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Genome-wide profiling of genetic alterations in acute lymphoblastic leukemia: recent insights and future directions

Leukemia, 2009-07, Vol.23 (7), p.1209-1218

2009

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Long-term results of St Jude Total Therapy Studies 11, 12, 13A, 13B, and 14 for childhood acute lymphoblastic leukemia

Leukemia, 2010-02, Vol.24 (2), p.371-382

2010

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Identification of acquired copy number alterations and uniparental disomies in cytogenetically normal acute myeloid leukemia using high-resolution single-nucleotide polymorphism analysis

Leukemia, 2010-02, Vol.24 (2), p.438-449

2010

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Molecular Classification of Cancer: Class Discovery and Class Prediction by Gene Expression Monitoring

Science (American Association for the Advancement of Science), 1999-10, Vol.286 (5439), p.531-537

1999

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IDH1 and IDH2 mutations in pediatric acute leukemia

Leukemia, 2011-10, Vol.25 (10), p.1570-1577

2011

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AMKL chimeric transcription factors are potent inducers of leukemia

Leukemia, 2017-10, Vol.31 (10), p.2228-2234

2017

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A revised definition for cure of childhood acute lymphoblastic leukemia

Leukemia, 2014-12, Vol.28 (12), p.2336-2343

2014

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Increased risk for CNS relapse in pre-B cell leukemia with the t(1;19)/TCF3-PBX1

Leukemia, 2009-08, Vol.23 (8), p.1406-1409

2009

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Comparison of genome sequencing and clinical genotyping for pharmacogenes

Clinical pharmacology and therapeutics, 2016-10, Vol.100 (4), p.380-388

2016

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Pediatric acute myeloid leukemia with NPM1 mutations is characterized by a gene expression profile with dysregulated HOX gene expression distinct from MLL-rearranged leukemias

Leukemia, 2007-09, Vol.21 (9), p.2000-2009

2007

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Impact of genetic variation in FKBP5 on clinical response in pediatric acute myeloid leukemia patients: a pilot study

Leukemia, 2011-08, Vol.25 (8), p.1354-1356

2011

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Shared acquired genomic changes in zebrafish and human T-ALL

Oncogene, 2011-10, Vol.30 (41), p.4289-4296

2011

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MN1 overexpression is an important step in the development of inv(16) AML

Leukemia, 2007-08, Vol.21 (8), p.1679-1690

2007

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Combination of cladribine and cytarabine is effective for childhood acute myeloid leukemia: results of the St Jude AML97 trial

Leukemia, 2009-08, Vol.23 (8), p.1410-1416

2009

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Clinical impact of minimal residual disease in children with different subtypes of acute lymphoblastic leukemia treated with Response-Adapted therapy

Leukemia, 2017-02, Vol.31 (2), p.333-339

2017

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THE AML1‐ETO CHIMAERIC TRANSCRIPTION FACTOR IN ACUTE MYELOID LEUKAEMIA: BIOLOGY AND CLINICAL SIGNIFICANCE

British journal of haematology, 1999-08, Vol.106 (2), p.296-308

Downing, James R.

1999

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Genome-wide single-nucleotide polymorphism analysis in juvenile myelomonocytic leukemia identifies uniparental disomy surrounding the NF1 locus in cases associated with neurofibromatosis but not in cases with mutant RAS or PTPN11

Oncogene, 2007-08, Vol.26 (39), p.5816-5821

2007

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Acute Myeloid Leukemia

The New England journal of medicine, 1999-09, Vol.341 (14), p.1051-1062

1999

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Identification of AML-1 and the (8;21) translocation protein (AML-1/ETO) as sequence-specific DNA-binding proteins: the runt homology domain is required for DNA binding and protein-protein interactions

Molecular and Cellular Biology, 1993-10, Vol.13 (10), p.6336-6345

1993

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TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis

Leukemia, 1995-12, Vol.9 (12), p.1985-1989

1995

Volltextzugriff (PDF)

3D field data of flow structures in dead-ended, coolant loop reactor branch lines for CFD validation of thermal fatigue onset modeling

Nuclear engineering and design, 2022-12, Vol.399, p.112012, Article 112012

2022

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TEL gene rearrangement in acute lymphoblastic leukemia: a new genetic marker with prognostic significance

Journal of clinical oncology, 1997-03, Vol.15 (3), p.1150-1157

1997

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Rearrangement of the MLL gene confers a poor prognosis in childhood acute lymphoblastic leukemia, regardless of presenting age

Blood, 1996-04, Vol.87 (7), p.2870-2877

1996

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The role of TEL fusion genes in pediatric leukemias

Leukemia, 1999, Vol.13 (1), p.6-13

1999

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Genome-wide approach to identify risk factors for therapy-related myeloid leukemia

Leukemia, 2006-02, Vol.20 (2), p.239-246

2006

Volltextzugriff (PDF)

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Genome-wide profiling of genetic alterations in acute lymphoblastic leukemia: recent insights and future directions

Long-term results of St Jude Total Therapy Studies 11, 12, 13A, 13B, and 14 for childhood acute lymphoblastic leukemia

Identification of acquired copy number alterations and uniparental disomies in cytogenetically normal acute myeloid leukemia using high-resolution single-nucleotide polymorphism analysis

Molecular Classification of Cancer: Class Discovery and Class Prediction by Gene Expression Monitoring

IDH1 and IDH2 mutations in pediatric acute leukemia

AMKL chimeric transcription factors are potent inducers of leukemia

A revised definition for cure of childhood acute lymphoblastic leukemia

Increased risk for CNS relapse in pre-B cell leukemia with the t(1;19)/TCF3-PBX1

Comparison of genome sequencing and clinical genotyping for pharmacogenes

Pediatric acute myeloid leukemia with NPM1 mutations is characterized by a gene expression profile with dysregulated HOX gene expression distinct from MLL-rearranged leukemias

Impact of genetic variation in FKBP5 on clinical response in pediatric acute myeloid leukemia patients: a pilot study

Shared acquired genomic changes in zebrafish and human T-ALL

MN1 overexpression is an important step in the development of inv(16) AML

Combination of cladribine and cytarabine is effective for childhood acute myeloid leukemia: results of the St Jude AML97 trial

Clinical impact of minimal residual disease in children with different subtypes of acute lymphoblastic leukemia treated with Response-Adapted therapy

THE AML1‐ETO CHIMAERIC TRANSCRIPTION FACTOR IN ACUTE MYELOID LEUKAEMIA: BIOLOGY AND CLINICAL SIGNIFICANCE

Genome-wide single-nucleotide polymorphism analysis in juvenile myelomonocytic leukemia identifies uniparental disomy surrounding the NF1 locus in cases associated with neurofibromatosis but not in cases with mutant RAS or PTPN11

Acute Myeloid Leukemia

Identification of AML-1 and the (8;21) translocation protein (AML-1/ETO) as sequence-specific DNA-binding proteins: the runt homology domain is required for DNA binding and protein-protein interactions

TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis

3D field data of flow structures in dead-ended, coolant loop reactor branch lines for CFD validation of thermal fatigue onset modeling

TEL gene rearrangement in acute lymphoblastic leukemia: a new genetic marker with prognostic significance

Rearrangement of the MLL gene confers a poor prognosis in childhood acute lymphoblastic leukemia, regardless of presenting age

The role of TEL fusion genes in pediatric leukemias

Genome-wide approach to identify risk factors for therapy-related myeloid leukemia

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