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Azathioprine in the Management of Autoimmune Uveitis

Ocular immunology and inflammation, 2008-01, Vol.16 (4), p.161-165

2008

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Anti-tumor necrosis factor-alpha therapy augments dipeptidyl peptidase IV activity and decreases autoantibodies to GRP78/BIP and phosphoglucose isomerase in patients with rheumatoid arthritis

Journal of rheumatology, 2005-11, Vol.32 (11), p.2116

2005

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B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and FcgammaRIIb, which are modulated by anti-tumor necrosis factor therapy

Arthritis research & therapy, 2010, Vol.12 (2), p.R68

2010

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Prostate cancer cell proliferation in vitro is modulated by antibodies against glucose-regulated protein 78 isolated from patient serum

Cancer research (Chicago, Ill.), 2006-12, Vol.66 (23), p.11424-11431

2006

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Catalytic autoantibodies against myelin basic protein (MBP) isolated from serum of autistic children impair in vitro models of synaptic plasticity in rat hippocampus

Journal of neuroimmunology, 2015-10, Vol.287, p.1-8

2015

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Established Murine Collagen-induced Arthritis and IL-17 Production are Specifically Inhibited by a Single Inoculation of Lipopolysaccharide-stimulated Dendritic Cells

Clinical immunology (Orlando, Fla.), 2007-01, Vol.123, p.S96-S96

2007

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Anti-TNF therapy in patients with rheumatoid arthritis decreases Th1 and Th17 cell populations and expands IFN-γ-producing NK cell and regulatory T cell subsets

Immunobiology (1979), 2011-12, Vol.216 (12), p.1256-1263

2011

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Clinical parameters and biomarkers for anti-TNF treatment prognosis in rheumatoid arthritis patients

Clinical rheumatology, 2014-12, Vol.33 (12), p.1707-1714

2014

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B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and Fc gamma RIIb, which are modulated by anti-tumor necrosis factor therapy

Arthritis research & therapy, 2010-01, Vol.12 (2), p.R68-R68

2010

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Antibodies against the voltage-dependent anion channel (VDAC) and its protective ligand hexokinase-I in children with autism

Journal of neuroimmunology, 2010-10, Vol.227 (1), p.153-161

2010

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Streptokinase Promotes Development of Dipeptidyl Peptidase IV (CD26) Autoantibodies after Fibrinolytic Therapy in Myocardial Infarction Patients

Clinical and Diagnostic Laboratory Immunology, 2002-11, Vol.9 (6), p.1253-1259

2002

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Acute herpes simplex virus 1 pneumonitis in a patient with systemic lupus erythematosus

Journal of clinical rheumatology, 2014-01, Vol.20 (1), p.42-44

2014

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Mycophenolate Mofetil Therapy in Refractory Inflammatory Eye Disease

Journal of ocular pharmacology and therapeutics, 2016-01, Vol.32 (1), p.55-61

2016

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Multiple imputation procedures allow the rescue of missing data: an application to determine serum tumor necrosis factor (TNF) concentration values during the treatment of rheumatoid arthritis patients with anti-TNF therapy

Biological research, 2005, Vol.38 (1), p.7-12

2005

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Basal anti-cyclic citrullinated peptide (anti-CCP) antibody levels and a decrease in anti-CCP titres are associated with clinical response to adalimumab in rheumatoid arthritis

Clinical and experimental rheumatology, 2008-11, Vol.26 (6), p.1067

2008

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Comparison of the Clinical Efficacy of Two Different Immunosuppressive Regimens in Patients with Chronic Vogt-Koyanagi-Harada Disease

Ocular immunology and inflammation, 2010-06, Vol.18 (3), p.200-207

2010

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Infliximab can induce a prolonged clinical remission and a decrease in thyroid hormonal requirements in a patient with SAPHO syndrome and hypothyroidism

Clinical rheumatology, 2008-04, Vol.27 (4), p.533-535

2008

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presence of anti-citrullinated protein antibodies (ACPA) does not affect the clinical response to adalimumab in a group of RA patients with the tumor necrosis factor (TNF) α-308 G/G promoter polymorphism

Clinical rheumatology, 2011-03, Vol.30 (3), p.391-395

2011

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Could single-nucleotide polymorphisms (SNPs) affecting the tumour necrosis factor promoter be considered as part of rheumatoid arthritis evolution?

Immunobiology (1979), 2006-01, Vol.211 (1), p.75-84

2006

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B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and Fc[gamma]RIIb, which are modulated by anti-tumor necrosis factor therapy

Arthritis research & therapy, 2010-04, Vol.12, p.R68

2010

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Papel de los polimorfismos -238 y -308 del promotor del factor de necrosis tumoral alfa en la patogenia y respuesta al tratamiento anti- factor de necrosis tumoral alfa en artritis reumatoide

Revista medíca de Chile, 2005-09, Vol.133 (9), p.1089-1095

2005

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Weak CD4+ T-cell responses to citrullinated vimentin in rheumatoid arthritis patients carrying HLA-DR9 alleles

Rheumatology international, 2012-06, Vol.32 (6), p.1819-1825

2012

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Anti-TNF therapy in patients with rheumatoid arthritis decreases Th1 and Th17 cell populations and expands IFN-I super(3)-producing NK cell and regulatory T cell subsets

Immunobiology (1979), 2011-12, Vol.216 (12), p.1256-1263

2011

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Adalimumab, a Tumor Necrosis Factor Blockade Therapy, Decreases Th1 and Th17 Populations in Rheumatoid Arthritis Patients

Clinical immunology (Orlando, Fla.), 2010, Vol.135, p.S105-S105

2010

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Treatment with Dexamethasone and Monophosphoryl Lipid A Removes Disease-Associated Transcriptional Signatures in Monocyte-Derived Dendritic Cells from Rheumatoid Arthritis Patients and Confers Tolerogenic Features

Azathioprine in the Management of Autoimmune Uveitis

Anti-tumor necrosis factor-alpha therapy augments dipeptidyl peptidase IV activity and decreases autoantibodies to GRP78/BIP and phosphoglucose isomerase in patients with rheumatoid arthritis

B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and FcgammaRIIb, which are modulated by anti-tumor necrosis factor therapy

Prostate cancer cell proliferation in vitro is modulated by antibodies against glucose-regulated protein 78 isolated from patient serum

Catalytic autoantibodies against myelin basic protein (MBP) isolated from serum of autistic children impair in vitro models of synaptic plasticity in rat hippocampus

Established Murine Collagen-induced Arthritis and IL-17 Production are Specifically Inhibited by a Single Inoculation of Lipopolysaccharide-stimulated Dendritic Cells

Anti-TNF therapy in patients with rheumatoid arthritis decreases Th1 and Th17 cell populations and expands IFN-γ-producing NK cell and regulatory T cell subsets

Clinical parameters and biomarkers for anti-TNF treatment prognosis in rheumatoid arthritis patients

B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and Fc gamma RIIb, which are modulated by anti-tumor necrosis factor therapy

Antibodies against the voltage-dependent anion channel (VDAC) and its protective ligand hexokinase-I in children with autism

Streptokinase Promotes Development of Dipeptidyl Peptidase IV (CD26) Autoantibodies after Fibrinolytic Therapy in Myocardial Infarction Patients

Acute herpes simplex virus 1 pneumonitis in a patient with systemic lupus erythematosus

Mycophenolate Mofetil Therapy in Refractory Inflammatory Eye Disease

Multiple imputation procedures allow the rescue of missing data: an application to determine serum tumor necrosis factor (TNF) concentration values during the treatment of rheumatoid arthritis patients with anti-TNF therapy

Basal anti-cyclic citrullinated peptide (anti-CCP) antibody levels and a decrease in anti-CCP titres are associated with clinical response to adalimumab in rheumatoid arthritis

Comparison of the Clinical Efficacy of Two Different Immunosuppressive Regimens in Patients with Chronic Vogt-Koyanagi-Harada Disease

Infliximab can induce a prolonged clinical remission and a decrease in thyroid hormonal requirements in a patient with SAPHO syndrome and hypothyroidism

presence of anti-citrullinated protein antibodies (ACPA) does not affect the clinical response to adalimumab in a group of RA patients with the tumor necrosis factor (TNF) α-308 G/G promoter polymorphism

Could single-nucleotide polymorphisms (SNPs) affecting the tumour necrosis factor promoter be considered as part of rheumatoid arthritis evolution?

B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and Fc[gamma]RIIb, which are modulated by anti-tumor necrosis factor therapy

Papel de los polimorfismos -238 y -308 del promotor del factor de necrosis tumoral alfa en la patogenia y respuesta al tratamiento anti- factor de necrosis tumoral alfa en artritis reumatoide

Weak CD4+ T-cell responses to citrullinated vimentin in rheumatoid arthritis patients carrying HLA-DR9 alleles

Anti-TNF therapy in patients with rheumatoid arthritis decreases Th1 and Th17 cell populations and expands IFN-I super(3)-producing NK cell and regulatory T cell subsets

Adalimumab, a Tumor Necrosis Factor Blockade Therapy, Decreases Th1 and Th17 Populations in Rheumatoid Arthritis Patients

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