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Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons

PLoS biology, 2010-01, Vol.8 (1), p.e1000300-e1000300

2010

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Traumatic Axonal Injury: Mechanisms and Translational Opportunities

Trends in neurosciences (Regular ed.), 2016-05, Vol.39 (5), p.311-324

2016

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Absence of SARM1 Rescues Development and Survival of NMNAT2-Deficient Axons

Cell reports (Cambridge), 2015-03, Vol.10 (12), p.1974-1981

2015

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Lipopolysaccharide-induced neuroinflammation induces presynaptic disruption through a direct action on brain tissue involving microglia-derived interleukin 1 beta

Journal of neuroinflammation, 2019-05, Vol.16 (1), p.106-106, Article 106

2019

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Subcellular localization determines the stability and axon protective capacity of axon survival factor Nmnat2

PLoS biology, 2013-04, Vol.11 (4), p.e1001539-e1001539

2013

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NAD + cleavage activity by animal and plant TIR domains in cell death pathways

Science (American Association for the Advancement of Science), 2019-08, Vol.365 (6455), p.793-799

2019

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Low levels of NMNAT2 compromise axon development and survival

Human molecular genetics, 2019-02, Vol.28 (3), p.448-458

2019

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Mitochondrial impairment activates the Wallerian pathway through depletion of NMNAT2 leading to SARM1-dependent axon degeneration

Neurobiology of disease, 2020-02, Vol.134, p.104678-104678, Article 104678

2020

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Rescue of peripheral and CNS axon defects in mice lacking NMNAT2

The Journal of neuroscience, 2013-08, Vol.33 (33), p.13410-13424

2013

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TDP-43 gains function due to perturbed autoregulation in a Tardbp knock-in mouse model of ALS-FTD

Nature neuroscience, 2018-04, Vol.21 (4), p.552-563

2018

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Adaptation of a Commercial NAD + Quantification Kit to Assay the Base-Exchange Activity and Substrate Preferences of SARM1

Molecules (Basel, Switzerland), 2024-02, Vol.29 (4), p.847

2024

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Natural variants of human SARM1 cause both intrinsic and dominant loss-of-function influencing axon survival

Scientific reports, 2022-08, Vol.12 (1), p.13846-14, Article 13846

2022

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Structural basis for RING-Cys-Relay E3 ligase activity and its role in axon integrity

Nature chemical biology, 2020-11, Vol.16 (11), p.1227-1236

2020

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Programmed axon degeneration: from mouse to mechanism to medicine

Nature reviews. Neuroscience, 2020-04, Vol.21 (4), p.183-196

2020

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SARM1 is a multi-functional NAD(P)ase with prominent base exchange activity, all regulated bymultiple physiologically relevant NAD metabolites

iScience, 2022-02, Vol.25 (2), p.103812-103812, Article 103812

2022

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NMNAT2 is a druggable target to drive neuronal NAD production

Nature communications, 2024-07, Vol.15 (1), p.6256-15, Article 6256

2024

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A Novel NAD Signaling Mechanism in Axon Degeneration and its Relationship to Innate Immunity

Frontiers in molecular biosciences, 2021-07, Vol.8, p.703532

2021

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Author Correction: NMNAT2 is a druggable target to drive neuronal NAD production

Nature communications, 2024-09, Vol.15 (1), p.8143-2, Article 8143

2024

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Mitochondrial dysfunction as a trigger of programmed axon death

Trends in neurosciences (Regular ed.), 2022-01, Vol.45 (1), p.53-63

2022

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Reduced number of axonal mitochondria and tau hypophosphorylation in mouse P301L tau knockin neurons

Neurobiology of disease, 2016-01, Vol.85, p.1-10

2016

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Wallerian degeneration: an emerging axon death pathway linking injury and disease

Nature reviews. Neuroscience, 2014-06, Vol.15 (6), p.394-409

2014

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Neurotoxin-mediated potent activation of the axon degeneration regulator SARM1

eLife, 2021-12, Vol.10

2021

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Axon Biology in ALS: Mechanisms of Axon Degeneration and Prospects for Therapy

Neurotherapeutics, 2022-07, Vol.19 (4), p.1133-1144

Coleman, Michael P.

2022

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Difference Tracker: ImageJ plugins for fully automated analysis of multiple axonal transport parameters

Journal of neuroscience methods, 2010-11, Vol.193 (2), p.281-287

2010

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Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons

Traumatic Axonal Injury: Mechanisms and Translational Opportunities

Absence of SARM1 Rescues Development and Survival of NMNAT2-Deficient Axons

Lipopolysaccharide-induced neuroinflammation induces presynaptic disruption through a direct action on brain tissue involving microglia-derived interleukin 1 beta

Subcellular localization determines the stability and axon protective capacity of axon survival factor Nmnat2

NAD + cleavage activity by animal and plant TIR domains in cell death pathways

Low levels of NMNAT2 compromise axon development and survival

Mitochondrial impairment activates the Wallerian pathway through depletion of NMNAT2 leading to SARM1-dependent axon degeneration

Rescue of peripheral and CNS axon defects in mice lacking NMNAT2

TDP-43 gains function due to perturbed autoregulation in a Tardbp knock-in mouse model of ALS-FTD

Adaptation of a Commercial NAD + Quantification Kit to Assay the Base-Exchange Activity and Substrate Preferences of SARM1

Natural variants of human SARM1 cause both intrinsic and dominant loss-of-function influencing axon survival

Structural basis for RING-Cys-Relay E3 ligase activity and its role in axon integrity

Programmed axon degeneration: from mouse to mechanism to medicine

SARM1 is a multi-functional NAD(P)ase with prominent base exchange activity, all regulated bymultiple physiologically relevant NAD metabolites

NMNAT2 is a druggable target to drive neuronal NAD production

A Novel NAD Signaling Mechanism in Axon Degeneration and its Relationship to Innate Immunity

Author Correction: NMNAT2 is a druggable target to drive neuronal NAD production

Mitochondrial dysfunction as a trigger of programmed axon death

Reduced number of axonal mitochondria and tau hypophosphorylation in mouse P301L tau knockin neurons

Enrichment of SARM1 alleles encoding variants with constitutively hyperactive NADase in patients with ALS and other motor nerve disorders

Wallerian degeneration: an emerging axon death pathway linking injury and disease

Neurotoxin-mediated potent activation of the axon degeneration regulator SARM1

Axon Biology in ALS: Mechanisms of Axon Degeneration and Prospects for Therapy

Difference Tracker: ImageJ plugins for fully automated analysis of multiple axonal transport parameters

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