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Targeting amphiregulin (AREG) derived from senescent stromal cells diminishes cancer resistance and averts programmed cell death 1 ligand (PD‐L1)‐mediated immunosuppression

Aging cell, 2019-12, Vol.18 (6), p.e13027-n/a

2019

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p53-dependent release of Alarmin HMGB1 is a central mediator of senescent phenotypes

The Journal of cell biology, 2013-05, Vol.201 (4), p.613-629

2013

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Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

PLoS biology, 2008-12, Vol.6 (12), p.2853-e301

2008

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Secretion of Vascular Endothelial Growth Factor by Primary Human Fibroblasts at Senescence

The Journal of biological chemistry, 2006-10, Vol.281 (40), p.29568-29574

2006

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Non‐canonical ATM/MRN activities temporally define the senescence secretory program

EMBO reports, 2020-10, Vol.21 (10), p.e50718-n/a

2020

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Targeting SPINK1 in the damaged tumour microenvironment alleviates therapeutic resistance

Nature communications, 2018-10, Vol.9 (1), p.4315-19, Article 4315

2018

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A protein interaction landscape of breast cancer

Science (American Association for the Advancement of Science), 2021-10, Vol.374 (6563), p.eabf3066-eabf3066

2021

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Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion

Nature cell biology, 2009-08, Vol.11 (8), p.973-979

2009

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DNA-SCARS: distinct nuclear structures that sustain damage-induced senescence growth arrest and inflammatory cytokine secretion

Journal of cell science, 2011-01, Vol.124 (1), p.68-81

2011

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A human-like senescence-associated secretory phenotype is conserved in mouse cells dependent on physiological oxygen

PloS one, 2010-02, Vol.5 (2), p.e9188-e9188

2010

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Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation

Journal of cell science, 2005-02, Vol.118 (3), p.485-496

2005

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Mapping phospho-catalytic dependencies of therapy-resistant tumours reveals actionable vulnerabilities

Nature cell biology, 2019-06, Vol.21 (6), p.778

2019

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A collection of breast cancer cell lines for the study of functionally distinct cancer subtypes

Cancer cell, 2006-12, Vol.10 (6), p.515-527

2006

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Ionizing radiation‐induced long‐term expression of senescence markers in mice is independent of p53 and immune status

Aging cell, 2010-06, Vol.9 (3), p.398-409

2010

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Targetable mechanisms driving immunoevasion of persistent senescent cells link chemotherapy-resistant cancer to aging

JCI insight, 2019-07, Vol.5 (14)

2019

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Hyperphosphorylation of BCL-2 family proteins underlies functional resistance to venetoclax in lymphoid malignancies

The Journal of clinical investigation, 2023-11, Vol.133 (22), p.1-20

2023

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Activation-induced cytidine deaminase (AID) is necessary for the epithelial–mesenchymal transition in mammary epithelial cells

Proceedings of the National Academy of Sciences - PNAS, 2013-08, Vol.110 (32), p.E2977-E2986

2013

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B-cells and regulatory T-cells in the microenvironment of HER2+ breast cancer are associated with decreased survival: a real-world analysis of women with HER2+ metastatic breast cancer

Breast cancer research : BCR, 2023-10, Vol.25 (1), p.1-117, Article 117

2023

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Senescent cells as a source of inflammatory factors for tumor progression

Cancer and metastasis reviews, 2010-06, Vol.29 (2), p.273-283

2010

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Cellular Senescence: The Sought or the Unwanted?

Trends in molecular medicine, 2018-10, Vol.24 (10), p.871-885

2018

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The senescence-associated secretory phenotype: the dark side of tumor suppression

Annual review of pathology, 2010-01, Vol.5 (1), p.99-118

2010

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An Atlas of the Human Kinome Reveals the Mutational Landscape Underlying Dysregulated Phosphorylation Cascades in Cancer

Cancer research (Chicago, Ill.), 2016-04, Vol.76 (7), p.1733-1745

2016

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Tumor suppressor and aging biomarker p16(INK4a) induces cellular senescence without the associated inflammatory secretory phenotype

The Journal of biological chemistry, 2011-10, Vol.286 (42), p.36396

2011

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Id-1 as a Molecular Target in Therapy for Breast Cancer Cell Invasion and Metastasis

Proceedings of the National Academy of Sciences - PNAS, 2003-11, Vol.100 (23), p.13543-13548

2003

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Id-1 and Id-2 Proteins as Molecular Markers for Human Prostate Cancer Progression

Clinical cancer research, 2004-03, Vol.10 (6), p.2044-2051

2004

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Targeting amphiregulin (AREG) derived from senescent stromal cells diminishes cancer resistance and averts programmed cell death 1 ligand (PD‐L1)‐mediated immunosuppression

p53-dependent release of Alarmin HMGB1 is a central mediator of senescent phenotypes

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

Secretion of Vascular Endothelial Growth Factor by Primary Human Fibroblasts at Senescence

Non‐canonical ATM/MRN activities temporally define the senescence secretory program

Targeting SPINK1 in the damaged tumour microenvironment alleviates therapeutic resistance

A protein interaction landscape of breast cancer

Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion

DNA-SCARS: distinct nuclear structures that sustain damage-induced senescence growth arrest and inflammatory cytokine secretion

A human-like senescence-associated secretory phenotype is conserved in mouse cells dependent on physiological oxygen

Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation

Mapping phospho-catalytic dependencies of therapy-resistant tumours reveals actionable vulnerabilities

A collection of breast cancer cell lines for the study of functionally distinct cancer subtypes

Ionizing radiation‐induced long‐term expression of senescence markers in mice is independent of p53 and immune status

Targetable mechanisms driving immunoevasion of persistent senescent cells link chemotherapy-resistant cancer to aging

Hyperphosphorylation of BCL-2 family proteins underlies functional resistance to venetoclax in lymphoid malignancies

Activation-induced cytidine deaminase (AID) is necessary for the epithelial–mesenchymal transition in mammary epithelial cells

B-cells and regulatory T-cells in the microenvironment of HER2+ breast cancer are associated with decreased survival: a real-world analysis of women with HER2+ metastatic breast cancer

Senescent cells as a source of inflammatory factors for tumor progression

Cellular Senescence: The Sought or the Unwanted?

The senescence-associated secretory phenotype: the dark side of tumor suppression

An Atlas of the Human Kinome Reveals the Mutational Landscape Underlying Dysregulated Phosphorylation Cascades in Cancer

Tumor suppressor and aging biomarker p16(INK4a) induces cellular senescence without the associated inflammatory secretory phenotype

Id-1 as a Molecular Target in Therapy for Breast Cancer Cell Invasion and Metastasis

Id-1 and Id-2 Proteins as Molecular Markers for Human Prostate Cancer Progression

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