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The contribution of astrocytes and microglia to traumatic brain injury

British journal of pharmacology, 2016-02, Vol.173 (4), p.692-702

2016

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The contribution of neuroinflammation to amyloid toxicity in Alzheimer's disease

Journal of neurochemistry, 2016-02, Vol.136 (3), p.457-474

2016

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COPD and stroke: are systemic inflammation and oxidative stress the missing links?

Clinical science (1979), 2016-07, Vol.130 (13), p.1039

2016

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The influence of neuroinflammation in Autism Spectrum Disorder

Brain, behavior, and immunity, 2019-07, Vol.79, p.75-90

2019

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Neuroinflammation and oxidative stress: Co-conspirators in the pathology of Parkinson’s disease

Neurochemistry international, 2013-04, Vol.62 (5), p.803-819

2013

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STING-mediated type-I interferons contribute to the neuroinflammatory process and detrimental effects following traumatic brain injury

Journal of neuroinflammation, 2018-11, Vol.15 (1), p.323-323, Article 323

2018

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Modulation of neuro-inflammation and vascular response by oxidative stress following cerebral ischemia-reperfusion injury

Current medicinal chemistry, 2008, Vol.15 (1), p.1

2008

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The involvement of microglia in Alzheimer's disease: a new dog in the fight

British journal of pharmacology, 2019-09, Vol.176 (18), p.3533-3543

2019

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Type-I interferon pathway in neuroinflammation and neurodegeneration: focus on Alzheimer’s disease

Journal of Neural Transmission, 2018-05, Vol.125 (5), p.797-807

2018

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Neural injury following stroke: are Toll‐like receptors the link between the immune system and the CNS?

British journal of pharmacology, 2010-08, Vol.160 (8), p.1872-1888

2010

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Type-1 interferon signaling mediates neuro-inflammatory events in models of Alzheimer's disease

Neurobiology of aging, 2014-05, Vol.35 (5), p.1012-1023

2014

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Type-1 interferons contribute to the neuroinflammatory response and disease progression of the MPTP mouse model of Parkinson's disease

Glia, 2016-09, Vol.64 (9), p.1590-1604

2016

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Inflammation in epileptogenesis after traumatic brain injury

Journal of neuroinflammation, 2017-01, Vol.14 (1), p.10-10, Article 10

2017

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An altered glial phenotype in the NL3R451C mouse model of autism

Scientific reports, 2020-09, Vol.10 (1), p.14492-14492, Article 14492

2020

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The Complexity of the cGAS-STING Pathway in CNS Pathologies

Frontiers in neuroscience, 2021-02, Vol.15, p.621501-621501

2021

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Targeting high-mobility group box protein 1 (HMGB1) in pediatric traumatic brain injury: Chronic neuroinflammatory, behavioral, and epileptogenic consequences

Experimental neurology, 2019-10, Vol.320, p.112979-112979, Article 112979

2019

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STING-Mediated Autophagy Is Protective against H2O2-Induced Cell Death

International journal of molecular sciences, 2020-09, Vol.21 (19), p.7059

2020

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Reactive oxygen species and the modulation of stroke

Free radical biology & medicine, 2005-06, Vol.38 (11), p.1433-1444

2005

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Pharmacological inhibition of STING reduces neuroinflammation‐mediated damage post‐traumatic brain injury

British journal of pharmacology, 2024-09, Vol.181 (17), p.3118-3135

2024

Volltextzugriff (PDF)

Reactive Oxygen Species Enhance Insulin Sensitivity

Cell metabolism, 2009-10, Vol.10 (4), p.260-272

2009

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Age‐dependent release of high‐mobility group box protein‐1 and cellular neuroinflammation after traumatic brain injury in mice

Journal of comparative neurology (1911), 2019-04, Vol.527 (6), p.1102-1117

2019

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Abrogation of type-I interferon signalling alters the microglial response to Aβ1–42

Scientific reports, 2020-02, Vol.10 (1), p.3153, Article 3153

2020

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Anti-lysophosphatidic acid antibodies improve traumatic brain injury outcomes

Journal of neuroinflammation, 2014-02, Vol.11 (1), p.37-37

2014

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The use of bioactive matrices in regenerative therapies for traumatic brain injury

Acta biomaterialia, 2020-01, Vol.102, p.1-12

2020

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Glutathione peroxidase-1 primes pro-inflammatory cytokine production after LPS challenge in vivo

PloS one, 2012-03, Vol.7 (3), p.e33172-e33172

2012

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The contribution of astrocytes and microglia to traumatic brain injury

The contribution of neuroinflammation to amyloid toxicity in Alzheimer's disease

COPD and stroke: are systemic inflammation and oxidative stress the missing links?

The influence of neuroinflammation in Autism Spectrum Disorder

Neuroinflammation and oxidative stress: Co-conspirators in the pathology of Parkinson’s disease

STING-mediated type-I interferons contribute to the neuroinflammatory process and detrimental effects following traumatic brain injury

Modulation of neuro-inflammation and vascular response by oxidative stress following cerebral ischemia-reperfusion injury

The involvement of microglia in Alzheimer's disease: a new dog in the fight

Type-I interferon pathway in neuroinflammation and neurodegeneration: focus on Alzheimer’s disease

Neural injury following stroke: are Toll‐like receptors the link between the immune system and the CNS?

Type-1 interferon signaling mediates neuro-inflammatory events in models of Alzheimer's disease

Type-1 interferons contribute to the neuroinflammatory response and disease progression of the MPTP mouse model of Parkinson's disease

Inflammation in epileptogenesis after traumatic brain injury

An altered glial phenotype in the NL3R451C mouse model of autism

The Complexity of the cGAS-STING Pathway in CNS Pathologies

Targeting high-mobility group box protein 1 (HMGB1) in pediatric traumatic brain injury: Chronic neuroinflammatory, behavioral, and epileptogenic consequences

STING-Mediated Autophagy Is Protective against H2O2-Induced Cell Death

Reactive oxygen species and the modulation of stroke

Pharmacological inhibition of STING reduces neuroinflammation‐mediated damage post‐traumatic brain injury

Reactive Oxygen Species Enhance Insulin Sensitivity

Age‐dependent release of high‐mobility group box protein‐1 and cellular neuroinflammation after traumatic brain injury in mice

Abrogation of type-I interferon signalling alters the microglial response to Aβ1–42

Anti-lysophosphatidic acid antibodies improve traumatic brain injury outcomes

The use of bioactive matrices in regenerative therapies for traumatic brain injury

Glutathione peroxidase-1 primes pro-inflammatory cytokine production after LPS challenge in vivo

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