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The role of mitogen activated protein kinase signaling in microglia and neurons in the initiation and maintenance of chronic pain

Experimental neurology, 2012-04, Vol.234 (2), p.330-339

Crown, Eric D.

2012

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HC-030031, a TRPA1 Selective Antagonist, Attenuates Inflammatory- and Neuropathy-Induced Mechanical Hypersensitivity

Molecular pain, 2008-10, Vol.4 (1), p.48-48

2008

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Activation of p38 MAP kinase is involved in central neuropathic pain following spinal cord injury

Experimental neurology, 2008-10, Vol.213 (2), p.257-267

2008

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Propentofylline attenuates allodynia, glial activation and modulates GABAergic tone after spinal cord injury in the rat

Pain (Amsterdam), 2008-08, Vol.138 (2), p.410-422

2008

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Efficacy and Safety of Glecaprevir/Pibrentasvir in Korean Patients with Chronic Hepatitis C: A Pooled Analysis of Five Phase II/III Trials

Gut and Liver, 2021, 15(6), , pp.895-903

2021

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Mechanisms of chronic central neuropathic pain after spinal cord injury

Brain Research Reviews, 2009-04, Vol.60 (1), p.202-213

2009

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Changes in Motoneuron Properties and Synaptic Inputs Related to Step Training after Spinal Cord Transection in Rats

The Journal of neuroscience, 2007-04, Vol.27 (16), p.4460-4471

2007

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Maladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury

Frontiers in physiology, 2012-01, Vol.3, p.399-399

2012

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Central nociceptive sensitization vs. spinal cord training: opposing forms of plasticity that dictate function after complete spinal cord injury

Frontiers in physiology, 2012-01, Vol.3, p.396-396

2012

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Impact of behavioral control on the processing of nociceptive stimulation

Frontiers in physiology, 2012-01, Vol.3, p.262-262

2012

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Increases in the activated forms of ERK 1/2, p38 MAPK, and CREB are correlated with the expression of at-level mechanical allodynia following spinal cord injury

Experimental neurology, 2006-06, Vol.199 (2), p.397-407

2006

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Calcium/calmodulin dependent kinase II contributes to persistent central neuropathic pain following spinal cord injury

Pain (Amsterdam), 2012-03, Vol.153 (3), p.710-721

2012

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BOLD and blood volume-weighted fMRI of rat lumbar spinal cord during non-noxious and noxious electrical hindpaw stimulation

NeuroImage (Orlando, Fla.), 2008-03, Vol.40 (1), p.133-147

2008

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Uncontrollable stimulation undermines recovery after spinal cord injury

Journal of neurotrauma, 2004-12, Vol.21 (12), p.1795-1817

2004

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Evidence that descending serotonergic systems protect spinal cord plasticity against the disruptive effect of uncontrollable stimulation

Experimental neurology, 2005-11, Vol.196 (1), p.164-176

2005

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Two chronic motor training paradigms differentially influence acute instrumental learning in spinally transected rats

Behavioural brain research, 2007-06, Vol.180 (1), p.95-101

2007

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Instrumental Learning Within the Spinal Cord: IV. Induction and Retention of the Behavioral Deficit Observed After Noncontingent Shock

Behavioral neuroscience, 2002-12, Vol.116 (6), p.1032-1051

2002

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Instrumental learning within the spinal cord: underlying mechanisms and implications for recovery after injury

Behavioral & cognitive neuroscience reviews, 2006-12, Vol.5 (4), p.191-239

2006

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Instrumental learning within the spinal cord: II. Evidence for central mediation

Physiology & behavior, 2002-11, Vol.77 (2), p.259-267

2002

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Preserving and Restoring Behavioral Potential Within the Spinal Cord Using an Instrumental Training Paradigm

Journal of neurophysiology, 2001-08, Vol.86 (2), p.845-855

2001

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Instrumental Learning Within the Rat Spinal Cord: Localization of the Essential Neural Circuit

Behavioral neuroscience, 2005-04, Vol.119 (2), p.538-547

2005

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Instrumental learning within the spinal cord: V. Evidence the behavioral deficit observed after noncontingent nociceptive stimulation reflects an intraspinal modification

Behavioural brain research, 2003-05, Vol.141 (2), p.159-170

2003

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GABAA Receptor Activation Is Involved in Noncontingent Shock Inhibition of Instrumental Conditioning in Spinal Rats

Behavioral neuroscience, 2003-08, Vol.117 (4), p.799-812

2003

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The Behavioral Deficit Observed Following Noncontingent Shock in Spinalized Rats Is Prevented by the Protein Synthesis Inhibitor Cycloheximide

Behavioral neuroscience, 2004-06, Vol.118 (3), p.653-658

2004

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2966 A Sub-Analysis of Puerto Rican Hepatitis C Infected Patients Enrolled in the Phase 2 and 3 Glecaprevir-Pibrentasvir Clinical Program

The American journal of gastroenterology, 2019-10, Vol.114 (1), p.S1612-S1612

2019

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The role of mitogen activated protein kinase signaling in microglia and neurons in the initiation and maintenance of chronic pain

HC-030031, a TRPA1 Selective Antagonist, Attenuates Inflammatory- and Neuropathy-Induced Mechanical Hypersensitivity

Activation of p38 MAP kinase is involved in central neuropathic pain following spinal cord injury

Propentofylline attenuates allodynia, glial activation and modulates GABAergic tone after spinal cord injury in the rat

Efficacy and Safety of Glecaprevir/Pibrentasvir in Korean Patients with Chronic Hepatitis C: A Pooled Analysis of Five Phase II/III Trials

Mechanisms of chronic central neuropathic pain after spinal cord injury

Changes in Motoneuron Properties and Synaptic Inputs Related to Step Training after Spinal Cord Transection in Rats

Maladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury

Central nociceptive sensitization vs. spinal cord training: opposing forms of plasticity that dictate function after complete spinal cord injury

Impact of behavioral control on the processing of nociceptive stimulation

Increases in the activated forms of ERK 1/2, p38 MAPK, and CREB are correlated with the expression of at-level mechanical allodynia following spinal cord injury

Calcium/calmodulin dependent kinase II contributes to persistent central neuropathic pain following spinal cord injury

BOLD and blood volume-weighted fMRI of rat lumbar spinal cord during non-noxious and noxious electrical hindpaw stimulation

Uncontrollable stimulation undermines recovery after spinal cord injury

Evidence that descending serotonergic systems protect spinal cord plasticity against the disruptive effect of uncontrollable stimulation

Two chronic motor training paradigms differentially influence acute instrumental learning in spinally transected rats

Instrumental Learning Within the Spinal Cord: IV. Induction and Retention of the Behavioral Deficit Observed After Noncontingent Shock

Instrumental learning within the spinal cord: underlying mechanisms and implications for recovery after injury

Instrumental learning within the spinal cord: II. Evidence for central mediation

Preserving and Restoring Behavioral Potential Within the Spinal Cord Using an Instrumental Training Paradigm

Instrumental Learning Within the Rat Spinal Cord: Localization of the Essential Neural Circuit

Instrumental learning within the spinal cord: V. Evidence the behavioral deficit observed after noncontingent nociceptive stimulation reflects an intraspinal modification

GABAA Receptor Activation Is Involved in Noncontingent Shock Inhibition of Instrumental Conditioning in Spinal Rats

The Behavioral Deficit Observed Following Noncontingent Shock in Spinalized Rats Is Prevented by the Protein Synthesis Inhibitor Cycloheximide

2966 A Sub-Analysis of Puerto Rican Hepatitis C Infected Patients Enrolled in the Phase 2 and 3 Glecaprevir-Pibrentasvir Clinical Program

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