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Endoplasmic reticulum dysfunction in neurological disease

Lancet neurology, 2013, Vol.12 (1), p.105-118

2013

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CCT complex restricts neuropathogenic protein aggregation via autophagy

Nature communications, 2016-12, Vol.7 (1), p.13821-13821, Article 13821

2016

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TREM2 shedding by cleavage at the H157‐S158 bond is accelerated for the Alzheimer's disease‐associated H157Y variant

EMBO molecular medicine, 2017-10, Vol.9 (10), p.1366-1378

2017

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ANS Binding Reveals Common Features of Cytotoxic Amyloid Species

ACS chemical biology, 2010-08, Vol.5 (8), p.735-740

2010

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Testing the Therapeutic Potential of Doxycycline in a Drosophila melanogaster Model of Alzheimer Disease

The Journal of biological chemistry, 2011-12, Vol.286 (48), p.41647-41655

2011

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Central and peripheral circadian clocks and their role in Alzheimer's disease

Disease models & mechanisms, 2017-10, Vol.10 (10), p.1187-1199

2017

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Endoplasmic Reticulum-associated Degradation (ERAD) and Autophagy Cooperate to Degrade Polymerogenic Mutant Serpins

The Journal of biological chemistry, 2009-08, Vol.284 (34), p.22793-22802

2009

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Iron is a specific cofactor for distinct oxidation- and aggregation-dependent Aβ toxicity mechanisms in a Drosophila model

Disease models & mechanisms, 2015-07, Vol.8 (7), p.657-667

2015

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Systematic in vivo analysis of the intrinsic determinants of amyloid Beta pathogenicity

PLoS biology, 2007-10, Vol.5 (11), p.e290

2007

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The TRiC/CCT chaperone is implicated in Alzheimer's disease based on patient GWAS and an RNAi screen in Aβ-expressing Caenorhabditis elegans

PloS one, 2014-07, Vol.9 (7), p.e102985-e102985

2014

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The central molecular clock is robust in the face of behavioural arrhythmia in a Drosophila model of Alzheimer's disease

Disease models & mechanisms, 2014-04, Vol.7 (4), p.445-458

2014

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p53 and Translation Attenuation Regulate Distinct Cell Cycle Checkpoints during Endoplasmic Reticulum (ER) Stress

The Journal of biological chemistry, 2013-03, Vol.288 (11), p.7606-7617

2013

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Ovine PrP transgenic Drosophila show reduced locomotor activity and decreased survival

Biochemical journal, 2012-06, Vol.444 (3), p.487-495

2012

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Impaired tissue growth is mediated by checkpoint kinase 1 (CHK1) in the integrated stress response

Journal of cell science, 2010-09, Vol.123 (17), p.2892-2900

2010

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Prion protein stabilizes amyloid-β (Aβ) oligomers and enhances Aβ neurotoxicity in a Drosophila model of Alzheimer's disease

The Journal of biological chemistry, 2018-08, Vol.293 (34), p.13090-13099

2018

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Iron Promotes the Toxicity of Amyloid β Peptide by Impeding Its Ordered Aggregation

The Journal of biological chemistry, 2011-02, Vol.286 (6), p.4248-4256

2011

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Mutant Neuroserpin (S49P) That Causes Familial Encephalopathy with Neuroserpin Inclusion Bodies Is a Poor Proteinase Inhibitor and Readily Forms Polymers in Vitro

The Journal of biological chemistry, 2002-05, Vol.277 (19), p.17367-17373

2002

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Alzheimer's disease: insights from Drosophila melanogaster models

Trends in biochemical sciences (Amsterdam. Regular ed.), 2010-04, Vol.35 (4), p.228-235

2010

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Utilizing microphysiological systems and induced pluripotent stem cells for disease modeling: a case study for blood brain barrier research in a pharmaceutical setting

Advanced drug delivery reviews, 2019-02, Vol.140, p.129-135

2019

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The pro-domains of neurotrophins, including BDNF, are linked to Alzheimer's disease through a toxic synergy with Aβ

Human molecular genetics, 2015-07, Vol.24 (14), p.3929-3938

2015

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Protein misfolding and disease: from the test tube to the organism

Current opinion in chemical biology, 2008-02, Vol.12 (1), p.25-31

2008

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Expression of the alternative oxidase mitigates beta-amyloid production and toxicity in model systems

Free radical biology & medicine, 2016-07, Vol.96, p.57-66

2016

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Metabolic changes may precede proteostatic dysfunction in a Drosophila model of amyloid β peptide toxicity

Neurobiology of aging, 2016-05, Vol.41, p.39-52

2016

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Fenton chemistry and oxidative stress mediate the toxicity of the β-amyloid peptide in a Drosophila model of Alzheimer's disease

The European journal of neuroscience, 2009-04, Vol.29 (7), p.1335-1347

2009

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Endoplasmic reticulum dysfunction in neurological disease

CCT complex restricts neuropathogenic protein aggregation via autophagy

TREM2 shedding by cleavage at the H157‐S158 bond is accelerated for the Alzheimer's disease‐associated H157Y variant

ANS Binding Reveals Common Features of Cytotoxic Amyloid Species

Testing the Therapeutic Potential of Doxycycline in a Drosophila melanogaster Model of Alzheimer Disease

Central and peripheral circadian clocks and their role in Alzheimer's disease

Co-aggregation with Apolipoprotein E modulates the function of Amyloid-β in Alzheimer's disease

Endoplasmic Reticulum-associated Degradation (ERAD) and Autophagy Cooperate to Degrade Polymerogenic Mutant Serpins

Iron is a specific cofactor for distinct oxidation- and aggregation-dependent Aβ toxicity mechanisms in a Drosophila model

Systematic in vivo analysis of the intrinsic determinants of amyloid Beta pathogenicity

The TRiC/CCT chaperone is implicated in Alzheimer's disease based on patient GWAS and an RNAi screen in Aβ-expressing Caenorhabditis elegans

The central molecular clock is robust in the face of behavioural arrhythmia in a Drosophila model of Alzheimer's disease

p53 and Translation Attenuation Regulate Distinct Cell Cycle Checkpoints during Endoplasmic Reticulum (ER) Stress

Ovine PrP transgenic Drosophila show reduced locomotor activity and decreased survival

Impaired tissue growth is mediated by checkpoint kinase 1 (CHK1) in the integrated stress response

Prion protein stabilizes amyloid-β (Aβ) oligomers and enhances Aβ neurotoxicity in a Drosophila model of Alzheimer's disease

Iron Promotes the Toxicity of Amyloid β Peptide by Impeding Its Ordered Aggregation

Mutant Neuroserpin (S49P) That Causes Familial Encephalopathy with Neuroserpin Inclusion Bodies Is a Poor Proteinase Inhibitor and Readily Forms Polymers in Vitro

Alzheimer's disease: insights from Drosophila melanogaster models

Utilizing microphysiological systems and induced pluripotent stem cells for disease modeling: a case study for blood brain barrier research in a pharmaceutical setting

The pro-domains of neurotrophins, including BDNF, are linked to Alzheimer's disease through a toxic synergy with Aβ

Protein misfolding and disease: from the test tube to the organism

Expression of the alternative oxidase mitigates beta-amyloid production and toxicity in model systems

Metabolic changes may precede proteostatic dysfunction in a Drosophila model of amyloid β peptide toxicity

Fenton chemistry and oxidative stress mediate the toxicity of the β-amyloid peptide in a Drosophila model of Alzheimer's disease

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