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LIN-9 phosphorylation on threonine-96 is required for transcriptional activation of LIN-9 target genes and promotes cell cycle progression

PloS one, 2014-01, Vol.9 (1), p.e87620-e87620

2014

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Mip/LIN-9 Regulates the Expression of B-Myb and the Induction of Cyclin A, Cyclin B, and CDK1

The Journal of biological chemistry, 2007-01, Vol.282 (1), p.168-175

2007

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The Presumptive Phosphatidylserine Receptor Is Dispensable for Innate Anti-inflammatory Recognition and Clearance of Apoptotic Cells

The Journal of biological chemistry, 2006-03, Vol.281 (9), p.5718-5725

2006

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Contribution of the Box 1 and Box 2 Motifs of Cytokine Receptors to Jak1 Association and Activation

The Journal of biological chemistry, 2002-12, Vol.277 (50), p.48220-48226

2002

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Deletion of the p107/p130-binding domain of Mip130/LIN-9 bypasses the requirement for CDK4 activity for the dissociation of Mip130/LIN-9 from p107/p130-E2F4 complex

Experimental cell research, 2009-10, Vol.315 (17), p.2914-2920

2009

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Jak2 Is Essential for Signaling through a Variety of Cytokine Receptors

Cell, 1998-05, Vol.93 (3), p.385-395

1998

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The WD Motif-Containing Protein RACK-1 Functions as a Scaffold Protein Within the Type I IFN Receptor-Signaling Complex

The Journal of immunology (1950), 2003-09, Vol.171 (6), p.2989-2994

2003

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Different requirements for the cytostatic and apoptotic effects of type I interferons. Induction of apoptosis requires ARF but not p53 in osteosarcoma cell lines

The Journal of biological chemistry, 2004-07, Vol.279 (31), p.32275-32280

2004

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Mip/LIN-9 can inhibit cell proliferation independent of the pocket proteins

Blood cells, molecules, & diseases, 2007-11, Vol.39 (3), p.272-277

2007

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Two Distinct Domains Within the N-Terminal Region of Janus Kinase 1 Interact with Cytokine Receptors

The Journal of immunology (1950), 2002-08, Vol.169 (3), p.1302-1308

2002

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Role of the cytoplasmic domains of the type I interferon receptor subunits in signaling

Seminars in cancer biology, 2000-04, Vol.10 (2), p.83-92

2000

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A mutant allele of BARA/LIN-9 rescues the cdk4 −/− phenotype by releasing the repression on E2F-regulated genes

Experimental cell research, 2006-08, Vol.312 (13), p.2465-2475

2006

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Phosphatidylinositol 3-Kinase Confers Resistance to Encephalomyocarditis and Herpes Simplex Virus-Induced Cell Death Through the Activation of Distinct Downstream Effectors

The Journal of immunology (1950), 2001-10, Vol.167 (8), p.4553-4559

2001

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Different Requirements for the Cytostatic and Apoptotic Effects of Type I Interferons

The Journal of biological chemistry, 2004-07, Vol.279 (31), p.32275-32280

2004

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The vav proto-oncogene product (p95 vav) interacts with the Tyk-2 protein tyrosine kinase

FEBS letters, 1997-02, Vol.403 (1), p.31-34

1997

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Activation of the Jak-Stat pathway in cells that exhibit selective sensitivity to the antiviral effects of IFN-beta compared with IFN-alpha

Journal of interferon & cytokine research, 1999-07, Vol.19 (7), p.797-801

1999

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Interferon α activates the tyrosine kinase Lyn in haemopoietic cells

British journal of haematology, 1998-06, Vol.101 (3), p.446-449

1998

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Vaccinia Virus B18R Gene Encodes a Type I Interferon-binding Protein That Blocks Interferon α Transmembrane Signaling (∗)

The Journal of biological chemistry, 1995-07, Vol.270 (27), p.15974-15978

1995

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The type-I interferon receptor. The long and short of it

Cytokine & growth factor reviews, 1996-08, Vol.7 (2), p.143-151

1996

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Cloning and Expression of a Long Form of the β Subunit of the Interferon αβ Receptor That Is Required for Signaling (∗)

The Journal of biological chemistry, 1995-09, Vol.270 (37), p.21606-21611

1995

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ARF-induced downregulation of Mip130/LIN-9 protein levels mediates a positive feedback that leads to increased expression of p16Ink4a and p19Arf

Oncogene, 2010-04, Vol.29 (13), p.1976-1986

2010

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LIN-9 Phosphorylation on Threonine-96 Is Required for Transcriptional Activation of LIN-9 Target Genes and Promotes Cell Cycle Progression: e87620

PloS one, 2014-01, Vol.9 (1)

2014

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The IRS-pathway operates distinctively from the stat-pathway in hematopoietic cells and transduces common and distinct signals during engagement of the insulin or interferon-α receptors

Blood, 1997-10, Vol.90 (7), p.2574-2582

1997

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Tyrosine phosphorylation of the alpha and beta subunits of the type I interferon receptor. Interferon-beta selectively induces tyrosine phosphorylation of an alpha subunit-associated protein

The Journal of biological chemistry, 1994-07, Vol.269 (27), p.17761-17764

1994

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Identification of a novel subunit of the type I interferon receptor localized to human chromosome 21

The Journal of biological chemistry, 1993-05, Vol.268 (15), p.10895-10899

1993

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LIN-9 phosphorylation on threonine-96 is required for transcriptional activation of LIN-9 target genes and promotes cell cycle progression

Mip/LIN-9 Regulates the Expression of B-Myb and the Induction of Cyclin A, Cyclin B, and CDK1

The Presumptive Phosphatidylserine Receptor Is Dispensable for Innate Anti-inflammatory Recognition and Clearance of Apoptotic Cells

Contribution of the Box 1 and Box 2 Motifs of Cytokine Receptors to Jak1 Association and Activation

Deletion of the p107/p130-binding domain of Mip130/LIN-9 bypasses the requirement for CDK4 activity for the dissociation of Mip130/LIN-9 from p107/p130-E2F4 complex

Jak2 Is Essential for Signaling through a Variety of Cytokine Receptors

The WD Motif-Containing Protein RACK-1 Functions as a Scaffold Protein Within the Type I IFN Receptor-Signaling Complex

Different requirements for the cytostatic and apoptotic effects of type I interferons. Induction of apoptosis requires ARF but not p53 in osteosarcoma cell lines

Mip/LIN-9 can inhibit cell proliferation independent of the pocket proteins

Two Distinct Domains Within the N-Terminal Region of Janus Kinase 1 Interact with Cytokine Receptors

Role of the cytoplasmic domains of the type I interferon receptor subunits in signaling

A mutant allele of BARA/LIN-9 rescues the cdk4 −/− phenotype by releasing the repression on E2F-regulated genes

Phosphatidylinositol 3-Kinase Confers Resistance to Encephalomyocarditis and Herpes Simplex Virus-Induced Cell Death Through the Activation of Distinct Downstream Effectors

Different Requirements for the Cytostatic and Apoptotic Effects of Type I Interferons

The vav proto-oncogene product (p95 vav) interacts with the Tyk-2 protein tyrosine kinase

Activation of the Jak-Stat pathway in cells that exhibit selective sensitivity to the antiviral effects of IFN-beta compared with IFN-alpha

Interferon α activates the tyrosine kinase Lyn in haemopoietic cells

Vaccinia Virus B18R Gene Encodes a Type I Interferon-binding Protein That Blocks Interferon α Transmembrane Signaling (∗)

The type-I interferon receptor. The long and short of it

Cloning and Expression of a Long Form of the β Subunit of the Interferon αβ Receptor That Is Required for Signaling (∗)

ARF-induced downregulation of Mip130/LIN-9 protein levels mediates a positive feedback that leads to increased expression of p16Ink4a and p19Arf

LIN-9 Phosphorylation on Threonine-96 Is Required for Transcriptional Activation of LIN-9 Target Genes and Promotes Cell Cycle Progression: e87620

The IRS-pathway operates distinctively from the stat-pathway in hematopoietic cells and transduces common and distinct signals during engagement of the insulin or interferon-α receptors

Tyrosine phosphorylation of the alpha and beta subunits of the type I interferon receptor. Interferon-beta selectively induces tyrosine phosphorylation of an alpha subunit-associated protein

Identification of a novel subunit of the type I interferon receptor localized to human chromosome 21

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