UB Paderborn / Katalog / Suche / in CDI

Early Treatment With Lisinopril and Spironolactone Preserves Cardiac and Skeletal Muscle in Duchenne Muscular Dystrophy Mice

Circulation (New York, N.Y.), 2011-08, Vol.124 (5), p.582-588

2011

Volltextzugriff (PDF)

Cardiomyopathy in the dystrophin/utrophin-deficient mouse model of severe muscular dystrophy is characterized by dysregulation of matrix metalloproteinases

Neuromuscular disorders : NMD, 2012-11, Vol.22 (11), p.1006-1014

2012

Volltextzugriff (PDF)

Claudin-5 levels are reduced from multiple cell types in human failing hearts and are associated with mislocalization of ephrin-B1

Cardiovascular pathology, 2015-05, Vol.24 (3), p.160-167

2015

Volltextzugriff (PDF)

Peptide-based inhibition of NF-κB rescues diaphragm muscle contractile dysfunction in a murine model of Duchenne muscular dystrophy

Molecular medicine (Cambridge, Mass.), 2011-05, Vol.17 (5-6), p.508-515

2011

Volltextzugriff (PDF)

Metabolic dysfunction and altered mitochondrial dynamics in the utrophin-dystrophin deficient mouse model of duchenne muscular dystrophy

PloS one, 2015-04, Vol.10 (4), p.e0123875-e0123875

2015

Volltextzugriff (PDF)

Antitrypanosomal Activity of 1,2-Dihydroquinolin-6-ols and Their Ester Derivatives

Journal of medicinal chemistry, 2010-02, Vol.53 (3), p.966-982

2010

Volltextzugriff (PDF)

The Extracellular Matrix Protein ABI3BP in Cardiovascular Health and Disease

Frontiers in cardiovascular medicine, 2019-03, Vol.6, p.23-23

2019

Volltextzugriff (PDF)

Redox-active dinitrodiphenylthioethers against Leishmania: Synthesis, structure–activity relationships and mechanism of action studies

Bioorganic & medicinal chemistry, 2009-01, Vol.17 (2), p.820-829

2009

Volltextzugriff (PDF)

Sustaining Cardiac Claudin-5 Levels Prevents Functional Hallmarks of Cardiomyopathy in a Muscular Dystrophy Mouse Model

Molecular therapy, 2012-07, Vol.20 (7), p.1378-1383

2012

Volltextzugriff (PDF)

Improvement of cardiac contractile function by peptide-based inhibition of NF-κB in the utrophin/dystrophin-deficient murine model of muscular dystrophy

Journal of translational medicine, 2011-05, Vol.9 (1), p.68-68, Article 68

2011

Volltextzugriff (PDF)

Lengthening-contractions in isolated myocardium impact force development and worsen cardiac contractile function in the mdx mouse model of muscular dystrophy

Journal of applied physiology (1985), 2011-02, Vol.110 (2), p.512-519

2011

Volltextzugriff (PDF)

Impaired adhesion of induced pluripotent stem cell-derived cardiac progenitor cells (iPSC-CPCs) to isolated extracellular matrix from failing hearts

Heliyon, 2018-10, Vol.4 (10), p.e00870-e00870, Article e00870

2018

Volltextzugriff (PDF)

Impairment of diastolic function by lack of frequency-dependent myofilament desensitization rabbit right ventricular hypertrophy

Circulation. Heart failure, 2009-09, Vol.2 (5), p.472-481

2009

Volltextzugriff (PDF)

Activity of and Initial Mechanistic Studies on a Novel Antileishmanial Agent Identified through in Silico Pharmacophore Development and Database Searching

Journal of medicinal chemistry, 2006-07, Vol.49 (14), p.4196-4207

2006

Volltextzugriff (PDF)

Response to Letter Regarding Article, “Early Treatment With Lisinopril and Spironolactone Preserves Cardiac and Skeletal Muscle in Duchenne Muscular Dystrophy Mice”

Circulation (New York, N.Y.), 2012-03, Vol.125 (9)

2012

Volltextzugriff (PDF)

Antikinetoplastid antimitotic activity and metabolic stability of dinitroaniline sulfonamides and benzamides

Bioorganic & medicinal chemistry, 2006-08, Vol.14 (16), p.5699-5710

2006

Volltextzugriff (PDF)

Impairment of Diastolic Function by Lack of Frequency-Dependent Myofilament Desensitizationin Rabbit Right Ventricular Hypertrophy

Circulation. Heart failure, 2009-09, Vol.2 (5), p.472-481

2009

Volltextzugriff (PDF)

Altered protein levels in the isolated extracellular matrix of failing human hearts with dilated cardiomyopathy

Cardiovascular pathology, 2017-01, Vol.26, p.12-20

2017

Volltextzugriff (PDF)

Synthesis, biological evaluation, and molecular modeling of 3,5 -substituted -N1 -phenyl -N4,N4 -di -n -butylsulfanilamides as antikinetoplastid antimicrotubule agents

Bioorganic & medicinal chemistry, 2007-09, Vol.15 (18), p.6071-6079

2007

Volltextzugriff (PDF)

The role of increased Sarcolipin expression in neonatal development and in muscle disease

The FASEB journal, 2013-04, Vol.27 (S1)

2013

Volltextzugriff (PDF)

Cholesterol regulation of PIP2: why cell type is so important

Frontiers in physiology, 2013-01, Vol.3

2013

Volltextzugriff (PDF)

Synthesis, biological evaluation, and molecular modeling of 3,5-substituted- N 1-phenyl- N 4, N 4-di- n-butylsulfanilamides as antikinetoplastid antimicrotubule agents

Bioorganic & medicinal chemistry, 2007-09, Vol.15 (18), p.6071-6079

2007

Volltextzugriff (PDF)

Abstract 352: The Effect of the Extracellular Matrix of Failing Hearts on Cardiac Stem Cells

Circulation research, 2016-07, Vol.119 (suppl_1)

2016

Volltextzugriff (PDF)

Abstract 85: Cardiac Pathology Is Worsened by Loss of the Cell Junction Protein Claudin-5 and Improved by Claudin-5 Gene Therapy

Circulation research, 2012-08, Vol.111 (suppl_1)

2012

Volltextzugriff (PDF)

Early Treatment with Lisinopril and Spironolactone Preserves Cardiac and Skeletal Muscle in Duchenne Muscular Dystrophy Mice: Rafael-Fortney: Spironolactone+ACEI Protects DMD Muscle

Circulation (New York, N.Y.), 2011-07, Vol.124 (5), p.582-588

2011

Volltextzugriff (PDF)

Menü

Weitere Dienste

Einstellungen

Early Treatment With Lisinopril and Spironolactone Preserves Cardiac and Skeletal Muscle in Duchenne Muscular Dystrophy Mice

Cardiomyopathy in the dystrophin/utrophin-deficient mouse model of severe muscular dystrophy is characterized by dysregulation of matrix metalloproteinases

Claudin-5 levels are reduced from multiple cell types in human failing hearts and are associated with mislocalization of ephrin-B1

Peptide-based inhibition of NF-κB rescues diaphragm muscle contractile dysfunction in a murine model of Duchenne muscular dystrophy

Metabolic dysfunction and altered mitochondrial dynamics in the utrophin-dystrophin deficient mouse model of duchenne muscular dystrophy

Antitrypanosomal Activity of 1,2-Dihydroquinolin-6-ols and Their Ester Derivatives

The Extracellular Matrix Protein ABI3BP in Cardiovascular Health and Disease

Redox-active dinitrodiphenylthioethers against Leishmania: Synthesis, structure–activity relationships and mechanism of action studies

Sustaining Cardiac Claudin-5 Levels Prevents Functional Hallmarks of Cardiomyopathy in a Muscular Dystrophy Mouse Model

Improvement of cardiac contractile function by peptide-based inhibition of NF-κB in the utrophin/dystrophin-deficient murine model of muscular dystrophy

Lengthening-contractions in isolated myocardium impact force development and worsen cardiac contractile function in the mdx mouse model of muscular dystrophy

Impaired adhesion of induced pluripotent stem cell-derived cardiac progenitor cells (iPSC-CPCs) to isolated extracellular matrix from failing hearts

Impairment of diastolic function by lack of frequency-dependent myofilament desensitization rabbit right ventricular hypertrophy

Activity of and Initial Mechanistic Studies on a Novel Antileishmanial Agent Identified through in Silico Pharmacophore Development and Database Searching

Response to Letter Regarding Article, “Early Treatment With Lisinopril and Spironolactone Preserves Cardiac and Skeletal Muscle in Duchenne Muscular Dystrophy Mice”

Antikinetoplastid antimitotic activity and metabolic stability of dinitroaniline sulfonamides and benzamides

Impairment of Diastolic Function by Lack of Frequency-Dependent Myofilament Desensitizationin Rabbit Right Ventricular Hypertrophy

Altered protein levels in the isolated extracellular matrix of failing human hearts with dilated cardiomyopathy

Synthesis, biological evaluation, and molecular modeling of 3,5 -substituted -N1 -phenyl -N4,N4 -di -n -butylsulfanilamides as antikinetoplastid antimicrotubule agents

The role of increased Sarcolipin expression in neonatal development and in muscle disease

Cholesterol regulation of PIP2: why cell type is so important

Synthesis, biological evaluation, and molecular modeling of 3,5-substituted- N 1-phenyl- N 4, N 4-di- n-butylsulfanilamides as antikinetoplastid antimicrotubule agents

Abstract 352: The Effect of the Extracellular Matrix of Failing Hearts on Cardiac Stem Cells

Abstract 85: Cardiac Pathology Is Worsened by Loss of the Cell Junction Protein Claudin-5 and Improved by Claudin-5 Gene Therapy

Early Treatment with Lisinopril and Spironolactone Preserves Cardiac and Skeletal Muscle in Duchenne Muscular Dystrophy Mice: Rafael-Fortney: Spironolactone+ACEI Protects DMD Muscle

Suchergebnisse filtern