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The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction

Journal of molecular and cellular cardiology, 2010-03, Vol.48 (3), p.504-511

2010

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Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction

Circulation research, 2010-08, Vol.107 (3), p.418-428

2010

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Interleukin-1 Receptor Type I Signaling Critically Regulates Infarct Healing and Cardiac Remodeling

The American journal of pathology, 2008-07, Vol.173 (1), p.57-67

2008

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Systematic Characterization of Myocardial Inflammation, Repair, and Remodeling in a Mouse Model of Reperfused Myocardial Infarction

The journal of histochemistry and cytochemistry, 2013-08, Vol.61 (8), p.555-570

2013

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Essential role of smad3 in infarct healing and in the pathogenesis of cardiac remodeling

Circulation (New York, N.Y.), 2007-11, Vol.116 (19), p.2127-2138

2007

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CXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycans

Cardiovascular research, 2014-07, Vol.103 (2), p.217-227

2014

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Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice

Circulation. Heart failure, 2015-07, Vol.8 (4), p.788-798

2015

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Endogenous Thrombospondin 1 Protects the Pressure-Overloaded Myocardium by Modulating Fibroblast Phenotype and Matrix Metabolism

Hypertension (Dallas, Tex. 1979), 2011-11, Vol.58 (5), p.902-911

2011

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The Extracellular Matrix Modulates Fibroblast Phenotype and Function in the Infarcted Myocardium

Journal of cardiovascular translational research, 2012-12, Vol.5 (6), p.837-847

2012

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Transforming growth factor (TGF)-β signaling in cardiac remodeling

Journal of molecular and cellular cardiology, 2011-10, Vol.51 (4), p.600-606

2011

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TGF-β signaling in fibrosis

Growth factors (Chur, Switzerland), 2011-10, Vol.29 (5), p.196-202

2011

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β-Resorcylidene aminoguanidine (RAG) dilates coronary arteries in an endothelium-independent manner

Pharmacological reports, 2015-06, Vol.67 (3), p.631-635

2015

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Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function

American journal of physiology. Heart and circulatory physiology, 2014-10, Vol.307 (8), p.H1233-H1242

2014

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Anti-diabetic effects of 1-methylnicotinamide (MNA) in streptozocin-induced diabetes in rats

Pharmacological reports, 2009-01, Vol.61 (1), p.86-98

2009

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CCR5 Signaling Suppresses Inflammation and Reduces Adverse Remodeling of the Infarcted Heart, Mediating Recruitment of Regulatory T Cells

The American journal of pathology, 2010-05, Vol.176 (5), p.2177-2187

2010

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Thrombospondin-1 Induction in the Diabetic Myocardium Stabilizes the Cardiac Matrix in Addition to Promoting Vascular Rarefaction Through Angiopoietin-2 Upregulation

Circulation research, 2013-12, Vol.113 (12), p.1331-1344

2013

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Induction of the CXC Chemokine Interferon-γ–Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction

Circulation research, 2009-11, Vol.105 (10), p.973-983

2009

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Endogenous IRAK-M attenuates postinfarction remodeling through effects on macrophages and fibroblasts

Arteriosclerosis, thrombosis, and vascular biology, 2012-11, Vol.32 (11), p.2598-2608

2012

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Tissue transglutaminase induction in the pressure-overloaded myocardium regulates matrix remodelling

Cardiovascular research, 2017-07, Vol.113 (8), p.892-905

2017

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Abstract 5055: CC Chemokine Receptor 5 Signaling Suppresses Inflammation and Attenuates Adverse Remodeling Following Myocardial Infarction by Recruiting Regulatory T Cells

Circulation (New York, N.Y.), 2009-11, Vol.120 (suppl_18)

2009

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Endogenous tissue transglutaminase critically regulates cardiac remodeling through distinct enzymatic and non‐enzymatic actions

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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The cellular specificity of leptin-mediated actions in the infarcted heart

Cardiovascular research, 2011-01, Vol.89 (1), p.9-11

2011

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Abstract 17049: Upregulation of the Matricellular Protein Thrombospondin (TSP)-1 in the db/db Heart Regulates Matrix Remodeling and Induces Vascular Rarefaction

Circulation (New York, N.Y.), 2011-11, Vol.124 (suppl_21)

2011

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Thrombospondin-1 induction in the diabetic myocardium stabilizes the cardiac matrix, while promoting vascular rarefaction through angiopoietin-2 upregulation

Circulation research, 2013-09, Vol.113 (12), p.1331-1344

2013

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Abstract 16644: TGF-β/Smad3 Activation in the Diabetic Mouse Heart Induces Fibrotic Cardiac Remodeling and Diastolic Dysfunction While Protecting From Excessive Matrix Degradation and Preventing the Development of Systolic Dysfunction

Circulation (New York, N.Y.), 2011-11, Vol.124 (suppl_21)

2011

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The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction

Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction

Interleukin-1 Receptor Type I Signaling Critically Regulates Infarct Healing and Cardiac Remodeling

Systematic Characterization of Myocardial Inflammation, Repair, and Remodeling in a Mouse Model of Reperfused Myocardial Infarction

Essential role of smad3 in infarct healing and in the pathogenesis of cardiac remodeling

CXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycans

Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice

Endogenous Thrombospondin 1 Protects the Pressure-Overloaded Myocardium by Modulating Fibroblast Phenotype and Matrix Metabolism

The Extracellular Matrix Modulates Fibroblast Phenotype and Function in the Infarcted Myocardium

Transforming growth factor (TGF)-β signaling in cardiac remodeling

TGF-β signaling in fibrosis

β-Resorcylidene aminoguanidine (RAG) dilates coronary arteries in an endothelium-independent manner

Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function

Anti-diabetic effects of 1-methylnicotinamide (MNA) in streptozocin-induced diabetes in rats

CCR5 Signaling Suppresses Inflammation and Reduces Adverse Remodeling of the Infarcted Heart, Mediating Recruitment of Regulatory T Cells

Thrombospondin-1 Induction in the Diabetic Myocardium Stabilizes the Cardiac Matrix in Addition to Promoting Vascular Rarefaction Through Angiopoietin-2 Upregulation

Induction of the CXC Chemokine Interferon-γ–Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction

Endogenous IRAK-M attenuates postinfarction remodeling through effects on macrophages and fibroblasts

Tissue transglutaminase induction in the pressure-overloaded myocardium regulates matrix remodelling

Abstract 5055: CC Chemokine Receptor 5 Signaling Suppresses Inflammation and Attenuates Adverse Remodeling Following Myocardial Infarction by Recruiting Regulatory T Cells

Endogenous tissue transglutaminase critically regulates cardiac remodeling through distinct enzymatic and non‐enzymatic actions

The cellular specificity of leptin-mediated actions in the infarcted heart

Abstract 17049: Upregulation of the Matricellular Protein Thrombospondin (TSP)-1 in the db/db Heart Regulates Matrix Remodeling and Induces Vascular Rarefaction

Thrombospondin-1 induction in the diabetic myocardium stabilizes the cardiac matrix, while promoting vascular rarefaction through angiopoietin-2 upregulation

Abstract 16644: TGF-β/Smad3 Activation in the Diabetic Mouse Heart Induces Fibrotic Cardiac Remodeling and Diastolic Dysfunction While Protecting From Excessive Matrix Degradation and Preventing the Development of Systolic Dysfunction

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