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β-arrestin-2 is an essential regulator of pancreatic β-cell function under physiological and pathophysiological conditions

Nature communications, 2017-02, Vol.8 (1), p.14295-14295, Article 14295

2017

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Inhibition of ATPase activity of Escherichia coli ATP synthase by polyphenols

International journal of biological macromolecules, 2009-07, Vol.45 (1), p.72-79

2009

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Gi/o protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na+/K+ ATPase activation

Nature communications, 2022-10, Vol.13 (1), p.6461-18, Article 6461

2022

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Neurog3-Independent Methylation Is the Earliest Detectable Mark Distinguishing Pancreatic Progenitor Identity

Developmental cell, 2019-01, Vol.48 (1), p.49-63.e7

2019

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Dietary bioflavonoids inhibit Escherichia coli ATP synthase in a differential manner

International journal of biological macromolecules, 2010-06, Vol.46 (5), p.478-486

2010

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TALK-1-mediated alterations of β-cell mitochondrial function and insulin secretion impair glucose homeostasis on a diabetogenic diet

Cell reports (Cambridge), 2024-01, Vol.43 (1), p.113673-113673, Article 113673

2024

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Osteopontin activates the diabetes-associated potassium channel TALK-1 in pancreatic β-cells

PloS one, 2017-04, Vol.12 (4), p.e0175069-e0175069

2017

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The physiological effects of deleting the mouse SLC30A8 gene encoding zinc transporter-8 are influenced by gender and genetic background

PloS one, 2012-07, Vol.7 (7), p.e40972-e40972

2012

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The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults

eLife, 2024-05, Vol.12

2024

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G6PC2: A Negative Regulator of Basal Glucose-Stimulated Insulin Secretion

Diabetes (New York, N.Y.), 2013-05, Vol.62 (5), p.1547-1556

2013

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Antagonistic epistasis of Hnf4α and FoxO1 metabolic networks through enhancer interactions in β-cell function

Molecular metabolism (Germany), 2021-11, Vol.53, p.101256-101256, Article 101256

2021

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Selective Small Molecule Activators of TREK‑2 Channels Stimulate Dorsal Root Ganglion c‑Fiber Nociceptor Two-Pore-Domain Potassium Channel Currents and Limit Calcium Influx

ACS chemical neuroscience, 2017-03, Vol.8 (3), p.558-568

2017

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A KCNK16 mutation causing TALK-1 gain of function is associated with maturity-onset diabetes of the young

JCI insight, 2021-07, Vol.6 (13)

2021

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TALK-1 reduces delta-cell endoplasmic reticulum and cytoplasmic calcium levels limiting somatostatin secretion

Molecular metabolism (Germany), 2018-03, Vol.9, p.84-97

2018

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Temporal Transcriptome Analysis Reveals Dynamic Gene Expression Patterns Driving β-Cell Maturation

Frontiers in cell and developmental biology, 2021-05, Vol.9, p.648791-648791

2021

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Lactate activation of α-cell KATP channels inhibits glucagon secretion by hyperpolarizing the membrane potential and reducing Ca2+ entry

Molecular metabolism (Germany), 2020-12, Vol.42, p.101056-101056, Article 101056

2020

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Type 2 Diabetes-Associated K+ Channel TALK-1 Modulates β-Cell Electrical Excitability, Second-Phase Insulin Secretion, and Glucose Homeostasis

Diabetes (New York, N.Y.), 2015-11, Vol.64 (11), p.3818-3828

2015

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Synaptotagmin 4 Regulates Pancreatic β Cell Maturation by Modulating the Ca2+ Sensitivity of Insulin Secretion Vesicles

Developmental cell, 2018-05, Vol.45 (3), p.347-361.e5

2018

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Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca2+-Regulated Genes

Diabetes (New York, N.Y.), 2017-08, Vol.66 (8), p.2175-2187

2017

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TASK-1 Potassium Channels Limit Pancreatic α-Cell Calcium Influx and Glucagon Secretion

Molecular endocrinology (Baltimore, Md.), 2015-05, Vol.29 (5), p.777-787

2015

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Pancreatic β-Cell–specific Ablation of TASK-1 Channels Augments Glucose-stimulated Calcium Entry and Insulin Secretion, Improving Glucose Tolerance

Endocrinology (Philadelphia), 2014-10, Vol.155 (10), p.3757-3768

2014

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Tetraspanin‐7 regulation of L‐type voltage‐dependent calcium channels controls pancreatic β‐cell insulin secretion

The Journal of physiology, 2020-11, Vol.598 (21), p.4887-4905

2020

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TRPM7 is a crucial regulator of pancreatic endocrine development and high-fat-diet-induced β-cell proliferation

Development (Cambridge), 2021-08, Vol.148 (16)

2021

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Inhibition of Pancreatic β-Cell Ca2+/Calmodulin-dependent Protein Kinase II Reduces Glucose-stimulated Calcium Influx and Insulin Secretion, Impairing Glucose Tolerance

The Journal of biological chemistry, 2014-05, Vol.289 (18), p.12435-12445

2014

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Liraglutide increases islet Ca2+ oscillation frequency and insulin secretion by activating hyperpolarization‐activated cyclic nucleotide‐gated channels

Diabetes, obesity & metabolism, 2022-09, Vol.24 (9), p.1741-1752

2022

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β-arrestin-2 is an essential regulator of pancreatic β-cell function under physiological and pathophysiological conditions

Inhibition of ATPase activity of Escherichia coli ATP synthase by polyphenols

Gi/o protein-coupled receptor inhibition of beta-cell electrical excitability and insulin secretion depends on Na+/K+ ATPase activation

Neurog3-Independent Methylation Is the Earliest Detectable Mark Distinguishing Pancreatic Progenitor Identity

Dietary bioflavonoids inhibit Escherichia coli ATP synthase in a differential manner

TALK-1-mediated alterations of β-cell mitochondrial function and insulin secretion impair glucose homeostasis on a diabetogenic diet

Osteopontin activates the diabetes-associated potassium channel TALK-1 in pancreatic β-cells

The physiological effects of deleting the mouse SLC30A8 gene encoding zinc transporter-8 are influenced by gender and genetic background

The MODY-associated KCNK16 L114P mutation increases islet glucagon secretion and limits insulin secretion resulting in transient neonatal diabetes and glucose dyshomeostasis in adults

G6PC2: A Negative Regulator of Basal Glucose-Stimulated Insulin Secretion

Antagonistic epistasis of Hnf4α and FoxO1 metabolic networks through enhancer interactions in β-cell function

Selective Small Molecule Activators of TREK‑2 Channels Stimulate Dorsal Root Ganglion c‑Fiber Nociceptor Two-Pore-Domain Potassium Channel Currents and Limit Calcium Influx

A KCNK16 mutation causing TALK-1 gain of function is associated with maturity-onset diabetes of the young

TALK-1 reduces delta-cell endoplasmic reticulum and cytoplasmic calcium levels limiting somatostatin secretion

Temporal Transcriptome Analysis Reveals Dynamic Gene Expression Patterns Driving β-Cell Maturation

Lactate activation of α-cell KATP channels inhibits glucagon secretion by hyperpolarizing the membrane potential and reducing Ca2+ entry

Type 2 Diabetes-Associated K+ Channel TALK-1 Modulates β-Cell Electrical Excitability, Second-Phase Insulin Secretion, and Glucose Homeostasis

Synaptotagmin 4 Regulates Pancreatic β Cell Maturation by Modulating the Ca2+ Sensitivity of Insulin Secretion Vesicles

Chronic β-Cell Depolarization Impairs β-Cell Identity by Disrupting a Network of Ca2+-Regulated Genes

TASK-1 Potassium Channels Limit Pancreatic α-Cell Calcium Influx and Glucagon Secretion

Pancreatic β-Cell–specific Ablation of TASK-1 Channels Augments Glucose-stimulated Calcium Entry and Insulin Secretion, Improving Glucose Tolerance

Tetraspanin‐7 regulation of L‐type voltage‐dependent calcium channels controls pancreatic β‐cell insulin secretion

TRPM7 is a crucial regulator of pancreatic endocrine development and high-fat-diet-induced β-cell proliferation

Inhibition of Pancreatic β-Cell Ca2+/Calmodulin-dependent Protein Kinase II Reduces Glucose-stimulated Calcium Influx and Insulin Secretion, Impairing Glucose Tolerance

Liraglutide increases islet Ca2+ oscillation frequency and insulin secretion by activating hyperpolarization‐activated cyclic nucleotide‐gated channels

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