UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Type V Collagen in Scar Tissue Regulates the Size of Scar after Heart Injury

Cell, 2020-08, Vol.182 (3), p.545-562.e23

2020

Volltextzugriff (PDF)

The Development of Robust Antibodies to Sarcospan, a Dystrophin- and Integrin-Associated Protein, for Basic and Translational Research

International journal of molecular sciences, 2024-06, Vol.25 (11), p.6121

2024

Volltextzugriff (PDF)

Mitochondrial Dysfunction Is an Early Consequence of Partial or Complete Dystrophin Loss in mdx Mice

Frontiers in physiology, 2020-06, Vol.11, p.690

2020

Volltextzugriff (PDF)

Quantitative immuno-mass spectrometry imaging of skeletal muscle dystrophin

Scientific reports, 2021-01, Vol.11 (1), p.1128-1128, Article 1128

2021

Volltextzugriff (PDF)

Multi-omics analysis of sarcospan overexpression in mdx skeletal muscle reveals compensatory remodeling of cytoskeleton-matrix interactions that promote mechanotransduction pathways

Skeletal muscle, 2023-01, Vol.13 (1), p.1-18, Article 1

2023

Volltextzugriff (PDF)

Modulating the extracellular matrix to treat wound healing defects in Ehlers-Danlos syndrome

iScience, 2024-09, Vol.27 (9), p.110676, Article 110676

2024

Volltextzugriff (PDF)

Development of a high-throughput screen to identify small molecule enhancers of sarcospan for the treatment of Duchenne muscular dystrophy

Skeletal muscle, 2019-12, Vol.9 (1), p.32-32, Article 32

2019

Volltextzugriff (PDF)

Sarcospan Regulates Cardiac Isoproterenol Response and Prevents Duchenne Muscular Dystrophy-Associated Cardiomyopathy

Journal of the American Heart Association, 2015-12, Vol.4 (12), p.n/a

2015

Volltextzugriff (PDF)

Emerging genetic therapies to treat Duchenne muscular dystrophy

Current opinion in neurology, 2009-10, Vol.22 (5), p.532-538

2009

Volltextzugriff (PDF)

High-throughput screening identifies modulators of sarcospan that stabilize muscle cells and exhibit activity in the mouse model of Duchenne muscular dystrophy

Skeletal muscle, 2020-09, Vol.10 (1), p.26-26, Article 26

2020

Volltextzugriff (PDF)

Myoscaffolds reveal laminin scarring is detrimental for stem cell function while sarcospan induces compensatory fibrosis

npj Regenerative medicine, 2023-03, Vol.8 (1), p.16-22, Article 16

2023

Volltextzugriff (PDF)

Student Satisfaction and Learning Outcomes in Asynchronous Online Lecture Videos

CBE life sciences education, 2019-12, Vol.18 (4), p.ar55-ar55

2019

Volltextzugriff (PDF)

Sarcospan increases laminin-binding capacity of α-dystroglycan to ameliorate DMD independent of Galgt2

Human molecular genetics, 2022-03, Vol.31 (5), p.718-732

2022

Volltextzugriff (PDF)

Spp1 (osteopontin) promotes TGFβ processing in fibroblasts of dystrophin-deficient muscles through matrix metalloproteinases

Human molecular genetics, 2019-10, Vol.28 (20), p.3431-3442

2019

Volltextzugriff (PDF)

Stabilization of the cardiac sarcolemma by sarcospan rescues DMD-associated cardiomyopathy

JCI insight, 2019-06, Vol.5 (11)

2019

Volltextzugriff (PDF)

Sarcospan-dependent Akt activation is required for utrophin expression and muscle regeneration

The Journal of cell biology, 2012-06, Vol.197 (7), p.1009-1027

2012

Volltextzugriff (PDF)

Myogenic Akt signaling upregulates the utrophin–glycoprotein complex and promotes sarcolemma stability in muscular dystrophy

Human molecular genetics, 2009-01, Vol.18 (2), p.318-327

2009

Volltextzugriff (PDF)

Benfotiamine improves dystrophic pathology and exercise capacity in mdx mice by reducing inflammation and fibrosis

Human molecular genetics, 2024-05, Vol.33 (15), p.1339-1355

2024

Volltextzugriff (PDF)

High-Throughput Screening to Identify Modulators of Sarcospan

Methods in molecular biology (Clifton, N.J.), 2023, Vol.2587, p.479

2023

Volltextzugriff (PDF)

Dystrophin and utrophin expression require sarcospan: loss of α7 integrin exacerbates a newly discovered muscle phenotype in sarcospan-null mice

Human molecular genetics, 2012-10, Vol.21 (20), p.4378-4393

2012

Volltextzugriff (PDF)

Engineering multicomponent tissue by spontaneous adhesion of myogenic and adipogenic microtissues cultured with customized scaffolds

Food research international, 2023-10, Vol.172, p.113080, Article 113080

2023

Volltextzugriff (PDF)

Loss of sarcospan exacerbates pathology in mdx mice, but does not affect utrophin amelioration of disease

Human molecular genetics, 2021-04, Vol.30 (3-4), p.149-159

2021

Volltextzugriff (PDF)

The extracellular matrix differentially directs myoblast motility and differentiation in distinct forms of muscular dystrophy: Dystrophic matrices alter myoblast motility

Matrix biology, 2024-05, Vol.129, p.44-58

2024

Volltextzugriff (PDF)

Loss of sarcolemma nNOS in sarcoglycan‐deficient muscle

The FASEB journal, 2002-11, Vol.16 (13), p.1786-1791

2002

Volltextzugriff (PDF)

The potential of sarcospan in adhesion complex replacement therapeutics for the treatment of muscular dystrophy

The FEBS journal, 2013-09, Vol.280 (17), p.4210-4229

2013

Volltextzugriff (PDF)

1 – 25 von 69

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(67)
Mit Volltextzugriff
(65)

Publikationsform

Artikel
(69)

Sprache

Englisch
(69)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Type V Collagen in Scar Tissue Regulates the Size of Scar after Heart Injury

The Development of Robust Antibodies to Sarcospan, a Dystrophin- and Integrin-Associated Protein, for Basic and Translational Research

Mitochondrial Dysfunction Is an Early Consequence of Partial or Complete Dystrophin Loss in mdx Mice

Quantitative immuno-mass spectrometry imaging of skeletal muscle dystrophin

Multi-omics analysis of sarcospan overexpression in mdx skeletal muscle reveals compensatory remodeling of cytoskeleton-matrix interactions that promote mechanotransduction pathways

Modulating the extracellular matrix to treat wound healing defects in Ehlers-Danlos syndrome

Development of a high-throughput screen to identify small molecule enhancers of sarcospan for the treatment of Duchenne muscular dystrophy

Sarcospan Regulates Cardiac Isoproterenol Response and Prevents Duchenne Muscular Dystrophy-Associated Cardiomyopathy

Emerging genetic therapies to treat Duchenne muscular dystrophy

High-throughput screening identifies modulators of sarcospan that stabilize muscle cells and exhibit activity in the mouse model of Duchenne muscular dystrophy

Myoscaffolds reveal laminin scarring is detrimental for stem cell function while sarcospan induces compensatory fibrosis

Student Satisfaction and Learning Outcomes in Asynchronous Online Lecture Videos

Sarcospan increases laminin-binding capacity of α-dystroglycan to ameliorate DMD independent of Galgt2

Spp1 (osteopontin) promotes TGFβ processing in fibroblasts of dystrophin-deficient muscles through matrix metalloproteinases

Stabilization of the cardiac sarcolemma by sarcospan rescues DMD-associated cardiomyopathy

Sarcospan-dependent Akt activation is required for utrophin expression and muscle regeneration

Myogenic Akt signaling upregulates the utrophin–glycoprotein complex and promotes sarcolemma stability in muscular dystrophy

Benfotiamine improves dystrophic pathology and exercise capacity in mdx mice by reducing inflammation and fibrosis

High-Throughput Screening to Identify Modulators of Sarcospan

Dystrophin and utrophin expression require sarcospan: loss of α7 integrin exacerbates a newly discovered muscle phenotype in sarcospan-null mice

Engineering multicomponent tissue by spontaneous adhesion of myogenic and adipogenic microtissues cultured with customized scaffolds

Loss of sarcospan exacerbates pathology in mdx mice, but does not affect utrophin amelioration of disease

The extracellular matrix differentially directs myoblast motility and differentiation in distinct forms of muscular dystrophy: Dystrophic matrices alter myoblast motility

Loss of sarcolemma nNOS in sarcoglycan‐deficient muscle

The potential of sarcospan in adhesion complex replacement therapeutics for the treatment of muscular dystrophy

Suchergebnisse filtern