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IL-33 drives group 2 innate lymphoid cell-mediated protection during Clostridium difficile infection

Nature communications, 2019-06, Vol.10 (1), p.2712-13, Article 2712

2019

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Mechanisms by which sialylated milk oligosaccharides impact bone biology in a gnotobiotic mouse model of infant undernutrition

Proceedings of the National Academy of Sciences - PNAS, 2019-06, Vol.116 (24), p.11988-11996

2019

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Microbiota-Regulated IL-25 Increases Eosinophil Number to Provide Protection during Clostridium difficile Infection

Cell reports (Cambridge), 2016-07, Vol.16 (2), p.432-443

2016

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Colonization during a key developmental window reveals microbiota-dependent shifts in growth and immunity during undernutrition

Microbiome, 2024-04, Vol.12 (1), p.71-71, Article 71

2024

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The binary toxin CDT enhances Clostridium difficile virulence by suppressing protective colonic eosinophilia

Nature microbiology, 2016-07, Vol.1 (8), p.16108, Article 16108

2016

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Inflammasome activation contributes to interleukin-23 production in response to Clostridium difficile

mBio, 2015-01, Vol.6 (1)

2015

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Cryptosporidium Priming Is More Effective than Vaccine for Protection against Cryptosporidiosis in a Murine Protein Malnutrition Model

PLoS neglected tropical diseases, 2016-07, Vol.10 (7), p.e0004820-e0004820

2016

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Role of Eosinophils and Tumor Necrosis Factor Alpha in Interleukin-25-Mediated Protection from Amebic Colitis

mBio, 2017-02, Vol.8 (1)

2017

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Environmental enteric dysfunction: gut and microbiota adaptation in pregnancy and infancy

Nature reviews. Gastroenterology & hepatology, 2023-04, Vol.20 (4), p.223-237

2023

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Bifidobacterium infantis treatment promotes weight gain in Bangladeshi infants with severe acute malnutrition

Science translational medicine, 2022-04, Vol.14 (640), p.eabk1107

2022

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Growing up: A NOD2 our microbes

Cell host & microbe, 2023-05, Vol.31 (5), p.685-687

2023

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Colitis-Induced Th17 Cells Increase the Risk for Severe Subsequent Clostridium difficile Infection

Cell host & microbe, 2019-05, Vol.25 (5), p.756-765.e5

2019

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Glucosylation Drives the Innate Inflammatory Response to Clostridium difficile Toxin A

Infection and immunity, 2016-08, Vol.84 (8), p.2317-2323

2016

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Role of Serum Amyloid A, Granulocyte-Macrophage Colony-Stimulating Factor, and Bone Marrow Granulocyte-Monocyte Precursor Expansion in Segmented Filamentous Bacterium-Mediated Protection from Entamoeba histolytica

Infection and immunity, 2016-10, Vol.84 (10), p.2824-2832

2016

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Host recognition of Clostridium difficile and the innate immune response

Anaerobe, 2014-12, Vol.30, p.205-209

2014

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Abstract 3090: Oral Administration Of Akkermansia Muciniphila Ameliorates Venous Wall Fibrosis: Therapeutic Implication Of A Novel Probiotic Therapy Against Post-thrombotic Syndrome

Arteriosclerosis, thrombosis, and vascular biology, 2024-05, Vol.44 (Suppl_1)

2024

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Microbiota-regulated IL-25 increases eosinophil number to provide 1 protection during Clostridium difficile infection

Cell reports (Cambridge), 2016-06, Vol.16 (2), p.432-443

2016

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Effects of microbiota-directed foods in gnotobiotic animals and undernourished children

Science (American Association for the Advancement of Science), 2019-07, Vol.365 (6449)

2019

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Transplantation of gut microbiota from old mice into young healthy mice reduces lean mass but not bone mass

Gut microbes, 2023, Vol.15 (1), p.2236755

2023

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Bone marrow dendritic cells from mice with an altered microbiota provide interleukin 17A-dependent protection against Entamoeba histolytica colitis

mBio, 2014-11, Vol.5 (6), p.e01817-e01817

2014

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Microbiota regulated IL-25 protects from Clostridium difficile infection via eosinophils

The Journal of immunology (1950), 2016-05, Vol.196 (1_Supplement), p.131-131.7

2016

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IL-33 drives group 2 innate lymphoid cell-mediated protection during Clostridium difficile infection

Mechanisms by which sialylated milk oligosaccharides impact bone biology in a gnotobiotic mouse model of infant undernutrition

Microbiota-Regulated IL-25 Increases Eosinophil Number to Provide Protection during Clostridium difficile Infection

Colonization during a key developmental window reveals microbiota-dependent shifts in growth and immunity during undernutrition

The binary toxin CDT enhances Clostridium difficile virulence by suppressing protective colonic eosinophilia

Inflammasome activation contributes to interleukin-23 production in response to Clostridium difficile

Cryptosporidium Priming Is More Effective than Vaccine for Protection against Cryptosporidiosis in a Murine Protein Malnutrition Model

Role of Eosinophils and Tumor Necrosis Factor Alpha in Interleukin-25-Mediated Protection from Amebic Colitis

Environmental enteric dysfunction: gut and microbiota adaptation in pregnancy and infancy

Bifidobacterium infantis treatment promotes weight gain in Bangladeshi infants with severe acute malnutrition

Growing up: A NOD2 our microbes

Colitis-Induced Th17 Cells Increase the Risk for Severe Subsequent Clostridium difficile Infection

Glucosylation Drives the Innate Inflammatory Response to Clostridium difficile Toxin A

Role of Serum Amyloid A, Granulocyte-Macrophage Colony-Stimulating Factor, and Bone Marrow Granulocyte-Monocyte Precursor Expansion in Segmented Filamentous Bacterium-Mediated Protection from Entamoeba histolytica

Host recognition of Clostridium difficile and the innate immune response

Abstract 3090: Oral Administration Of Akkermansia Muciniphila Ameliorates Venous Wall Fibrosis: Therapeutic Implication Of A Novel Probiotic Therapy Against Post-thrombotic Syndrome

Microbiota-regulated IL-25 increases eosinophil number to provide 1 protection during Clostridium difficile infection

Effects of microbiota-directed foods in gnotobiotic animals and undernourished children

Transplantation of gut microbiota from old mice into young healthy mice reduces lean mass but not bone mass

Bone marrow dendritic cells from mice with an altered microbiota provide interleukin 17A-dependent protection against Entamoeba histolytica colitis

Microbiota regulated IL-25 protects from Clostridium difficile infection via eosinophils

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