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Direct binding of polymeric GBP1 to LPS disrupts bacterial cell envelope functions

The EMBO journal, 2020-07, Vol.39 (13), p.e104926-n/a

2020

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Heteroresistance: A cause of unexplained antibiotic treatment failure?

PLoS pathogens, 2019-06, Vol.15 (6), p.e1007726

2019

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Inflammasome Activation by Bacterial Outer Membrane Vesicles Requires Guanylate Binding Proteins

mBio, 2017-10, Vol.8 (5)

2017

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Human immunity to Toxoplasma gondii

PLoS pathogens, 2019-12, Vol.15 (12), p.e1008097-e1008097

2019

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Manipulating neutrophil degranulation as a bacterial virulence strategy

PLoS pathogens, 2020-12, Vol.16 (12), p.e1009054-e1009054

2020

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Interferon-Inducible E3 Ligase RNF213 Facilitates Host-Protective Linear and K63-Linked Ubiquitylation of Toxoplasma gondii Parasitophorous Vacuoles

mBio, 2022-10, Vol.13 (5), p.e0188822

2022

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The Chlamydia trachomatis Inclusion Membrane Protein CpoS Counteracts STING-Mediated Cellular Surveillance and Suicide Programs

Cell host & microbe, 2017-01, Vol.21 (1), p.113-121

2017

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The acetylase activity of Cdu1 regulates bacterial exit from infected cells by protecting Chlamydia effectors from degradation

eLife, 2024-02, Vol.12

2024

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Portrait of a killer: Uncovering resistance mechanisms and global spread of Acinetobacter baumannii

PLoS pathogens, 2023-08, Vol.19 (8), p.e1011520-e1011520

2023

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Galectin-3 directs antimicrobial guanylate binding proteins to vacuoles furnished with bacterial secretion systems

Proceedings of the National Academy of Sciences - PNAS, 2017-02, Vol.114 (9), p.E1698-E1706

2017

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IRG and GBP host resistance factors target aberrant, "non-self" vacuoles characterized by the missing of "self" IRGM proteins

PLoS pathogens, 2013-06, Vol.9 (6), p.e1003414

2013

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Constitutive Interferon Maintains GBP Expression Required for Release of Bacterial Components Upstream of Pyroptosis and Anti-DNA Responses

Cell reports (Cambridge), 2018-07, Vol.24 (1), p.155-168.e5

2018

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Chlamydia trachomatis induces disassembly of the primary cilium to promote the intracellular infection

PLoS pathogens, 2024-06, Vol.20 (6), p.e1012303

2024

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Human guanylate binding proteins: nanomachines orchestrating host defense

The FEBS journal, 2021-10, Vol.288 (20), p.5826-5849

2021

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Viral Replication Complexes Are Targeted by LC3-Guided Interferon-Inducible GTPases

Cell host & microbe, 2017-07, Vol.22 (1), p.74-85.e7

2017

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It's uncomplicated: Prevention of urinary tract infections in an era of increasing antibiotic resistance

PLoS pathogens, 2024-02, Vol.20 (2), p.e1011930-e1011930

2024

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What bacterial cell death teaches us about life

PLoS pathogens, 2022-10, Vol.18 (10), p.e1010879

2022

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Acyloxyacyl hydrolase promotes the resolution of lipopolysaccharide-induced acute lung injury

PLoS pathogens, 2017-06, Vol.13 (6), p.e1006436-e1006436

2017

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Impact of interkingdom microbial interactions in the vaginal tract

PLoS pathogens, 2024-03, Vol.20 (3), p.e1012018

2024

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Translational gene expression control in Chlamydia trachomatis

PloS one, 2022-01, Vol.17 (1), p.e0257259-e0257259

2022

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Chlamydia trachomatis inclusion membrane protein MrcA interacts with the inositol 1,4,5-trisphosphate receptor type 3 (ITPR3) to regulate extrusion formation

PLoS pathogens, 2018-03, Vol.14 (3), p.e1006911

2018

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Elimination of Chlamydia muridarum from the female reproductive tract is IL-12p40 dependent, but independent of Th1 and Th2 cells

PLoS pathogens, 2024-01, Vol.20 (1), p.e1011914-e1011914

2024

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The interplay between bile acids and mucosal adaptive immunity

PLoS pathogens, 2023-06, Vol.19 (6), p.e1011356

2023

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Bacterial filamentation during urinary tract infections

PLoS pathogens, 2022-12, Vol.18 (12), p.e1010950-e1010950

2022

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Can we microbe-manage our vitamin acquisition for better health?

PLoS pathogens, 2023-05, Vol.19 (5), p.e1011361-e1011361

2023

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Direct binding of polymeric GBP1 to LPS disrupts bacterial cell envelope functions

Heteroresistance: A cause of unexplained antibiotic treatment failure?

Inflammasome Activation by Bacterial Outer Membrane Vesicles Requires Guanylate Binding Proteins

Human immunity to Toxoplasma gondii

Manipulating neutrophil degranulation as a bacterial virulence strategy

Interferon-Inducible E3 Ligase RNF213 Facilitates Host-Protective Linear and K63-Linked Ubiquitylation of Toxoplasma gondii Parasitophorous Vacuoles

The Chlamydia trachomatis Inclusion Membrane Protein CpoS Counteracts STING-Mediated Cellular Surveillance and Suicide Programs

The acetylase activity of Cdu1 regulates bacterial exit from infected cells by protecting Chlamydia effectors from degradation

Portrait of a killer: Uncovering resistance mechanisms and global spread of Acinetobacter baumannii

Galectin-3 directs antimicrobial guanylate binding proteins to vacuoles furnished with bacterial secretion systems

IRG and GBP host resistance factors target aberrant, "non-self" vacuoles characterized by the missing of "self" IRGM proteins

Constitutive Interferon Maintains GBP Expression Required for Release of Bacterial Components Upstream of Pyroptosis and Anti-DNA Responses

Chlamydia trachomatis induces disassembly of the primary cilium to promote the intracellular infection

Human guanylate binding proteins: nanomachines orchestrating host defense

Viral Replication Complexes Are Targeted by LC3-Guided Interferon-Inducible GTPases

It's uncomplicated: Prevention of urinary tract infections in an era of increasing antibiotic resistance

What bacterial cell death teaches us about life

Acyloxyacyl hydrolase promotes the resolution of lipopolysaccharide-induced acute lung injury

Impact of interkingdom microbial interactions in the vaginal tract

Translational gene expression control in Chlamydia trachomatis

Chlamydia trachomatis inclusion membrane protein MrcA interacts with the inositol 1,4,5-trisphosphate receptor type 3 (ITPR3) to regulate extrusion formation

Elimination of Chlamydia muridarum from the female reproductive tract is IL-12p40 dependent, but independent of Th1 and Th2 cells

The interplay between bile acids and mucosal adaptive immunity

Bacterial filamentation during urinary tract infections

Can we microbe-manage our vitamin acquisition for better health?

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