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The kinase triad, AMPK, mTORC1 and ULK1, maintains energy and nutrient homoeostasis

Biochemical Society transactions, 2013-08, Vol.41 (4), p.939

2013

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Control of TSC2-Rheb signaling axis by arginine regulates mTORC1 activity

eLife, 2016-01, Vol.5

2016

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mTOR Ser-2481 Autophosphorylation Monitors mTORC-specific Catalytic Activity and Clarifies Rapamycin Mechanism of Action

The Journal of biological chemistry, 2010-03, Vol.285 (11), p.7866-7879

2010

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The lysosome: a crucial hub for AMPK and mTORC1 signalling

Biochemical journal, 2017-05, Vol.474 (9), p.1453-1466

2017

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ULK1 inhibits mTORC1 signaling, promotes multisite Raptor phosphorylation and hinders substrate binding

Autophagy, 2011-07, Vol.7 (7), p.737-747

2011

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The tumor suppressor folliculin regulates AMPK-dependent metabolic transformation

The Journal of clinical investigation, 2014-06, Vol.124 (6), p.2640-2650

2014

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Mammalian target of rapamycin complex 1-mediated phosphorylation of eukaryotic initiation factor 4E-binding protein 1 requires multiple protein–protein interactions for substrate recognition

Cellular signalling, 2009-07, Vol.21 (7), p.1073-1084

2009

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Tuberous Sclerosis Complex cell‐derived EVs have an altered protein cargo capable of regulating their microenvironment and have potential as disease biomarkers

Journal of extracellular vesicles, 2023-06, Vol.12 (6), p.e12336-n/a

2023

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Energy Stress-Mediated Cytotoxicity in Tuberous Sclerosis Complex 2-Deficient Cells with Nelfinavir and Mefloquine Treatment

Cancers, 2018-10, Vol.10 (10), p.375

2018

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A tuberous sclerosis complex signalling node at the peroxisome regulates mTORC1 and autophagy in response to ROS

Nature cell biology, 2013-10, Vol.15 (10), p.1186

2013

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Tuberous sclerosis—A model for tumour growth

Seminars in cell & developmental biology, 2016-04, Vol.52, p.3-11

2016

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Targeting protein homeostasis with nelfinavir/salinomycin dual therapy effectively induces death of mTORC1 hyperactive cells

Oncotarget, 2017-07, Vol.8 (30), p.48711-48724

2017

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FLCN, a novel autophagy component, interacts with GABARAP and is regulated by ULK1 phosphorylation

Autophagy, 2014-10, Vol.10 (10), p.1749-1760

2014

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Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir-bortezomib therapy to intensify endoplasmic reticulum stress-induced cell death

Oncogene, 2018-11, Vol.37 (45), p.5913-5925

2018

Volltextzugriff (PDF)

Correction: Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir-bortezomib therapy to intensify endoplasmic reticulum stress-induced cell death

Oncogene, 2019-04, Vol.38 (16), p.3102-3102

2019

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Erythropoietin-induced activation of the JAK2/STAT5, PI3K/Akt, and Ras/ERK pathways promotes malignant cell behavior in a modified breast cancer cell line

Molecular cancer research, 2010-04, Vol.8 (4), p.615-626

2010

Volltextzugriff (PDF)

Correction: Control of TSC2-Rheb signaling axis by arginine regulates mTORC1 activity

eLife, 2020-12, Vol.9

2020

Volltextzugriff (PDF)

Erythropoietin receptor expression in non-small cell lung carcinoma: a question of antibody specificity

Stem cells (Dayton, Ohio), 2007-03, Vol.25 (3), p.718-722

2007

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Determining the pathogenicity of patient-derived TSC2 mutations by functional characterization and clinical evidence

European journal of human genetics : EJHG, 2011-07, Vol.19 (7), p.789-795

2011

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Impaired Downregulation Following Erythropoietin Receptor Activation in Non‐Small Cell Lung Carcinoma

Stem cells (Dayton, Ohio), 2007-02, Vol.25 (2), p.380-384

2007

Volltextzugriff (PDF)

Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir,eAEbortezomib therapy to intensify endoplasmic reticulum stress-induced cell death

Oncogene, 2018-11, Vol.37 (45), p.5913

2018

Volltextzugriff (PDF)

Determining the pathogenicity of patient-derived mutations by functional characterization and clinical evidence

European journal of human genetics : EJHG, 2011-07, Vol.19 (7), p.789-795

2011

Volltextzugriff (PDF)

Complete loss of ATM function augments replication catastrophe induced by ATR inhibition and gemcitabine in pancreatic cancer models

British journal of cancer, 2020-10, Vol.123 (9), p.1424-1436

2020

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Inflammatory bowel disease associates with proinflammatory potential of the immunoglobulin G glycome

Inflammatory bowel diseases, 2015-06, Vol.21 (6), p.1237-1247

2015

Volltextzugriff (PDF)

Antibody Status and Incidence of SARS-CoV-2 Infection in Health Care Workers

The New England journal of medicine, 2021-02, Vol.384 (6), p.533-540

2021

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The kinase triad, AMPK, mTORC1 and ULK1, maintains energy and nutrient homoeostasis

Control of TSC2-Rheb signaling axis by arginine regulates mTORC1 activity

mTOR Ser-2481 Autophosphorylation Monitors mTORC-specific Catalytic Activity and Clarifies Rapamycin Mechanism of Action

The lysosome: a crucial hub for AMPK and mTORC1 signalling

ULK1 inhibits mTORC1 signaling, promotes multisite Raptor phosphorylation and hinders substrate binding

The tumor suppressor folliculin regulates AMPK-dependent metabolic transformation

Mammalian target of rapamycin complex 1-mediated phosphorylation of eukaryotic initiation factor 4E-binding protein 1 requires multiple protein–protein interactions for substrate recognition

Tuberous Sclerosis Complex cell‐derived EVs have an altered protein cargo capable of regulating their microenvironment and have potential as disease biomarkers

Energy Stress-Mediated Cytotoxicity in Tuberous Sclerosis Complex 2-Deficient Cells with Nelfinavir and Mefloquine Treatment

A tuberous sclerosis complex signalling node at the peroxisome regulates mTORC1 and autophagy in response to ROS

Tuberous sclerosis—A model for tumour growth

Targeting protein homeostasis with nelfinavir/salinomycin dual therapy effectively induces death of mTORC1 hyperactive cells

FLCN, a novel autophagy component, interacts with GABARAP and is regulated by ULK1 phosphorylation

Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir-bortezomib therapy to intensify endoplasmic reticulum stress-induced cell death

Correction: Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir-bortezomib therapy to intensify endoplasmic reticulum stress-induced cell death

Erythropoietin-induced activation of the JAK2/STAT5, PI3K/Akt, and Ras/ERK pathways promotes malignant cell behavior in a modified breast cancer cell line

Correction: Control of TSC2-Rheb signaling axis by arginine regulates mTORC1 activity

Erythropoietin receptor expression in non-small cell lung carcinoma: a question of antibody specificity

Determining the pathogenicity of patient-derived TSC2 mutations by functional characterization and clinical evidence

Impaired Downregulation Following Erythropoietin Receptor Activation in Non‐Small Cell Lung Carcinoma

Loss of tuberous sclerosis complex 2 sensitizes tumors to nelfinavir,eAEbortezomib therapy to intensify endoplasmic reticulum stress-induced cell death

Determining the pathogenicity of patient-derived mutations by functional characterization and clinical evidence

Complete loss of ATM function augments replication catastrophe induced by ATR inhibition and gemcitabine in pancreatic cancer models

Inflammatory bowel disease associates with proinflammatory potential of the immunoglobulin G glycome

Antibody Status and Incidence of SARS-CoV-2 Infection in Health Care Workers

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