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ALS-associated TDP-43 induces endoplasmic reticulum stress, which drives cytoplasmic TDP-43 accumulation and stress granule formation

PloS one, 2013-11, Vol.8 (11), p.e81170-e81170

2013

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Therapeutic Redistribution of Metal Ions To Treat Alzheimer’s Disease

Accounts of chemical research, 2012-09, Vol.45 (9), p.1604-1611

2012

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CuII(atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease

British journal of pharmacology, 2020-02, Vol.177 (3), p.656-667

2020

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Superoxide Dismutase 1 in Health and Disease: How a Frontline Antioxidant Becomes Neurotoxic

Angewandte Chemie (International ed.), 2021-04, Vol.60 (17), p.9215-9246

2021

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CuII(atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Neuropathology and applied neurobiology, 2023-10, Vol.49 (5), p.e12938-n/a

2023

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Metal ionophore treatment restores dendritic spine density and synaptic protein levels in a mouse model of Alzheimer's disease

PloS one, 2011-03, Vol.6 (3), p.e17669

2011

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Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SODG93A mice co-expressing the Copper-Chaperone-for-SOD

Neurobiology of disease, 2016-05, Vol.89, p.1-9

2016

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SLN124, a GalNac‐siRNA targeting transmembrane serine protease 6, in combination with deferiprone therapy reduces ineffective erythropoiesis and hepatic iron‐overload in a mouse model of β‐thalassaemia

British journal of haematology, 2021-07, Vol.194 (1), p.200-210

2021

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Comment: Cu-ATSM to treat and image amyotrophic lateral sclerosis

Neurology, 2015-05, Vol.84 (20), p.2038

Crouch, Peter J

2015

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Cu II (atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Neuropathology and applied neurobiology, 2023-10, Vol.49 (5), p.e12938

2023

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Metal-deficient SOD1 in amyotrophic lateral sclerosis

Journal of molecular medicine (Berlin, Germany), 2015-05, Vol.93 (5), p.481-487

2015

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CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord

Scientific reports, 2017-02, Vol.7 (1), p.42292-42292, Article 42292

2017

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Neuroprotective copper bis(thiosemicarbazonato) complexes promote neurite elongation

PloS one, 2014-02, Vol.9 (2), p.e90070-e90070

2014

Volltextzugriff (PDF)

Fundamental Neurochemistry Review: Copper availability as a potential therapeutic target in progressive supranuclear palsy: Insight from other neurodegenerative diseases

Journal of neurochemistry, 2023-11, Vol.167 (3), p.337-346

2023

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Evidence for disrupted copper availability in human spinal cord supports CuII(atsm) as a treatment option for sporadic cases of ALS

Scientific reports, 2024-03, Vol.14 (1), p.5929-5929, Article 5929

2024

Volltextzugriff (PDF)

Altered SOD1 maturation and post-translational modification in amyotrophic lateral sclerosis spinal cord

Brain (London, England : 1878), 2022-09, Vol.145 (9), p.3108-3130

2022

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The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

Experimental neurology, 2018-09, Vol.307, p.118-128

2018

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Quantification of metallothionein-III in brain tissues using liquid chromatography tandem mass spectrometry

Analytical biochemistry, 2021-10, Vol.630, p.114326-114326, Article 114326

2021

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Kinase Inhibitor Screening Identifies Cyclin-Dependent Kinases and Glycogen Synthase Kinase 3 as Potential Modulators of TDP-43 Cytosolic Accumulation during Cell Stress

PloS one, 2013-06, Vol.8 (6), p.e67433

2013

Volltextzugriff (PDF)

TDP-43 mutations causing amyotrophic lateral sclerosis are associated with altered expression of RNA-binding protein hnRNP K and affect the Nrf2 antioxidant pathway

Human molecular genetics, 2017-05, Vol.26 (9), p.1732-1746

2017

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Regular Physical Exercise Modulates Iron Homeostasis in the 5xFAD Mouse Model of Alzheimer’s Disease

International journal of molecular sciences, 2021-08, Vol.22 (16), p.8715

2021

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Prion protein “gamma-cleavage”: characterizing a novel endoproteolytic processing event

Cellular and molecular life sciences : CMLS, 2016-02, Vol.73 (3), p.667-683

2016

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Potential Diagnostic Imaging of Alzheimer’s Disease with Copper-64 Complexes That Bind to Amyloid‑β Plaques

Inorganic chemistry, 2019-03, Vol.58 (5), p.3382-3395

2019

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Circumventing the Crabtree Effect: A method to induce lactate consumption and increase oxidative phosphorylation in cell culture

The international journal of biochemistry & cell biology, 2016-10, Vol.79, p.128-138

2016

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ALS-associated TDP-43 induces endoplasmic reticulum stress, which drives cytoplasmic TDP-43 accumulation and stress granule formation

Therapeutic Redistribution of Metal Ions To Treat Alzheimer’s Disease

CuII(atsm) inhibits ferroptosis: Implications for treatment of neurodegenerative disease

TDP-43 Triggers Mitochondrial DNA Release via mPTP to Activate cGAS/STING in ALS

Superoxide Dismutase 1 in Health and Disease: How a Frontline Antioxidant Becomes Neurotoxic

CuII(atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Metal ionophore treatment restores dendritic spine density and synaptic protein levels in a mouse model of Alzheimer's disease

Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SODG93A mice co-expressing the Copper-Chaperone-for-SOD

SLN124, a GalNac‐siRNA targeting transmembrane serine protease 6, in combination with deferiprone therapy reduces ineffective erythropoiesis and hepatic iron‐overload in a mouse model of β‐thalassaemia

Comment: Cu-ATSM to treat and image amyotrophic lateral sclerosis

Cu II (atsm) significantly decreases microglial reactivity in patients with sporadic amyotrophic lateral sclerosis

Metal-deficient SOD1 in amyotrophic lateral sclerosis

CuII(atsm) improves the neurological phenotype and survival of SOD1G93A mice and selectively increases enzymatically active SOD1 in the spinal cord

Neuroprotective copper bis(thiosemicarbazonato) complexes promote neurite elongation

Fundamental Neurochemistry Review: Copper availability as a potential therapeutic target in progressive supranuclear palsy: Insight from other neurodegenerative diseases

Evidence for disrupted copper availability in human spinal cord supports CuII(atsm) as a treatment option for sporadic cases of ALS

Altered SOD1 maturation and post-translational modification in amyotrophic lateral sclerosis spinal cord

The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

Quantification of metallothionein-III in brain tissues using liquid chromatography tandem mass spectrometry

Kinase Inhibitor Screening Identifies Cyclin-Dependent Kinases and Glycogen Synthase Kinase 3 as Potential Modulators of TDP-43 Cytosolic Accumulation during Cell Stress

TDP-43 mutations causing amyotrophic lateral sclerosis are associated with altered expression of RNA-binding protein hnRNP K and affect the Nrf2 antioxidant pathway

Regular Physical Exercise Modulates Iron Homeostasis in the 5xFAD Mouse Model of Alzheimer’s Disease

Prion protein “gamma-cleavage”: characterizing a novel endoproteolytic processing event

Potential Diagnostic Imaging of Alzheimer’s Disease with Copper-64 Complexes That Bind to Amyloid‑β Plaques

Circumventing the Crabtree Effect: A method to induce lactate consumption and increase oxidative phosphorylation in cell culture

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