UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Acid-sensing ion channel-1 contributes to axonal degeneration in autoimmune inflammation of the central nervous system

Nature medicine, 2007-12, Vol.13 (12), p.1483-1489

2007

Volltextzugriff (PDF)

Acid-sensing ion channel 1 is involved in both axonal injury and demyelination in multiple sclerosis and its animal model

Brain (London, England : 1878), 2011-02, Vol.134 (Pt 2), p.571-584

2011

Volltextzugriff (PDF)

IL-21 and IL-21 Receptor Expression in Lymphocytes and Neurons in Multiple Sclerosis Brain

The American journal of pathology, 2011-02, Vol.178 (2), p.794-802

2011

Volltextzugriff (PDF)

Altered Sodium Channel Expression in Second-Order Spinal Sensory Neurons Contributes to Pain after Peripheral Nerve Injury

The Journal of neuroscience, 2004-05, Vol.24 (20), p.4832-4839

2004

Volltextzugriff (PDF)

Changes of sodium channel expression in experimental painful diabetic neuropathy

Annals of neurology, 2002-12, Vol.52 (6), p.786-792

2002

Volltextzugriff (PDF)

Interleukin-17 Production in Central Nervous System-Infiltrating T Cells and Glial Cells Is Associated with Active Disease in Multiple Sclerosis

The American journal of pathology, 2008, Vol.172 (1), p.146-155

2008

Volltextzugriff (PDF)

Contribution of Fibrinogen to Inflammation and Neuronal Density in Human Traumatic Brain Injury

Journal of neurotrauma, 2018-10, Vol.35 (19), p.2259-2271

2018

Volltextzugriff (PDF)

Foveal changes in aquaporin‐4 antibody seropositive neuromyelitis optica spectrum disorder are independent of optic neuritis and not overtly progressive

European journal of neurology, 2021-07, Vol.28 (7), p.2280-2293

2021

Volltextzugriff (PDF)

Co‐localization of sodium channel Nav1.6 and the sodium–calcium exchanger at sites of axonal injury in the spinal cord in EAE

Brain (London, England : 1878), 2004-02, Vol.127 (2), p.294-303

2004

Volltextzugriff (PDF)

Opposing effects of HLA class I molecules in tuning autoreactive CD8 + T cells in multiple sclerosis

Nature Medicine, 2008-11, Vol.14 (11), p.1227-1235

2008

Volltextzugriff (PDF)

Upregulation of Sodium Channel Nav1.3 and Functional Involvement in Neuronal Hyperexcitability Associated with Central Neuropathic Pain after Spinal Cord Injury

The Journal of neuroscience, 2003-10, Vol.23 (26), p.8881-8892

2003

Volltextzugriff (PDF)

Preferential expression of IGF-I in small DRG neurons and down-regulation following injury

Neuroreport, 2002-09, Vol.13 (13), p.1649-1652

2002

Volltextzugriff (PDF)

Sodium channels contribute to microglia/macrophage activation and function in EAE and MS

Glia, 2005-01, Vol.49 (2), p.220-229

2005

Volltextzugriff (PDF)

Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination

Brain (London, England : 1878), 2003-07, Vol.126 (7), p.1552-1561

2003

Volltextzugriff (PDF)

Apoptosis of vasopressinergic hypothalamic neurons in chronic diabetes mellitus

Neurobiology of disease, 2004-03, Vol.15 (2), p.221-228

2004

Volltextzugriff (PDF)

Na + channel expression along axons in multiple sclerosis and its models

Trends in pharmacological sciences (Regular ed.), 2004-11, Vol.25 (11), p.584-591

2004

Volltextzugriff (PDF)

Contactin regulates the current density and axonal expression of tetrodotoxin-resistant but not tetrodotoxin-sensitive sodium channels in DRG neurons

The European journal of neuroscience, 2005-07, Vol.22 (1), p.39-49

2005

Volltextzugriff (PDF)

Molecular Changes in Neurons in Multiple Sclerosis: Altered Axonal Expression of Nav1.2 and Nav1.6 Sodium Channels and Na+/ Ca2+Exchanger

Proceedings of the National Academy of Sciences - PNAS, 2004-05, Vol.101 (21), p.8168-8173

2004

Volltextzugriff (PDF)

Abnormal Purkinje cell activity in vivo in experimental allergic encephalomyelitis

Experimental brain research, 2004-09, Vol.158 (1), p.1-8

2004

Volltextzugriff (PDF)

Annexin II/pII is up-regulated in Purkinje cells in EAE and MS

Neuroreport, 2003-03, Vol.14 (4), p.555-558

2003

Volltextzugriff (PDF)

Temporal Course of Upregulation of Nav1.8 in Purkinje Neurons Parallels the Progression of Clinical Deficit in Experimental Allergic Encephalomyelitis

Journal of neuropathology and experimental neurology, 2003-09, Vol.62 (9), p.968-975

2003

Volltextzugriff (PDF)

Co-Localization of Sodium Channel Na[v]1.6 and the Sodium--Calcium Exchanger at Sites of Axonal Injury in the Spinal Cord in EAE

Brain (London, England : 1878), 2004-02, Vol.127 (2), p.294

2004

Volltextzugriff (PDF)

Upregulation and colocalization of p75 and Nav1.8 in Purkinje neurons in experimental autoimmune encephalomyelitis

Neuroscience letters, 2004-10, Vol.369 (3), p.186-190

2004

Volltextzugriff (PDF)

Fibroblast Growth Factor Homologous Factor 2B: Association with Nav1.6 and Selective Colocalization at Nodes of Ranvier of Dorsal Root Axons

The Journal of neuroscience, 2004-07, Vol.24 (30), p.6765-6775

2004

Volltextzugriff (PDF)

Sodium channel expression in hypothalamic osmosensitive neurons in experimental diabetes

Neuroreport, 2002-08, Vol.13 (11), p.1481-1484

2002

Volltextzugriff (PDF)

1 – 25 von 47

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(46)
Mit Volltextzugriff
(44)

Publikationsform

Artikel
(47)

Sprache

Englisch
(47)

Erscheinungsjahr

n.n

Thema

Life Sciences & Biomedicine
(27)
Science & Technology
(27)
Animals
(21)
Neurosciences & Neurology
(18)
Neurosciences
(17)
Humans
(16)
Multiple Sclerosis
(15)
Neurology
(15)
Biological And Medical Sciences
(13)
Male
(13)
Sodium Channels - Metabolism
(13)
Mice
(12)
Medical Sciences
(11)
Multiple Sclerosis - Metabolism
(11)
Clinical Neurology
(10)
Disease Models, Animal
(10)
Encephalomyelitis, Autoimmune, Experimental - Metabolism
(9)
Female
(8)
Middle Aged
(8)
Adult
(7)

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Acid-sensing ion channel-1 contributes to axonal degeneration in autoimmune inflammation of the central nervous system

Acid-sensing ion channel 1 is involved in both axonal injury and demyelination in multiple sclerosis and its animal model

IL-21 and IL-21 Receptor Expression in Lymphocytes and Neurons in Multiple Sclerosis Brain

Altered Sodium Channel Expression in Second-Order Spinal Sensory Neurons Contributes to Pain after Peripheral Nerve Injury

Changes of sodium channel expression in experimental painful diabetic neuropathy

Interleukin-17 Production in Central Nervous System-Infiltrating T Cells and Glial Cells Is Associated with Active Disease in Multiple Sclerosis

Contribution of Fibrinogen to Inflammation and Neuronal Density in Human Traumatic Brain Injury

Foveal changes in aquaporin‐4 antibody seropositive neuromyelitis optica spectrum disorder are independent of optic neuritis and not overtly progressive

Co‐localization of sodium channel Nav1.6 and the sodium–calcium exchanger at sites of axonal injury in the spinal cord in EAE

Opposing effects of HLA class I molecules in tuning autoreactive CD8 + T cells in multiple sclerosis

Upregulation of Sodium Channel Nav1.3 and Functional Involvement in Neuronal Hyperexcitability Associated with Central Neuropathic Pain after Spinal Cord Injury

Preferential expression of IGF-I in small DRG neurons and down-regulation following injury

Sodium channels contribute to microglia/macrophage activation and function in EAE and MS

Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination

Apoptosis of vasopressinergic hypothalamic neurons in chronic diabetes mellitus

Na + channel expression along axons in multiple sclerosis and its models

Contactin regulates the current density and axonal expression of tetrodotoxin-resistant but not tetrodotoxin-sensitive sodium channels in DRG neurons

Molecular Changes in Neurons in Multiple Sclerosis: Altered Axonal Expression of Nav1.2 and Nav1.6 Sodium Channels and Na+/ Ca2+Exchanger

Abnormal Purkinje cell activity in vivo in experimental allergic encephalomyelitis

Annexin II/pII is up-regulated in Purkinje cells in EAE and MS

Temporal Course of Upregulation of Nav1.8 in Purkinje Neurons Parallels the Progression of Clinical Deficit in Experimental Allergic Encephalomyelitis

Co-Localization of Sodium Channel Na[v]1.6 and the Sodium--Calcium Exchanger at Sites of Axonal Injury in the Spinal Cord in EAE

Upregulation and colocalization of p75 and Nav1.8 in Purkinje neurons in experimental autoimmune encephalomyelitis

Fibroblast Growth Factor Homologous Factor 2B: Association with Nav1.6 and Selective Colocalization at Nodes of Ranvier of Dorsal Root Axons

Sodium channel expression in hypothalamic osmosensitive neurons in experimental diabetes

Suchergebnisse filtern