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A Positive Feedback Loop Regulates the Expression of Polycomb Group Protein BMI1 via WNT Signaling Pathway

The Journal of biological chemistry, 2013-02, Vol.288 (5), p.3406-3418

2013

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A miR-200c/141-BMI1 autoregulatory loop regulates oncogenic activity of BMI1 in cancer cells

Oncotarget, 2016-06, Vol.7 (24), p.36220-36234

2016

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Oxidative stress, cellular senescence and ageing

AIMS molecular science, 2016-01, Vol.3 (3), p.300-324

2016

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CBX7 regulates stem cell-like properties of gastric cancer cells via p16 and AKT-NF-κB-miR-21 pathways

Journal of hematology and oncology, 2018-02, Vol.11 (1), p.17-17, Article 17

2018

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Dietary omega-3 polyunsaturated fatty acids suppress expression of EZH2 in breast cancer cells

Carcinogenesis (New York), 2010-03, Vol.31 (3), p.489-495

2010

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Mel-18, a polycomb group protein, regulates cell proliferation and senescence via transcriptional repression of Bmi-1 and c-Myc oncoproteins

Molecular biology of the cell, 2007-02, Vol.18 (2), p.536-546

2007

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The polycomb group protein BMI1 is a transcriptional target of HDAC inhibitors

Cell cycle (Georgetown, Tex.), 2010-07, Vol.9 (13), p.2663-2673

2010

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Bmi-1 is a novel molecular marker of nasopharyngeal carcinoma progression and immortalizes primary human nasopharyngeal epithelial cells

Cancer research (Chicago, Ill.), 2006-06, Vol.66 (12), p.6225-6232

2006

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BMI1 and Mel-18 oppositely regulate carcinogenesis and progression of gastric cancer

Molecular cancer, 2010-02, Vol.9 (1), p.40-40, Article 40

2010

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Mechanisms of cellular senescence in human and mouse cells

Biogerontology (Dordrecht), 2004-01, Vol.5 (1), p.1-10

2004

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Methods to detect biomarkers of cellular senescence: the senescence-associated beta-galactosidase assay

Methods in molecular biology (Clifton, N.J.), 2007, Vol.371, p.21

2007

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Colorimetric detection of senescence-associated β galactosidase

Methods in molecular biology (Clifton, N.J.), 2013, Vol.965, p.143

2013

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Control of the Replicative Life Span of Human Fibroblasts by p16 and the Polycomb Protein Bmi-1

Molecular and Cellular Biology, 2003-01, Vol.23 (1), p.389-401

2003

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βTrCP regulates BMI1 protein turnover via ubiquitination and degradation

Cell cycle (Georgetown, Tex.), 2011-04, Vol.10 (8), p.1322-1330

2011

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MicroRNA-31 Is a Transcriptional Target of Histone Deacetylase Inhibitors and a Regulator of Cellular Senescence

The Journal of biological chemistry, 2015-04, Vol.290 (16), p.10555-10567

2015

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Mel-18 acts as a tumor suppressor by repressing bmi-1 expression and down-regulating akt activity in breast cancer cells

Cancer research (Chicago, Ill.), 2007-06, Vol.67 (11), p.5083-5089

2007

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What has senescence got to do with cancer?

Cancer cell, 2005-06, Vol.7 (6), p.505-512

Dimri, Goberdhan P.

2005

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FoxM1c Counteracts Oxidative Stress-induced Senescence and Stimulates Bmi-1 Expression

The Journal of biological chemistry, 2008-06, Vol.283 (24), p.16545-16553

2008

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Bmi1 Functions as an Oncogene Independent of Ink4A/Arf Repression in Hepatic Carcinogenesis

Molecular cancer research, 2009-12, Vol.7 (12), p.1937-1945

2009

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Expression of Bmi-1 in Epidermis Enhances Cell Survival by Altering Cell Cycle Regulatory Protein Expression and Inhibiting Apoptosis

Journal of investigative dermatology, 2008-01, Vol.128 (1), p.9-17

2008

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Deletion analysis of BMI1 oncoprotein identifies its negative regulatory domain

Molecular cancer, 2010-06, Vol.9 (1), p.158-158, Article 158

2010

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pRb or its cousins: Who controls the family business?

Cell cycle (Georgetown, Tex.), 2012-04, Vol.11 (8), p.1486-1486

2012

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The original colorimetric method to detect cellular senescence

Cellular Senescence and Aging, 2024, Vol.181, p.59-72

2024

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Timosaponin A‐III inhibits oncogenic phenotype via regulation of PcG protein BMI1 in breast cancer cells

Molecular carcinogenesis, 2018-07, Vol.57 (7), p.831-841

2018

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PLK1 Inhibition Down-regulates Polycomb Group Protein BMI1 via Modulation of the miR-200c/141 Cluster

The Journal of biological chemistry, 2015-01, Vol.290 (5), p.3033-3044

2015

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A Positive Feedback Loop Regulates the Expression of Polycomb Group Protein BMI1 via WNT Signaling Pathway

A miR-200c/141-BMI1 autoregulatory loop regulates oncogenic activity of BMI1 in cancer cells

Oxidative stress, cellular senescence and ageing

CBX7 regulates stem cell-like properties of gastric cancer cells via p16 and AKT-NF-κB-miR-21 pathways

Dietary omega-3 polyunsaturated fatty acids suppress expression of EZH2 in breast cancer cells

Mel-18, a polycomb group protein, regulates cell proliferation and senescence via transcriptional repression of Bmi-1 and c-Myc oncoproteins

The polycomb group protein BMI1 is a transcriptional target of HDAC inhibitors

Bmi-1 is a novel molecular marker of nasopharyngeal carcinoma progression and immortalizes primary human nasopharyngeal epithelial cells

BMI1 and Mel-18 oppositely regulate carcinogenesis and progression of gastric cancer

Mechanisms of cellular senescence in human and mouse cells

Methods to detect biomarkers of cellular senescence: the senescence-associated beta-galactosidase assay

Colorimetric detection of senescence-associated β galactosidase

Control of the Replicative Life Span of Human Fibroblasts by p16 and the Polycomb Protein Bmi-1

βTrCP regulates BMI1 protein turnover via ubiquitination and degradation

MicroRNA-31 Is a Transcriptional Target of Histone Deacetylase Inhibitors and a Regulator of Cellular Senescence

Mel-18 acts as a tumor suppressor by repressing bmi-1 expression and down-regulating akt activity in breast cancer cells

What has senescence got to do with cancer?

FoxM1c Counteracts Oxidative Stress-induced Senescence and Stimulates Bmi-1 Expression

Bmi1 Functions as an Oncogene Independent of Ink4A/Arf Repression in Hepatic Carcinogenesis

Expression of Bmi-1 in Epidermis Enhances Cell Survival by Altering Cell Cycle Regulatory Protein Expression and Inhibiting Apoptosis

Deletion analysis of BMI1 oncoprotein identifies its negative regulatory domain

pRb or its cousins: Who controls the family business?

The original colorimetric method to detect cellular senescence

Timosaponin A‐III inhibits oncogenic phenotype via regulation of PcG protein BMI1 in breast cancer cells

PLK1 Inhibition Down-regulates Polycomb Group Protein BMI1 via Modulation of the miR-200c/141 Cluster

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