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Oxidative stress and proinflammatory cytokines contribute to demyelination and axonal damage in a cerebellar culture model of neuroinflammation

PloS one, 2013-02, Vol.8 (2), p.e54722

2013

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Phagocytic clearance of presynaptic dystrophies by reactive astrocytes in Alzheimer's disease

Glia, 2018-03, Vol.66 (3), p.637-653

2018

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Intracellular pathways involved in cell survival are deregulated in mouse and human spinal muscular atrophy motoneurons

Neurobiology of disease, 2021-07, Vol.155, p.105366-105366, Article 105366

2021

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Reelin Regulates the Maturation of Dendritic Spines, Synaptogenesis and Glial Ensheathment of Newborn Granule Cells

Cerebral cortex (New York, N.Y. 1991), 2016-10, Vol.26 (11), p.4282-4298

2016

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FAIM-L - SIVA-1: Two Modulators of XIAP in Non-Apoptotic Caspase Function

Frontiers in cell and developmental biology, 2022-01, Vol.9, p.826037-826037

2022

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The Absence of FAIM Leads to a Delay in Dark Adaptation and Hampers Arrestin-1 Translocation upon Light Reception in the Retina

Cells (Basel, Switzerland), 2023-02, Vol.12 (3), p.487

2023

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Identification and characterization of new isoforms of human fas apoptotic inhibitory molecule (FAIM)

PloS one, 2017-10, Vol.12 (10), p.e0185327-e0185327

2017

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Chromatin Collapse during Caspase-dependent Apoptotic Cell Death Requires DNA Fragmentation Factor, 40-kDa Subunit-/Caspase-activated Deoxyribonuclease-mediated 3′-OH Single-strand DNA Breaks

The Journal of biological chemistry, 2013-03, Vol.288 (13), p.9200-9215

2013

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6-Hydroxydopamine activates the mitochondrial apoptosis pathway through p38 MAPK-mediated, p53-independent activation of Bax and PUMA

Journal of neurochemistry, 2008-03, Vol.104 (6), p.1599-1612

2008

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FIB/SEM technology and high-throughput 3D reconstruction of dendritic spines and synapses in GFP-labeled adult-generated neurons

Frontiers in neuroanatomy, 2015-05, Vol.9, p.60-60

2015

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FAIM-L regulation of XIAP degradation modulates Synaptic Long-Term Depression and Axon Degeneration

Scientific reports, 2016-10, Vol.6 (1), p.35775-35775, Article 35775

2016

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FAIM Is Regulated by MiR-206, MiR-1-3p and MiR-133b

Frontiers in cell and developmental biology, 2020-12, Vol.8, p.584606-584606

2020

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Ubiquitination of TrkA by Nedd4‐2 regulates receptor lysosomal targeting and mediates receptor signaling

Journal of neurochemistry, 2011-05, Vol.117 (3), p.479-493

2011

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EndoG links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes

PloS one, 2011-03, Vol.6 (3), p.e17998-e17998

2011

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Activation of caspase-8 by tumour necrosis factor receptor 1 is necessary for caspase-3 activation and apoptosis in oxygen–glucose deprived cultured cortical cells

Neurobiology of disease, 2009-09, Vol.35 (3), p.438-447

2009

Volltextzugriff (PDF)

A new model to study spinal muscular atrophy: Neurite degeneration and cell death is counteracted by BCL-XL Overexpression in motoneurons

Neurobiology of disease, 2011-06, Vol.42 (3), p.415-426

2011

Volltextzugriff (PDF)

Neurodegeneration and neuroinflammation： two processes, one target

Neural regeneration research, 2015-10, Vol.10 (10), p.1581-1583

2015

Volltextzugriff (PDF)

Bcl-2 Is a Key Factor for Cardiac Fibroblast Resistance to Programmed Cell Death

The Journal of biological chemistry, 2004-08, Vol.279 (33), p.34882-34889

2004

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The Contribution of Apoptosis-inducing Factor, Caspase-activated DNase, and Inhibitor of Caspase-activated DNase to the Nuclear Phenotype and DNA Degradation during Apoptosis

The Journal of biological chemistry, 2005-10, Vol.280 (42), p.35670-35683

2005

Volltextzugriff (PDF)

The single AmphiTrk receptor highlights increased complexity of neurotrophin signalling in vertebrates and suggests an early role in developing sensory neuroepidermal cells

Development (Cambridge), 2005-05, Vol.132 (9), p.2191-2202

2005

Volltextzugriff (PDF)

Receptors of the Glial Cell Line-Derived Neurotrophic Factor Family of Neurotrophic Factors Signal Cell Survival through the Phosphatidylinositol 3-Kinase Pathway in Spinal Cord Motoneurons

The Journal of neuroscience, 1999-11, Vol.19 (21), p.9160-9169

1999

Volltextzugriff (PDF)

Reelin Induces the Detachment of Postnatal Subventricular Zone Cells and the Expression of the Egr-1 through Erk1/2 Activation

Cerebral cortex (New York, N.Y. 1991), 2007-02, Vol.17 (2), p.294-303

2007

Volltextzugriff (PDF)

Combining magnetic nanoparticles and icosahedral boron clusters in biocompatible inorganic nanohybrids for cancer therapy

Nanomedicine, 2019-08, Vol.20, p.101986-101986, Article 101986

2019

Volltextzugriff (PDF)

c-Src Is Required for Glial Cell Line-Derived Neurotrophic Factor (GDNF) Family Ligand-Mediated Neuronal Survival via a Phosphatidylinositol-3 Kinase (PI-3K)-Dependent Pathway

The Journal of neuroscience, 2001-03, Vol.21 (5), p.1464-1472

2001

Volltextzugriff (PDF)

Disruption of cortical cell type composition and function underlies diabetes-associated cognitive decline

Diabetologia, 2023-08, Vol.66 (8), p.1557-1575

2023

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Oxidative stress and proinflammatory cytokines contribute to demyelination and axonal damage in a cerebellar culture model of neuroinflammation

Phagocytic clearance of presynaptic dystrophies by reactive astrocytes in Alzheimer's disease

Intracellular pathways involved in cell survival are deregulated in mouse and human spinal muscular atrophy motoneurons

Reelin Regulates the Maturation of Dendritic Spines, Synaptogenesis and Glial Ensheathment of Newborn Granule Cells

FAIM-L - SIVA-1: Two Modulators of XIAP in Non-Apoptotic Caspase Function

The Absence of FAIM Leads to a Delay in Dark Adaptation and Hampers Arrestin-1 Translocation upon Light Reception in the Retina

Identification and characterization of new isoforms of human fas apoptotic inhibitory molecule (FAIM)

Chromatin Collapse during Caspase-dependent Apoptotic Cell Death Requires DNA Fragmentation Factor, 40-kDa Subunit-/Caspase-activated Deoxyribonuclease-mediated 3′-OH Single-strand DNA Breaks

6-Hydroxydopamine activates the mitochondrial apoptosis pathway through p38 MAPK-mediated, p53-independent activation of Bax and PUMA

FIB/SEM technology and high-throughput 3D reconstruction of dendritic spines and synapses in GFP-labeled adult-generated neurons

FAIM-L regulation of XIAP degradation modulates Synaptic Long-Term Depression and Axon Degeneration

FAIM Is Regulated by MiR-206, MiR-1-3p and MiR-133b

Ubiquitination of TrkA by Nedd4‐2 regulates receptor lysosomal targeting and mediates receptor signaling

EndoG links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes

Activation of caspase-8 by tumour necrosis factor receptor 1 is necessary for caspase-3 activation and apoptosis in oxygen–glucose deprived cultured cortical cells

A new model to study spinal muscular atrophy: Neurite degeneration and cell death is counteracted by BCL-XL Overexpression in motoneurons

Neurodegeneration and neuroinflammation： two processes, one target

Bcl-2 Is a Key Factor for Cardiac Fibroblast Resistance to Programmed Cell Death

The Contribution of Apoptosis-inducing Factor, Caspase-activated DNase, and Inhibitor of Caspase-activated DNase to the Nuclear Phenotype and DNA Degradation during Apoptosis

The single AmphiTrk receptor highlights increased complexity of neurotrophin signalling in vertebrates and suggests an early role in developing sensory neuroepidermal cells

Receptors of the Glial Cell Line-Derived Neurotrophic Factor Family of Neurotrophic Factors Signal Cell Survival through the Phosphatidylinositol 3-Kinase Pathway in Spinal Cord Motoneurons

Reelin Induces the Detachment of Postnatal Subventricular Zone Cells and the Expression of the Egr-1 through Erk1/2 Activation

Combining magnetic nanoparticles and icosahedral boron clusters in biocompatible inorganic nanohybrids for cancer therapy

c-Src Is Required for Glial Cell Line-Derived Neurotrophic Factor (GDNF) Family Ligand-Mediated Neuronal Survival via a Phosphatidylinositol-3 Kinase (PI-3K)-Dependent Pathway

Disruption of cortical cell type composition and function underlies diabetes-associated cognitive decline

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