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Mucus increases cell iron uptake to impact the release of pro-inflammatory mediators after particle exposure

Scientific reports, 2023-03, Vol.13 (1), p.3925-3925, Article 3925

2023

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Transcriptional Profiling of Mucociliary Differentiation in Human Airway Epithelial Cells

American journal of respiratory cell and molecular biology, 2007-08, Vol.37 (2), p.169-185

2007

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Particulate Matter in Cigarette Smoke Alters Iron Homeostasis to Produce a Biological Effect

American journal of respiratory and critical care medicine, 2008-12, Vol.178 (11), p.1130-1138

2008

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ERK1/2 and p38 regulate inter-individual variability in ozone-mediated IL-8 gene expression in primary human bronchial epithelial cells

Scientific reports, 2018-06, Vol.8 (1), p.9398-11, Article 9398

2018

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Liquid application dosing alters the physiology of air-liquid interface (ALI) primary human bronchial epithelial cell/lung fibroblast co-cultures and in vitro testing relevant endpoints

Frontiers in toxicology, 2023, Vol.5, p.1264331-1264331

2023

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Sequestration of mitochondrial iron by silica particle initiates a biological effect

American journal of physiology. Lung cellular and molecular physiology, 2013-11, Vol.305 (10), p.L712-L724

2013

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Ex Vivo Chemical Cytometric Analysis of Protein Tyrosine Phosphatase Activity in Single Human Airway Epithelial Cells

Analytical chemistry (Washington), 2014-01, Vol.86 (2), p.1291-1297

2014

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Comparison of Gene Expression Profiles Induced By Coarse, Fine, and Ultrafine Particulate Matter

Journal of Toxicology and Environmental Health, Part A, 2011-01, Vol.74 (5), p.296-312

2011

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Air pollutants disrupt iron homeostasis to impact oxidant generation, biological effects, and tissue injury

Free radical biology & medicine, 2020-05, Vol.151, p.38-55

2020

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Up-Regulation of Tissue Factor in Human Pulmonary Artery Endothelial Cells after Ultrafine Particle Exposure

Environmental health perspectives, 2007-04, Vol.115 (4), p.535-540

2007

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Differential expression of pro-inflammatory and oxidative stress mediators induced by nitrogen dioxide and ozone in primary human bronchial epithelial cells

Inhalation toxicology, 2016-07, Vol.28 (8), p.374-382

2016

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Air pollution particles and iron homeostasis

Biochimica et biophysica acta, 2016-12, Vol.1860 (12), p.2816-2825

2016

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Exposure to wood smoke particles produces inflammation in healthy volunteers

Occupational and environmental medicine (London, England), 2012-03, Vol.69 (3), p.170-175

2012

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Ozone Responsive Gene Expression as a Model for Describing Repeat Exposure Response Trajectories and Interindividual Toxicodynamic Variability In Vitro

Toxicological sciences, 2021-12, Vol.185 (1), p.38-49

2021

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Benchmark Dose Modeling Approaches for Volatile Organic Chemicals Using a Novel Air-Liquid Interface In Vitro Exposure System

Toxicological sciences, 2022-06, Vol.188 (1), p.88-107

2022

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Lung injury after ozone exposure is iron dependent

American journal of physiology. Lung cellular and molecular physiology, 2007-01, Vol.292 (1), p.L134-L143

2007

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Seasonal Variations in Air Pollution Particle-Induced Inflammatory Mediator Release and Oxidative Stress

Environmental health perspectives, 2005-08, Vol.113 (8), p.1032-1038

2005

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Ozone Reacts With Carbon Black to Produce a Fulvic Acid-Like Substance and Increase an Inflammatory Effect

Toxicologic pathology, 2020-10, Vol.48 (7), p.887-898

2020

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Ozone induces a proinflammatory response in primary human bronchial epithelial cells through mitogen-activated protein kinase activation without nuclear factor-κB activation

American journal of respiratory cell and molecular biology, 2014-09, Vol.51 (3), p.426-435

2014

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Growth of human bronchial epithelial cells at an air-liquid interface alters the response to particle exposure

Particle and fibre toxicology, 2013-06, Vol.10 (1), p.25-25

2013

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Wood Smoke Particle Sequesters Cell Iron to Impact a Biological Effect

Chemical research in toxicology, 2015-11, Vol.28 (11), p.2104-2111

2015

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A new cell culture exposure system for studying the toxicity of volatile chemicals at the air-liquid interface

Inhalation toxicology, 2018-04, Vol.30 (4-5), p.169-177

2018

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Ambient Particulate Matter Induces Interleukin-8 Expression through an Alternative NF-κB (Nuclear Factor-Kappa B) Mechanism in Human Airway Epithelial Cells

Environmental health perspectives, 2011-10, Vol.119 (10), p.1379-1383

2011

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Lactate Production can Function to Increase Human Epithelial Cell Iron Concentration

Cellular and molecular bioengineering, 2022-12, Vol.15 (6), p.571-585

2022

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Baseline Chromatin Modification Levels May Predict Interindividual Variability in Ozone-Induced Gene Expression

Toxicological sciences, 2016-03, Vol.150 (1), p.216-224

2016

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Mucus increases cell iron uptake to impact the release of pro-inflammatory mediators after particle exposure

Transcriptional Profiling of Mucociliary Differentiation in Human Airway Epithelial Cells

Particulate Matter in Cigarette Smoke Alters Iron Homeostasis to Produce a Biological Effect

ERK1/2 and p38 regulate inter-individual variability in ozone-mediated IL-8 gene expression in primary human bronchial epithelial cells

Liquid application dosing alters the physiology of air-liquid interface (ALI) primary human bronchial epithelial cell/lung fibroblast co-cultures and in vitro testing relevant endpoints

Sequestration of mitochondrial iron by silica particle initiates a biological effect

Ex Vivo Chemical Cytometric Analysis of Protein Tyrosine Phosphatase Activity in Single Human Airway Epithelial Cells

Comparison of Gene Expression Profiles Induced By Coarse, Fine, and Ultrafine Particulate Matter

Air pollutants disrupt iron homeostasis to impact oxidant generation, biological effects, and tissue injury

Up-Regulation of Tissue Factor in Human Pulmonary Artery Endothelial Cells after Ultrafine Particle Exposure

Differential expression of pro-inflammatory and oxidative stress mediators induced by nitrogen dioxide and ozone in primary human bronchial epithelial cells

Air pollution particles and iron homeostasis

Exposure to wood smoke particles produces inflammation in healthy volunteers

Ozone Responsive Gene Expression as a Model for Describing Repeat Exposure Response Trajectories and Interindividual Toxicodynamic Variability In Vitro

Benchmark Dose Modeling Approaches for Volatile Organic Chemicals Using a Novel Air-Liquid Interface In Vitro Exposure System

Lung injury after ozone exposure is iron dependent

Seasonal Variations in Air Pollution Particle-Induced Inflammatory Mediator Release and Oxidative Stress

Ozone Reacts With Carbon Black to Produce a Fulvic Acid-Like Substance and Increase an Inflammatory Effect

Ozone induces a proinflammatory response in primary human bronchial epithelial cells through mitogen-activated protein kinase activation without nuclear factor-κB activation

Growth of human bronchial epithelial cells at an air-liquid interface alters the response to particle exposure

Wood Smoke Particle Sequesters Cell Iron to Impact a Biological Effect

A new cell culture exposure system for studying the toxicity of volatile chemicals at the air-liquid interface

Ambient Particulate Matter Induces Interleukin-8 Expression through an Alternative NF-κB (Nuclear Factor-Kappa B) Mechanism in Human Airway Epithelial Cells

Lactate Production can Function to Increase Human Epithelial Cell Iron Concentration

Baseline Chromatin Modification Levels May Predict Interindividual Variability in Ozone-Induced Gene Expression

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