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Tau Protein Hyperphosphorylation and Aggregation in Alzheimer's Disease and Other Tauopathies, and Possible Neuroprotective Strategies

Biomolecules, 2016-01, Vol.6 (1), p.6

2016

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Assessment of common variability and expression quantitative trait loci for genome-wide associations for progressive supranuclear palsy

Neurobiology of aging, 2014-06, Vol.35 (6), p.1514.e1-1514.e12

2014

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Genetic variability in the regulation of gene expression in ten regions of the human brain

Nature neuroscience, 2014-10, Vol.17 (10), p.1418-1428

2014

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The precursor protein of non-Aβ component of Alzheimer's disease amyloid is a presynaptic protein of the central nervous system

Neuron (Cambridge, Mass.), 1995-02, Vol.14 (2), p.467-475

1995

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MAPT expression and splicing is differentially regulated by brain region: relation to genotype and implication for tauopathies

Human molecular genetics, 2012-09, Vol.21 (18), p.4094-4103

2012

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Pathological tau burden and distribution distinguishes progressive supranuclear palsy-parkinsonism from Richardson's syndrome

Brain (London, England : 1878), 2007-06, Vol.130 (6), p.1566-1576

2007

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The contribution of DNA methylation to the (dys)function of oligodendroglia in neurodegeneration

Acta neuropathologica communications, 2023-06, Vol.11 (1), p.106-106, Article 106

2023

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Characteristics of two distinct clinical phenotypes in pathologically proven progressive supranuclear palsy: Richardson's syndrome and PSP-parkinsonism

Brain (London, England : 1878), 2005-06, Vol.128 (6), p.1247-1258

2005

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The tau H2 haplotype is almost exclusively Caucasian in origin

Neuroscience letters, 2004-10, Vol.369 (3), p.183-185

2004

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Pre-clinical characterisation of E2814, a high-affinity antibody targeting the microtubule-binding repeat domain of tau for passive immunotherapy in Alzheimer's disease

Acta neuropathologica communications, 2020-02, Vol.8 (1), p.13-13, Article 13

2020

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Evolving prion-like tau conformers differentially alter postsynaptic proteins in neurons inoculated with distinct isolates of Alzheimer’s disease tau

Cell & bioscience, 2023-09, Vol.13 (1), p.1-174, Article 174

2023

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Hyperphosphorylation and aggregation of tau in mice expressing normal human tau isoforms

Journal of neurochemistry, 2003-08, Vol.86 (3), p.582-590

2003

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The analysis of C9orf72 repeat expansions in a large series of clinically and pathologically diagnosed cases with atypical parkinsonism

Neurobiology of aging, 2015-02, Vol.36 (2), p.1221.e1-1221.e6

2015

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Variation in tau isoform expression in different brain regions and disease states

Neurobiology of aging, 2013-07, Vol.34 (7), p.1922.e7-1922.e12

2013

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Development and assessment of sensitive immuno-PCR assays for the quantification of cerebrospinal fluid three- and four-repeat tau isoforms in tauopathies

Journal of neurochemistry, 2012-11, Vol.123 (3), p.396-405

2012

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The MAPT H1c risk haplotype is associated with increased expression of tau and especially of 4 repeat containing transcripts

Neurobiology of disease, 2007-03, Vol.25 (3), p.561-570

2007

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Differential incorporation of tau isoforms in Alzheimer's disease

Journal of Alzheimer's disease, 2008-05, Vol.14 (1), p.1

2008

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Extensive Anti-CoA Immunostaining in Alzheimer's Disease and Covalent Modification of Tau by a Key Cellular Metabolite Coenzyme A

Frontiers in cellular neuroscience, 2021-10, Vol.15, p.739425-739425

2021

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Multiple system atrophy/progressive supranuclear palsy: alpha-Synuclein, synphilin, tau, and APOE

Neurology, 2000-12, Vol.55 (12), p.1918

2000

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Widespread RNA metabolism impairment in sporadic inclusion body myositis TDP43-proteinopathy

Neurobiology of aging, 2014-06, Vol.35 (6), p.1491-1498

2014

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Immunohistochemical study of tau accumulation in early stages of Alzheimer-type neurofibrillary lesions

Acta neuropathologica, 2004-06, Vol.107 (6), p.504-508

2004

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Corrigendum to “Assessment of common variability and expression quantitative trait loci for genome-wide associations for progressive supranuclear palsy.” [Neurobiol. Aging 35 (2014) 1514.e1–1514.e12]

Neurobiology of aging, 2015-11, Vol.36 (11), p.3118-3118

2015

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Foamy Virus Vectors Transduce Visceral Organs and Hippocampal Structures following In Vivo Delivery to Neonatal Mice

Molecular therapy. Nucleic acids, 2018-09, Vol.12, p.626-634

2018

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MAPT S305I mutation: implications for argyrophilic grain disease

Acta neuropathologica, 2008-07, Vol.116 (1), p.103-118

2008

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Tau Protein Hyperphosphorylation and Aggregation in Alzheimer's Disease and Other Tauopathies, and Possible Neuroprotective Strategies

Assessment of common variability and expression quantitative trait loci for genome-wide associations for progressive supranuclear palsy

Genetic variability in the regulation of gene expression in ten regions of the human brain

The precursor protein of non-Aβ component of Alzheimer's disease amyloid is a presynaptic protein of the central nervous system

MAPT expression and splicing is differentially regulated by brain region: relation to genotype and implication for tauopathies

Pathological tau burden and distribution distinguishes progressive supranuclear palsy-parkinsonism from Richardson's syndrome

The contribution of DNA methylation to the (dys)function of oligodendroglia in neurodegeneration

Characteristics of two distinct clinical phenotypes in pathologically proven progressive supranuclear palsy: Richardson's syndrome and PSP-parkinsonism

The tau H2 haplotype is almost exclusively Caucasian in origin

Pre-clinical characterisation of E2814, a high-affinity antibody targeting the microtubule-binding repeat domain of tau for passive immunotherapy in Alzheimer's disease

Evolving prion-like tau conformers differentially alter postsynaptic proteins in neurons inoculated with distinct isolates of Alzheimer’s disease tau

Hyperphosphorylation and aggregation of tau in mice expressing normal human tau isoforms

The analysis of C9orf72 repeat expansions in a large series of clinically and pathologically diagnosed cases with atypical parkinsonism

Fulminant corticobasal degeneration: a distinct variant with predominant neuronal tau aggregates

Variation in tau isoform expression in different brain regions and disease states

Development and assessment of sensitive immuno-PCR assays for the quantification of cerebrospinal fluid three- and four-repeat tau isoforms in tauopathies

The MAPT H1c risk haplotype is associated with increased expression of tau and especially of 4 repeat containing transcripts

Differential incorporation of tau isoforms in Alzheimer's disease

Extensive Anti-CoA Immunostaining in Alzheimer's Disease and Covalent Modification of Tau by a Key Cellular Metabolite Coenzyme A

Multiple system atrophy/progressive supranuclear palsy: alpha-Synuclein, synphilin, tau, and APOE

Widespread RNA metabolism impairment in sporadic inclusion body myositis TDP43-proteinopathy

Immunohistochemical study of tau accumulation in early stages of Alzheimer-type neurofibrillary lesions

Corrigendum to “Assessment of common variability and expression quantitative trait loci for genome-wide associations for progressive supranuclear palsy.” [Neurobiol. Aging 35 (2014) 1514.e1–1514.e12]

Foamy Virus Vectors Transduce Visceral Organs and Hippocampal Structures following In Vivo Delivery to Neonatal Mice

MAPT S305I mutation: implications for argyrophilic grain disease

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