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The effects of NSAID on the matrix of human articular cartilages

Zeitschrift für Rheumatologie, 1999-06, Vol.58 (3), p.125-129

DINGLE, J. T

1999

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The effect of nonsteroidal antiinflammatory drugs on human articular cartilage glycosaminoglycan synthesis

Osteoarthritis and cartilage, 1999-05, Vol.7 (3), p.313-314

Dingle, J.T.

1999

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Cartilage maintenance in osteoarthritis: interaction of cytokines, NSAID and prostaglandins in articular cartilage damage and repair

Journal of rheumatology. Supplement, 1991-03, Vol.28, p.30

Dingle, J T

1991

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In vivo studies of cartilage regeneration after damage induced by catabolin/interleukin-1

Annals of the rheumatic diseases, 1991-02, Vol.50 (2), p.75-80

1991

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In vivo studies of articular tissue damage mediated by catabolin/interleukin 1

Annals of the rheumatic diseases, 1987-07, Vol.46 (7), p.527-533

1987

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Characterization of a specific antiserum for mammalian collagenase from several species: immunolocalization of collagenase in rabbit chondrocytes and uterus

Journal of cell science, 1986-03, Vol.81 (1), p.105-123

1986

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Effect of tenidap on cartilage integrity in vitro

Annals of the rheumatic diseases, 1993-04, Vol.52 (4), p.292-299

1993

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The sensitivity of synthesis of human cartilage matrix to inhibition by IL-1 suggests a mechanism for the development of osteoarthritis

Cell biochemistry and function, 1991-04, Vol.9 (2), p.99-102

1991

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Metalloproteinase production by rabbit articular cartilage : comparison of the effects of interleukin-1α in vitro and in vivo

Virchows Archiv : an international journal of pathology, 1994-11, Vol.425 (4), p.413-424

1994

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Human mononuclear cell factors mediate cartilage matrix degradation through chondrocyte activation

The Journal of clinical investigation, 1981-09, Vol.68 (3), p.571-581

1981

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The role of cellular interactions in joint erosions

Clinical orthopaedics and related research, 1984, Vol.182 (182), p.24-30

DINGLE, J. T

1984

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Clearance of proteoglycan from joint cavities

Annals of the rheumatic diseases, 1987-12, Vol.46 (12), p.934-937

1987

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cartilage catabolic factor from synovium

Biochemical journal, 1979-10, Vol.184 (1), p.177-180

1979

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Expression of Intercellular Adhesion Molecule 1 (Icam-1) on Human Articular Cartilage Chondrocytes

Connective tissue research, 1991, Vol.26 (3), p.207-216

1991

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Catabolin--a cartilage catabolic factor from synovium

Clinical orthopaedics and related research, 1981-05, Vol.156 (156), p.219-231

Dingle, J T

1981

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The role of catabolin in the control of cartilage matrix integrity

Journal of rheumatology. Supplement, 1983-12, Vol.11, p.38

Dingle, J T

1983

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Articular damage in arthritis and its control

Annals of internal medicine, 1978-06, Vol.88 (6), p.821

Dingle, J T

1978

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The site of cartilage matrix degradation

Biochemical journal, 1980-08, Vol.190 (2), p.431-438

1980

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Production of cartilage degrading activity by human synovial tissues

Annals of the rheumatic diseases, 1987-04, Vol.46 (4), p.319-323

1987

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Studies on the mode of action of excess of vitamin A. 3. Release of a bound protease by the action of vitamin A

Biochemical journal, 1961-06, Vol.79 (3), p.509-512

DINGLE, J T

1961

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Porcine Heart Valves Produce a Protein That Induces Cell-Mediated Connective Tissue Degradation: I. Biological Properties

Circulation research, 1986-09, Vol.59 (3), p.321-328

1986

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Porcine Heart Valves Produce a Protein that Induces Cell-Mediated Connective Tissue Degradation: II. Biochemical Properties of the Partially Purified Protein

Circulation research, 1986-09, Vol.59 (3), p.329-341

1986

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Immunorecognition of chondrocytes in articular cartilage activated by synovial interleukin 1

Connective tissue research, 1991, Vol.25 (3-4), p.243-249

1991

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The secretion of enzymes into the pericellular environment

Philosophical transactions of the Royal Society of London. Series B, Biological sciences, 1975-07, Vol.271 (912), p.315-324

Dingle, J T

1975

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Novel treatment for joint inflammation

Nature (London), 1978-01, Vol.271 (5643), p.372-373

1978

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The effects of NSAID on the matrix of human articular cartilages

The effect of nonsteroidal antiinflammatory drugs on human articular cartilage glycosaminoglycan synthesis

Cartilage maintenance in osteoarthritis: interaction of cytokines, NSAID and prostaglandins in articular cartilage damage and repair

In vivo studies of cartilage regeneration after damage induced by catabolin/interleukin-1

In vivo studies of articular tissue damage mediated by catabolin/interleukin 1

Characterization of a specific antiserum for mammalian collagenase from several species: immunolocalization of collagenase in rabbit chondrocytes and uterus

Effect of tenidap on cartilage integrity in vitro

The sensitivity of synthesis of human cartilage matrix to inhibition by IL-1 suggests a mechanism for the development of osteoarthritis

Metalloproteinase production by rabbit articular cartilage : comparison of the effects of interleukin-1α in vitro and in vivo

Human mononuclear cell factors mediate cartilage matrix degradation through chondrocyte activation

The role of cellular interactions in joint erosions

Clearance of proteoglycan from joint cavities

cartilage catabolic factor from synovium

Expression of Intercellular Adhesion Molecule 1 (Icam-1) on Human Articular Cartilage Chondrocytes

Catabolin--a cartilage catabolic factor from synovium

The role of catabolin in the control of cartilage matrix integrity

Articular damage in arthritis and its control

The site of cartilage matrix degradation

Production of cartilage degrading activity by human synovial tissues

Studies on the mode of action of excess of vitamin A. 3. Release of a bound protease by the action of vitamin A

Porcine Heart Valves Produce a Protein That Induces Cell-Mediated Connective Tissue Degradation: I. Biological Properties

Porcine Heart Valves Produce a Protein that Induces Cell-Mediated Connective Tissue Degradation: II. Biochemical Properties of the Partially Purified Protein

Immunorecognition of chondrocytes in articular cartilage activated by synovial interleukin 1

The secretion of enzymes into the pericellular environment

Novel treatment for joint inflammation

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