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β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4515-4525

2014

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Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study

Neoplasia (New York, N.Y.), 2012-08, Vol.14 (8), p.742-IN21

2012

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Wilms tumor genetics: Mutations in WT1, WTX, and CTNNB1 account for only about one-third of tumors

Genes chromosomes & cancer, 2008-06, Vol.47 (6), p.461-470

2008

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Role of Cyclooxygenase-2 Pathway in Creating an Immunosuppressive Microenvironment and in Initiation and Progression of Wilms' Tumor

Neoplasia (New York, N.Y.), 2017-03, Vol.19 (3), p.237-249

2017

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WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Journal of clinical oncology, 2011-02, Vol.29 (6), p.698-703

2011

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Ubiquitin specific protease 18 (Usp18) is a WT1 transcriptional target

Experimental cell research, 2013-03, Vol.319 (5), p.612-622

2013

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Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

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Rare variant detection using family-based sequencing analysis

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (10), p.3985-3990

2013

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Loss of Heterozygosity at 2q37 in Sporadic Wilms' Tumor: Putative Role for miR-562

Clinical cancer research, 2009-10, Vol.15 (19), p.5985-5992

2009

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Generation of a Wt1 conditional deletion, nuclear red fluorescent protein reporter allele in the mouse

Differentiation (London), 2024-07, Vol.138, p.100791, Article 100791

2024

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Familial Wilms tumor

American journal of medical genetics. Part C, Seminars in medical genetics, 2004-08, Vol.129C (1), p.29-34

2004

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Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

Journal of medical genetics, 2016-06, Vol.53 (6), p.385-388

2016

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Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation

The Journal of clinical investigation, 2011-01, Vol.121 (1), p.174-183

2011

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Absence of PPP2R1A mutations in Wilms tumor

Oncogene, 2001-04, Vol.20 (16), p.2050-2054

2001

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Genome-wide loss of heterozygosity analysis of WT1-wild-type and WT1-mutant Wilms tumors

Genes chromosomes & cancer, 2005-06, Vol.43 (2), p.172-180

2005

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Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Journal of medical genetics, 2015-11, Vol.53 (6), p.385-388

2015

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Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

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Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

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Pulmonary dysplasia, Denys-Drash syndrome and wilms tumor 1 gene mutation in twins

Pediatric nephrology (Berlin, West), 2001-03, Vol.16 (3), p.227-231

2001

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Reply to B. Royer-Pokora et al

Journal of clinical oncology, 2011-06, Vol.29 (16), p.e487-e488

2011

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Identification and characterization of transcription termination sites in the Escherichia coli lacZ gene

Journal of molecular biology, 1989-07, Vol.208 (1), p.23-43

1989

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Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study12

Neoplasia (New York, N.Y.), 2012-08, Vol.14 (8), p.742-756

2012

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Three different polypeptides are necessary for DNA binding of the mammalian heteromeric CCAAT binding factor

The Journal of biological chemistry, 1992-08, Vol.267 (23), p.16574-16580

1992

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Genome-wide loss of heterozygosity analysis ofWT1-wild-type andWT1-mutant Wilms tumors

Genes chromosomes & cancer, 2005, Vol.43 (2), p.172-180

2005

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Purification of BBF, a DNA-binding protein recognizing a positive cis-acting element in the mouse α1(III) collagen promoter

The Journal of biological chemistry, 1992-07, Vol.267 (20), p.14398-14404

1992

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β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study

Wilms tumor genetics: Mutations in WT1, WTX, and CTNNB1 account for only about one-third of tumors

Role of Cyclooxygenase-2 Pathway in Creating an Immunosuppressive Microenvironment and in Initiation and Progression of Wilms' Tumor

WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Ubiquitin specific protease 18 (Usp18) is a WT1 transcriptional target

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Rare variant detection using family-based sequencing analysis

Loss of Heterozygosity at 2q37 in Sporadic Wilms' Tumor: Putative Role for miR-562

Generation of a Wt1 conditional deletion, nuclear red fluorescent protein reporter allele in the mouse

Familial Wilms tumor

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation

Absence of PPP2R1A mutations in Wilms tumor

Genome-wide loss of heterozygosity analysis of WT1-wild-type and WT1-mutant Wilms tumors

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Pulmonary dysplasia, Denys-Drash syndrome and wilms tumor 1 gene mutation in twins

Reply to B. Royer-Pokora et al

Identification and characterization of transcription termination sites in the Escherichia coli lacZ gene

Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study12

Three different polypeptides are necessary for DNA binding of the mammalian heteromeric CCAAT binding factor

Genome-wide loss of heterozygosity analysis ofWT1-wild-type andWT1-mutant Wilms tumors

Purification of BBF, a DNA-binding protein recognizing a positive cis-acting element in the mouse α1(III) collagen promoter

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