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Inducible Brown Adipose Tissue, or Beige Fat, Is Anabolic for the Skeleton

Endocrinology (Philadelphia), 2013-08, Vol.154 (8), p.2687-2701

2013

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Partial agonist, telmisartan, maintains PPARγ serine 112 phosphorylation, and does not affect osteoblast differentiation and bone mass

PloS one, 2014-05, Vol.9 (5), p.e96323-e96323

2014

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Osteocytes contribute via nuclear receptor PPAR-alpha to maintenance of bone and systemic energy metabolism

Frontiers in endocrinology (Lausanne), 2023-04, Vol.14, p.1145467-1145467

2023

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Marrow Adipose Tissue: Skeletal Location, Sexual Dimorphism, and Response to Sex Steroid Deficiency

Frontiers in endocrinology (Lausanne), 2017-08, Vol.8, p.188-188

2017

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PPARG in osteocytes controls cell bioenergetics and systemic energy metabolism independently of sclerostin levels in circulation

Molecular metabolism (Germany), 2024-10, Vol.88, p.102000, Article 102000

2024

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β-catenin directly sequesters adipocytic and insulin sensitizing activities but not osteoblastic activity of PPARγ2 in marrow mesenchymal stem cells

PloS one, 2012-12, Vol.7 (12), p.e51746-e51746

2012

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Bone marrow fat has brown adipose tissue characteristics, which are attenuated with aging and diabetes

Bone (New York, N.Y.), 2012-02, Vol.50 (2), p.546-552

2012

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Synapsins as mediators of BDNF-enhanced neurotransmitter release

Nature neuroscience, 2000-04, Vol.3 (4), p.323-329

2000

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PPARγ2 nuclear receptor controls multiple regulatory pathways of osteoblast differentiation from marrow mesenchymal stem cells

Journal of cellular biochemistry, 2009-02, Vol.106 (2), p.232-246

2009

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High bone mass in adult mice with diet-induced obesity results from a combination of initial increase in bone mass followed by attenuation in bone formation; implications for high bone mass and decreased bone quality in obesity

Molecular and cellular endocrinology, 2015-07, Vol.410, p.35-41

2015

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PPARG in osteocytes controls sclerostin expression, bone mass, marrow adiposity and mediates TZD-induced bone loss

Bone (New York, N.Y.), 2021-06, Vol.147, p.115913-115913, Article 115913

2021

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Nonenzymatic and Trophic Activities of Carboxypeptidase E Regulate Bone Mass and Bioenergetics of Skeletal Stem Cells in Mice

JBMR plus, 2020-09, Vol.4 (9), p.e10392-n/a

2020

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Synaptic Vesicle Phosphoproteins and Regulation of Synaptic Function

Science (American Association for the Advancement of Science), 1993-02, Vol.259 (5096), p.780-785

1993

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Phosphorylation of DARPP-32 by Cdk5 modulates dopamine signalling in neurons

Nature (London), 1999-12, Vol.402 (6762), p.669-671

1999

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Synapsins as regulators of neurotransmitter release

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 1999-02, Vol.354 (1381), p.269-279

1999

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Distinct pools of synaptic vesicles in neurotransmitter release

Nature (London), 1995-06, Vol.375 (6531), p.493-497

1995

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Opposing Changes in Phosphorylation of Specific Sites in Synapsin I During Ca2+-Dependent Glutamate Release in Isolated Nerve Terminals

The Journal of neuroscience, 2001-10, Vol.21 (20), p.7944-7953

2001

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14-3-3ζ: A suppressor of inflammatory arthritis

Proceedings of the National Academy of Sciences - PNAS, 2021-08, Vol.118 (34), p.1-10

2021

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Neuronal Nitric-Oxide Synthase Localization Mediated by a Ternary Complex with Synapsin and CAPON

Proceedings of the National Academy of Sciences - PNAS, 2002-03, Vol.99 (5), p.3199-3204

2002

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STRUCTURAL AND FUNCTIONAL STUDIES OF UDP-GLUCURONOSYLTRANSFERASES

Drug metabolism reviews, 1999-01, Vol.31 (4), p.817-899

1999

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Physiological role of mGSTA4-4, a glutathione S-transferase metabolizing 4-hydroxynonenal: generation and analysis of mGsta4 null mouse

Toxicology and applied pharmacology, 2004-02, Vol.194 (3), p.296-308

2004

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Structural Domains Involved in the Regulation of Transmitter Release by Synapsins

The Journal of neuroscience, 2005-03, Vol.25 (10), p.2658-2669

2005

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A Third Member of the Synapsin Gene Family

Proceedings of the National Academy of Sciences - PNAS, 1998-04, Vol.95 (8), p.4667-4672

1998

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PPARG Post-translational Modifications Regulate Bone Formation and Bone Resorption

EBioMedicine, 2016-08, Vol.10 (C), p.174-184

2016

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Diabetes increases risk of lumbar spinal fusion complications: association with altered structure of newly formed bone at the fusion site

JBMR plus, 2024-06, Vol.8 (6), p.ziae053-ziae053

2024

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Inducible Brown Adipose Tissue, or Beige Fat, Is Anabolic for the Skeleton

Partial agonist, telmisartan, maintains PPARγ serine 112 phosphorylation, and does not affect osteoblast differentiation and bone mass

Osteocytes contribute via nuclear receptor PPAR-alpha to maintenance of bone and systemic energy metabolism

Marrow Adipose Tissue: Skeletal Location, Sexual Dimorphism, and Response to Sex Steroid Deficiency

PPARG in osteocytes controls cell bioenergetics and systemic energy metabolism independently of sclerostin levels in circulation

β-catenin directly sequesters adipocytic and insulin sensitizing activities but not osteoblastic activity of PPARγ2 in marrow mesenchymal stem cells

Bone marrow fat has brown adipose tissue characteristics, which are attenuated with aging and diabetes

Synapsins as mediators of BDNF-enhanced neurotransmitter release

PPARγ2 nuclear receptor controls multiple regulatory pathways of osteoblast differentiation from marrow mesenchymal stem cells

High bone mass in adult mice with diet-induced obesity results from a combination of initial increase in bone mass followed by attenuation in bone formation; implications for high bone mass and decreased bone quality in obesity

PPARG in osteocytes controls sclerostin expression, bone mass, marrow adiposity and mediates TZD-induced bone loss

Nonenzymatic and Trophic Activities of Carboxypeptidase E Regulate Bone Mass and Bioenergetics of Skeletal Stem Cells in Mice

Synaptic Vesicle Phosphoproteins and Regulation of Synaptic Function

Phosphorylation of DARPP-32 by Cdk5 modulates dopamine signalling in neurons

Synapsins as regulators of neurotransmitter release

Distinct pools of synaptic vesicles in neurotransmitter release

Opposing Changes in Phosphorylation of Specific Sites in Synapsin I During Ca2+-Dependent Glutamate Release in Isolated Nerve Terminals

14-3-3ζ: A suppressor of inflammatory arthritis

Neuronal Nitric-Oxide Synthase Localization Mediated by a Ternary Complex with Synapsin and CAPON

STRUCTURAL AND FUNCTIONAL STUDIES OF UDP-GLUCURONOSYLTRANSFERASES

Physiological role of mGSTA4-4, a glutathione S-transferase metabolizing 4-hydroxynonenal: generation and analysis of mGsta4 null mouse

Structural Domains Involved in the Regulation of Transmitter Release by Synapsins

A Third Member of the Synapsin Gene Family

PPARG Post-translational Modifications Regulate Bone Formation and Bone Resorption

Diabetes increases risk of lumbar spinal fusion complications: association with altered structure of newly formed bone at the fusion site

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