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Liver-brain inflammation axis

American journal of physiology: Gastrointestinal and liver physiology, 2011-11, Vol.301 (5), p.G749-G761

2011

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Glioma-derived IL-33 orchestrates an inflammatory brain tumor microenvironment that accelerates glioma progression

Nature communications, 2020-10, Vol.11 (1), p.4997-24, Article 4997

2020

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Re-programming mouse liver-resident invariant natural killer T cells for suppressing hepatic and diabetogenic autoimmunity

Nature communications, 2022-06, Vol.13 (1), p.3279-3279, Article 3279

2022

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P-selectin-mediated monocyte-cerebral endothelium adhesive interactions link peripheral organ inflammation to sickness behaviors

The Journal of neuroscience, 2013-09, Vol.33 (37), p.14878-14888

2013

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Blunted cardiac response to hemorrhage in cirrhotic rats is mediated by local macrophage-released endocannabinoids

Journal of hepatology, 2015-06, Vol.62 (6), p.1272-1277

2015

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Immune-to-Brain Communication Pathways in Inflammation-Associated Sickness and Depression

Current topics in behavioral neurosciences, 2017-01, Vol.31, p.73-94

2017

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Oxidized phosphatidylcholines found in multiple sclerosis lesions mediate neurodegeneration and are neutralized by microglia

Nature neuroscience, 2021-04, Vol.24 (4), p.489-503

2021

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Liver–brain interactions in inflammatory liver diseases: Implications for fatigue and mood disorders

Brain, behavior, and immunity, 2014-01, Vol.35, p.9-20

2014

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Cerebral Microglia Recruit Monocytes into the Brain in Response to Tumor Necrosis Factorα Signaling during Peripheral Organ Inflammation

The Journal of neuroscience, 2009-02, Vol.29 (7), p.2089-2102

2009

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Elevated Galectin-3 Is Associated with Aging, Multiple Sclerosis, and Oxidized Phosphatidylcholine-Induced Neurodegeneration

The Journal of neuroscience, 2023-06, Vol.43 (25), p.4725-4737

2023

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Uncovering Novel Extracellular Matrix Transcriptome Alterations in Lesions of Multiple Sclerosis

International journal of molecular sciences, 2024-01, Vol.25 (2), p.1240

2024

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Expression of antioxidant enzymes in lesions of multiple sclerosis and its models

Scientific reports, 2022-07, Vol.12 (1), p.12761-12761, Article 12761

2022

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Interactions Between Platelets and Inflammatory Monocytes Affect Sickness Behavior in Mice With Liver Inflammation

Gastroenterology (New York, N.Y. 1943), 2017-11, Vol.153 (5), p.1416-1428.e2

2017

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Fibulin-2 is an extracellular matrix inhibitor of oligodendrocytes relevant to multiple sclerosis

The Journal of clinical investigation, 2024-07, Vol.134 (13), p.1-18

2024

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ADAM Metalloproteinase Domain 17 Regulates Cholestasis-Associated Liver Injury and Sickness Behavior Development in Mice

Frontiers in immunology, 2022-01, Vol.12, p.779119-779119

2022

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Probiotics Improve Inflammation-Associated Sickness Behavior by Altering Communication between the Peripheral Immune System and the Brain

The Journal of neuroscience, 2015-07, Vol.35 (30), p.10821-10830

2015

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Single-Cell Spatial Analysis Identifies Regulators of Brain Tumor-Initiating Cells

Cancer research (Chicago, Ill.), 2023-05, Vol.83 (10), p.1725-1741

2023

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Regulatory T cells suppress sickness behaviour development without altering liver injury in cholestatic mice

Journal of hepatology, 2012-03, Vol.56 (3), p.626-631

2012

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Single-cell and spatial RNA sequencing identify perturbators of microglial functions with aging

Nature aging, 2022-06, Vol.2 (6), p.508-525

2022

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273 - Tumor Necrosis Factor Alpha (Tnfα) Cleaving Enzyme (TACE/ADAM17) Expression is Enhanced in Cholestatic Liver Disease and Regulates Cholestasis-Associated Liver Injury and Sickness Behavior Development

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S1045-S1045

2017

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Liver-brain inflammation axis

American journal of physiology: Gastrointestinal and liver physiology, 2011, Vol.64 (5)

2011

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Tumor Necrosis Factor Alpha (Tnfα) Cleaving Enzyme (TACE/ADAM17) Expression is Enhanced in Cholestatic Liver Disease and Regulates Cholestasis-Associated Liver Injury and Sickness Behavior Development

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S1045-S1045

2017

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TNF-α-secreting monocytes are recruited into the brain of cholestatic mice

Hepatology (Baltimore, Md.), 2006, Vol.43 (1), p.154-162

2006

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Endogenous opioid-mediated antinociception in cholestatic mice is peripherally, not centrally, mediated

Journal of hepatology, 2006-06, Vol.44 (6), p.1141-1149

2006

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Liver-Brain Inflammation Axis: Irnmune Cell Driven Sickness Behavior Regulation

D'Mello, Charlotte

2011

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Liver-brain inflammation axis

Glioma-derived IL-33 orchestrates an inflammatory brain tumor microenvironment that accelerates glioma progression

Re-programming mouse liver-resident invariant natural killer T cells for suppressing hepatic and diabetogenic autoimmunity

P-selectin-mediated monocyte-cerebral endothelium adhesive interactions link peripheral organ inflammation to sickness behaviors

Blunted cardiac response to hemorrhage in cirrhotic rats is mediated by local macrophage-released endocannabinoids

Immune-to-Brain Communication Pathways in Inflammation-Associated Sickness and Depression

Oxidized phosphatidylcholines found in multiple sclerosis lesions mediate neurodegeneration and are neutralized by microglia

Liver–brain interactions in inflammatory liver diseases: Implications for fatigue and mood disorders

Cerebral Microglia Recruit Monocytes into the Brain in Response to Tumor Necrosis Factorα Signaling during Peripheral Organ Inflammation

Elevated Galectin-3 Is Associated with Aging, Multiple Sclerosis, and Oxidized Phosphatidylcholine-Induced Neurodegeneration

Uncovering Novel Extracellular Matrix Transcriptome Alterations in Lesions of Multiple Sclerosis

Expression of antioxidant enzymes in lesions of multiple sclerosis and its models

Interactions Between Platelets and Inflammatory Monocytes Affect Sickness Behavior in Mice With Liver Inflammation

Fibulin-2 is an extracellular matrix inhibitor of oligodendrocytes relevant to multiple sclerosis

ADAM Metalloproteinase Domain 17 Regulates Cholestasis-Associated Liver Injury and Sickness Behavior Development in Mice

Probiotics Improve Inflammation-Associated Sickness Behavior by Altering Communication between the Peripheral Immune System and the Brain

Single-Cell Spatial Analysis Identifies Regulators of Brain Tumor-Initiating Cells

Regulatory T cells suppress sickness behaviour development without altering liver injury in cholestatic mice

Single-cell and spatial RNA sequencing identify perturbators of microglial functions with aging

273 - Tumor Necrosis Factor Alpha (Tnfα) Cleaving Enzyme (TACE/ADAM17) Expression is Enhanced in Cholestatic Liver Disease and Regulates Cholestasis-Associated Liver Injury and Sickness Behavior Development

Liver-brain inflammation axis

Tumor Necrosis Factor Alpha (Tnfα) Cleaving Enzyme (TACE/ADAM17) Expression is Enhanced in Cholestatic Liver Disease and Regulates Cholestasis-Associated Liver Injury and Sickness Behavior Development

TNF-α-secreting monocytes are recruited into the brain of cholestatic mice

Endogenous opioid-mediated antinociception in cholestatic mice is peripherally, not centrally, mediated

Liver-Brain Inflammation Axis: Irnmune Cell Driven Sickness Behavior Regulation

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