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Targeting of Lung Cancer Mutational Hotspots by Polycyclic Aromatic Hydrocarbons

JNCI : Journal of the National Cancer Institute, 2000-05, Vol.92 (10), p.803-811

2000

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Preferential Formation of Benzo[a]pyrene Adducts at Lung Cancer Mutational Hotspots in P53

Science (American Association for the Advancement of Science), 1996-10, Vol.274 (5286), p.430-432

1996

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Sunlight induces pyrimidine dimers preferentially at 5-methylcytosine bases

Cancer research (Chicago, Ill.), 1997-11, Vol.57 (21), p.4727-4730

1997

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Tobacco smoke carcinogens, DNA damage and p53 mutations in smoking-associated cancers

Oncogene, 2002-10, Vol.21 (48), p.7435-7451

2002

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Slow repair of bulky DNA adducts along the nontranscribed strand of the human p53 gene may explain the strand bias of transversion mutations in cancers

Oncogene, 1998-03, Vol.16 (10), p.1241-1247

1998

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Mutation hotspots and DNA methylation

Current topics in microbiology and immunology, 2000, Vol.249, p.1

2000

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Cytosine Methylation Determines Hot Spots of DNA Damage in the Human P53 Gene

Proceedings of the National Academy of Sciences - PNAS, 1997-04, Vol.94 (8), p.3893-3898

1997

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Formation and repair of DNA lesions in the p53 gene: Relation to cancer mutations?

Environmental and molecular mutagenesis, 1998, Vol.31 (3), p.197-205

1998

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A single nucleotide polymorphism based approach for the identification and characterization of gene expression modulation using MassARRAY

Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis, 2005-06, Vol.573 (1), p.83-95

2005

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Expression of base excision DNA repair genes is a sensitive biomarker for in vivo detection of chemical-induced chronic oxidative stress: Identification of the molecular source of radicals responsible for DNA damage by peroxisome proliferators

Cancer research (Chicago, Ill.), 2004-02, Vol.64 (3), p.1050-1057

2004

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Use of UvrABC nuclease to quantify benzo[a]pyrene diol epoxide–DNA adduct formation at methylated versus unmethylated CpG sites in the p53 gene

Carcinogenesis (New York), 1999-06, Vol.20 (6), p.1085-1089

1999

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The p53 codon 249 mutational hotspot in hepatocellular carcinoma is not related to selective formation or persistence of aflatoxin B1 adducts

Oncogene, 1998-12, Vol.17 (23), p.3007-3014

1998

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Analysis of sequence variations in several human genes using phosphoramidite bond DNA fragmentation and chip-based MALDI-TOF

Genome research, 2004-01, Vol.14 (1), p.134-141

2004

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p53 mutations, benzo[a]pyrene and lung cancer: a reply

Mutagenesis, 1998-11, Vol.13 (6), p.537-538

1998

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Matrix-induced fragmentation of P3'-N5' phosphoramidate-containing DNA: high-throughput MALDI-TOF analysis of genomic sequence polymorphisms

Nucleic acids research, 2001-09, Vol.29 (18), p.3864-3872

2001

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Role of ICAM1 in invasion of human breast cancer cells

Carcinogenesis (New York), 2005-05, Vol.26 (5), p.943-950

2005

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Expression of base excision repair enzymes in rat and mouse liver is induced by peroxisome proliferators and is dependent upon carcinogenic potency

Carcinogenesis (New York), 2000-12, Vol.21 (12), p.2141-2145

2000

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Single-Tube Immuno-Capture and PCR of Genotoxin-Modified DNA: Application to Gene-Specific Damage Analysis

BioTechniques, 1996-08, Vol.21 (2), p.187-188

1996

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Suppression of RAD21 gene expression decreases cell growth and enhances cytotoxicity of etoposide and bleomycin in human breast cancer cells

Molecular cancer therapeutics, 2005-03, Vol.4 (3), p.361-368

2005

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PCR-based approaches to adduct analysis

Toxicology letters, 1998-12, Vol.102-103 (1-3), p.447-451

1998

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Rapid Activation of Apoptosis in Human Promyelocytic Leukemic Cells by (±)-anti-Benzo[a]pyrene Diol Epoxide-Induced DNA Damage

Biochemical and biophysical research communications, 1993-12, Vol.197 (2), p.722-729

1993

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Mapping polycyclic aromatic hydrocarbon and aromatic amine-induced DNA damage in cancer-related genes at the sequence level

International journal of hygiene and environmental health, 2002-03, Vol.205 (1-2), p.103-113

2002

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Assessment of DNA damage and repair in specific genomic regions by quantitative immuno-coupled PCR

Nucleic acids research, 1994-06, Vol.22 (12), p.2351-2359

1994

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Quantitation and mapping of aflatoxin B1-induced DNA damage in genomic DNA using aflatoxin B1-8,9-epoxide and microsomal activation systems

Mutation research, 1999-04, Vol.425 (2), p.205-211

1999

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Mining Disease Susceptibility Genes through SNP Analyses and Expression Profiling Using MALDI-TOF Mass Spectrometry

Journal of proteome research, 2004-03, Vol.3 (2), p.218-227

2004

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Targeting of Lung Cancer Mutational Hotspots by Polycyclic Aromatic Hydrocarbons

Preferential Formation of Benzo[a]pyrene Adducts at Lung Cancer Mutational Hotspots in P53

Sunlight induces pyrimidine dimers preferentially at 5-methylcytosine bases

Tobacco smoke carcinogens, DNA damage and p53 mutations in smoking-associated cancers

Slow repair of bulky DNA adducts along the nontranscribed strand of the human p53 gene may explain the strand bias of transversion mutations in cancers

Mutation hotspots and DNA methylation

Cytosine Methylation Determines Hot Spots of DNA Damage in the Human P53 Gene

Formation and repair of DNA lesions in the p53 gene: Relation to cancer mutations?

A single nucleotide polymorphism based approach for the identification and characterization of gene expression modulation using MassARRAY

Expression of base excision DNA repair genes is a sensitive biomarker for in vivo detection of chemical-induced chronic oxidative stress: Identification of the molecular source of radicals responsible for DNA damage by peroxisome proliferators

Use of UvrABC nuclease to quantify benzo[a]pyrene diol epoxide–DNA adduct formation at methylated versus unmethylated CpG sites in the p53 gene

The p53 codon 249 mutational hotspot in hepatocellular carcinoma is not related to selective formation or persistence of aflatoxin B1 adducts

Analysis of sequence variations in several human genes using phosphoramidite bond DNA fragmentation and chip-based MALDI-TOF

p53 mutations, benzo[a]pyrene and lung cancer: a reply

Matrix-induced fragmentation of P3'-N5' phosphoramidate-containing DNA: high-throughput MALDI-TOF analysis of genomic sequence polymorphisms

Role of ICAM1 in invasion of human breast cancer cells

Expression of base excision repair enzymes in rat and mouse liver is induced by peroxisome proliferators and is dependent upon carcinogenic potency

Single-Tube Immuno-Capture and PCR of Genotoxin-Modified DNA: Application to Gene-Specific Damage Analysis

Suppression of RAD21 gene expression decreases cell growth and enhances cytotoxicity of etoposide and bleomycin in human breast cancer cells

PCR-based approaches to adduct analysis

Rapid Activation of Apoptosis in Human Promyelocytic Leukemic Cells by (±)-anti-Benzo[a]pyrene Diol Epoxide-Induced DNA Damage

Mapping polycyclic aromatic hydrocarbon and aromatic amine-induced DNA damage in cancer-related genes at the sequence level

Assessment of DNA damage and repair in specific genomic regions by quantitative immuno-coupled PCR

Quantitation and mapping of aflatoxin B1-induced DNA damage in genomic DNA using aflatoxin B1-8,9-epoxide and microsomal activation systems

Mining Disease Susceptibility Genes through SNP Analyses and Expression Profiling Using MALDI-TOF Mass Spectrometry

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