induces chromosome abnormalities that precede malignant transformation of benign Barrett's epithelium

Molecular cytogenetics, 2012-11, Vol.5 (1), p.43-43, Article 43

2012

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Novel Recurring Fusions: Predictive Markers of Esophageal Adenocarcinoma

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S1029-S1029

2017

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Evaluation of a U.S. National Cohort to Determine Utilization in Colectomy Rates for Ulcerative Colitis Among Ethnicities

Inflammatory bowel diseases, 2022-01, Vol.28 (1), p.54-61

2022

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Comparison of Disease Phenotypes and Clinical Characteristics Among South Asian and White Patients with Inflammatory Bowel Disease at a Tertiary Referral Center

Inflammatory bowel diseases, 2020-12, Vol.26 (12), p.1869-1877

2020

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DNA methylation silencing of microRNA gene methylator in the precancerous background mucosa with and without gastric cancer: Analysis of the effects of H. pylori eradication and long-term aspirin use

Scientific reports, 2019-08, Vol.9 (1), p.12559-10, Article 12559

2019

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Cross Validation of the Monoclonal Antibody Das-1 in Identification of High-Risk Mucinous Pancreatic Cystic Lesions

Gastroenterology (New York, N.Y. 1943), 2019-09, Vol.157 (3), p.720-730.e2

2019

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Musculoskeletal Manifestations in Inflammatory Bowel Disease: A Revisit in Search of Immunopathophysiological Mechanisms

Journal of clinical gastroenterology, 2014-04, Vol.48 (4), p.308-317

2014

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Effect of Eradication of Helicobacter pylori on the Histology and Cellular Phenotype of Gastric Intestinal Metaplasia

Clinical gastroenterology and hepatology, 2008-04, Vol.6 (4), p.409-417

2008

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Mesalamine Suppresses the Expression of TC22, a Novel Tropomyosin Isoform Associated with Colonic Neoplasia

Molecular pharmacology, 2009-07, Vol.76 (1), p.183-191

2009

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Barrett's metaplasia develops from cellular reprograming of esophageal squamous epithelium due to gastroesophageal reflux

American journal of physiology: Gastrointestinal and liver physiology, 2017-06, Vol.312 (6), p.G615-G622

2017

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Helicobacter pylori associated chronic gastritis,clinical syndromes,precancerous lesions,and pathogenesis of gastric cancer development

mAb Das-1 recognizes 3'-Sulfated Lewis A/C, which is aberrantly expressed during metaplastic and oncogenic transformation of several gastrointestinal Epithelia

Long-term effects of H. pylori eradication on epigenetic alterations related to gastric carcinogenesis

mAb Das-1 is specific for high-risk and malignant intraductal papillary mucinous neoplasm (IPMN)

Intestinal Differentiation in Metaplastic, Nongoblet Columnar Epithelium in the Esophagus

Recurring Translocations in Barrett’s Esophageal Adenocarcinoma

Molecular alterations and PD-L1 expression in non-ampullary duodenal adenocarcinoma: Associations among clinicopathological, immunophenotypic and molecular features

368 3’-SULFATED LEWIS A IS A BIOMARKER FOR METAPLASTIC AND CARCINOMATOUS TRANSFORMATION OF SEVERAL GASTROINTESTINAL EPITHELIA

Cellular origins and molecular mechanisms of Barrett's esophagus and esophageal adenocarcinoma

Localization of specialized intestinal metaplasia and the molecular alterations in Barrett esophagus in a Japanese population: an analysis of biopsy samples based on the “Seattle” biopsy protocol

Management of Musculoskeletal Manifestations in Inflammatory Bowel Disease

High resolution integrative analysis reveals widespread genetic and epigenetic changes after chronic in-vitro acid and bile exposure in barrett's epithelium cells

Pathogenesis and clinical spectrum of primary sclerosing cholangitis

Biomarkers predicting development of metachronous gastric cancer after endoscopic resection: an analysis of molecular pathology of Helicobacter pylori eradication

543 Support for Wound Healing As the Mechanism for Columnar Metaplasia of the Esophagus in a Rodent Model of Barrett's Esophagus

Prolonged exposure to acid and bile induces chromosome abnormalities that precede malignant transformation of benign Barrett's epithelium

Novel Recurring Fusions: Predictive Markers of Esophageal Adenocarcinoma

Evaluation of a U.S. National Cohort to Determine Utilization in Colectomy Rates for Ulcerative Colitis Among Ethnicities

Comparison of Disease Phenotypes and Clinical Characteristics Among South Asian and White Patients with Inflammatory Bowel Disease at a Tertiary Referral Center

DNA methylation silencing of microRNA gene methylator in the precancerous background mucosa with and without gastric cancer: Analysis of the effects of H. pylori eradication and long-term aspirin use

Cross Validation of the Monoclonal Antibody Das-1 in Identification of High-Risk Mucinous Pancreatic Cystic Lesions

Musculoskeletal Manifestations in Inflammatory Bowel Disease: A Revisit in Search of Immunopathophysiological Mechanisms

Effect of Eradication of Helicobacter pylori on the Histology and Cellular Phenotype of Gastric Intestinal Metaplasia

Mesalamine Suppresses the Expression of TC22, a Novel Tropomyosin Isoform Associated with Colonic Neoplasia

Barrett's metaplasia develops from cellular reprograming of esophageal squamous epithelium due to gastroesophageal reflux

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