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Periodontitis: a polymicrobial disruption of host homeostasis

Nature reviews. Microbiology, 2010-07, Vol.8 (7), p.481-490

Darveau, Richard P

2010

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Oral biofilms revisited: A novel host tissue of bacteriological origin

Periodontology 2000, 2021-06, Vol.86 (1), p.8-13

2021

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The keystone-pathogen hypothesis

Nature reviews. Microbiology, 2012-10, Vol.10 (10), p.717-725

2012

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Microbial transitions from health to disease

Periodontology 2000, 2021-06, Vol.86 (1), p.201-209

2021

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Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis

Periodontology 2000, 2010-10, Vol.54 (1), p.53-70

2010

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The dental plaque biofilm matrix

Periodontology 2000, 2021-06, Vol.86 (1), p.32-56

2021

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The Relationship of the Oral Microbiotia to Periodontal Health and Disease

Cell host & microbe, 2011-10, Vol.10 (4), p.302-306

2011

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Microbial protection and virulence in periodontal tissue as a function of polymicrobial communities: symbiosis and dysbiosis

Periodontology 2000, 2015-10, Vol.69 (1), p.18-27

2015

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Low-Abundance Biofilm Species Orchestrates Inflammatory Periodontal Disease through the Commensal Microbiota and Complement

Cell host & microbe, 2011-11, Vol.10 (5), p.497-506

2011

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Tetra- and penta-acylated lipid A structures of Porphyromonas gingivalis LPS differentially activate TLR4-mediated NF-κB signal transduction cascade and immuno-inflammatory response in human gingival fibroblasts

PloS one, 2013-03, Vol.8 (3), p.e58496

2013

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Microbial shift and periodontitis

Periodontology 2000, 2011-02, Vol.55 (1), p.36-47

2011

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Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation

PLoS pathogens, 2014-07, Vol.10 (7), p.e1004215-e1004215

2014

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Maintaining homeostatic control of periodontal epithelial tissue

Periodontology 2000, 2021-06, Vol.86 (1), p.188-200

2021

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The oral microbial consortium's interaction with the periodontal innate defense system

DNA and cell biology, 2009-08, Vol.28 (8), p.389

Darveau, Richard P

2009

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3LPS-binding protein and its interactions with P. gingivalis LPS modulate pro-inflammatory response and Toll-like receptor signaling in human oral keratinocytes

PloS one, 2017-04, Vol.12 (4), p.e0173223-e0173223

2017

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Porphyromonas gingivalis induces penetration of lipopolysaccharide and peptidoglycan through the gingival epithelium via degradation of junctional adhesion molecule 1

PLoS pathogens, 2019-11, Vol.15 (11), p.e1008124-e1008124

2019

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Defensins and LL-37: a review of function in the gingival epithelium

Periodontology 2000, 2013-10, Vol.63 (1), p.67-79

2013

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Gingival Epithelial Cell Recognition of Lipopolysaccharide

Oral Mucosal Immunity and Microbiome, p.55-67

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Periodontal pathogens promote cancer aggressivity via TLR/MyD88 triggered activation of Integrin/FAK signaling that is therapeutically reversible by a probiotic bacteriocin

PLoS pathogens, 2020-10, Vol.16 (10), p.e1008881

2020

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Macrophage migration inhibitory factor regulates specific innate immune sensor responses in gingival epithelial cells

Journal of periodontology (1970), 2022-12, Vol.93 (12), p.1940-1950

2022

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A Novel Class of Lipoprotein Lipase-Sensitive Molecules Mediates Toll-Like Receptor 2 Activation by Porphyromonas gingivalis

Infection and Immunity, 2013-04, Vol.81 (4), p.1277-1286

2013

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Genomic and functional characterization of a mucosal symbiont involved in early-stage colorectal cancer

Cell host & microbe, 2021-10, Vol.29 (10), p.1589-1598.e6

2021

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Does Oral Endotoxin Contribute to Systemic Inflammation?

Frontiers in oral health, 2022-05, Vol.3, p.911420-911420

2022

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Effects of a stabilized stannous fluoride dentifrice on clinical, immunomodulatory, and microbial outcomes in a human experimental gingivitis model

Journal of periodontology (1970), 2024-05, Vol.95 (5), p.421-431

2024

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Toll‐like receptor‐2 and ‐4 responses regulate neutrophil infiltration into the junctional epithelium and significantly contribute to the composition of the oral microbiota

Journal of periodontology (1970), 2019-10, Vol.90 (10), p.1202-1212

2019

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Periodontitis: a polymicrobial disruption of host homeostasis

Oral biofilms revisited: A novel host tissue of bacteriological origin

The keystone-pathogen hypothesis

Microbial transitions from health to disease

Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis

The dental plaque biofilm matrix

The Relationship of the Oral Microbiotia to Periodontal Health and Disease

Microbial protection and virulence in periodontal tissue as a function of polymicrobial communities: symbiosis and dysbiosis

Low-Abundance Biofilm Species Orchestrates Inflammatory Periodontal Disease through the Commensal Microbiota and Complement

Tetra- and penta-acylated lipid A structures of Porphyromonas gingivalis LPS differentially activate TLR4-mediated NF-κB signal transduction cascade and immuno-inflammatory response in human gingival fibroblasts

Microbial shift and periodontitis

Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation

Maintaining homeostatic control of periodontal epithelial tissue

The oral microbial consortium's interaction with the periodontal innate defense system

3LPS-binding protein and its interactions with P. gingivalis LPS modulate pro-inflammatory response and Toll-like receptor signaling in human oral keratinocytes

Porphyromonas gingivalis induces penetration of lipopolysaccharide and peptidoglycan through the gingival epithelium via degradation of junctional adhesion molecule 1

Defensins and LL-37: a review of function in the gingival epithelium

Gingival Epithelial Cell Recognition of Lipopolysaccharide

Periodontal pathogens promote cancer aggressivity via TLR/MyD88 triggered activation of Integrin/FAK signaling that is therapeutically reversible by a probiotic bacteriocin

Macrophage migration inhibitory factor regulates specific innate immune sensor responses in gingival epithelial cells

A Novel Class of Lipoprotein Lipase-Sensitive Molecules Mediates Toll-Like Receptor 2 Activation by Porphyromonas gingivalis

Genomic and functional characterization of a mucosal symbiont involved in early-stage colorectal cancer

Does Oral Endotoxin Contribute to Systemic Inflammation?

Effects of a stabilized stannous fluoride dentifrice on clinical, immunomodulatory, and microbial outcomes in a human experimental gingivitis model

Toll‐like receptor‐2 and ‐4 responses regulate neutrophil infiltration into the junctional epithelium and significantly contribute to the composition of the oral microbiota

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