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Metformin: Experimental and Clinical Evidence for a Potential Role in Emphysema Treatment

American journal of respiratory and critical care medicine, 2021-09, Vol.204 (6), p.651-666

2021

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Carvedilol inhibits EGF-mediated JB6 P+ colony formation through a mechanism independent of adrenoceptors

PloS one, 2019-05, Vol.14 (5), p.e0217038

2019

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Pharmacological Targeting of Mitochondria in Diabetic Kidney Disease

Pharmacological reviews, 2023-03, Vol.75 (2), p.250-262

2023

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The β2-adrenergic receptor agonist formoterol restores mitochondrial homeostasis in glucose-induced renal proximal tubule injury through separate integrated pathways

Biochemical pharmacology, 2023-03, Vol.209, p.115436-115436, Article 115436

2023

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Regulation of mitochondrial dynamics and energetics in the diabetic renal proximal tubule by the β2-adrenergic receptor agonist formoterol

American journal of physiology. Renal physiology, 2020-11, Vol.319 (5), p.F773-F779

2020

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Mitochondrial Fission and Fusion Dynamics are Regulated by Multiple Pathways in Renal Proximal Tubule Cells Treated with High Glucose

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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5-hydroxytryptamine 1F Receptor Agonist Induces Mitochondrial Biogenesis and Promotes Recovery from Spinal Cord Injury

The Journal of pharmacology and experimental therapeutics, 2020-02, Vol.372 (2), p.216-223

2020

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Regulation of mitochondrial dynamics and energetics in the diabetic renal proximal tubule by the β 2 -adrenergic receptor agonist formoterol

American journal of physiology. Renal physiology, 2020-11, Vol.319 (5), p.F773-F779

2020

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Prevention of Skin Carcinogenesis by the Non-β-blocking R-carvedilol Enantiomer

Cancer prevention research (Philadelphia, Pa.), 2021-05, Vol.14 (5), p.527-540

2021

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Phosphoproteome profiling provides insight into the mechanism of action for carvedilol‐mediated cancer prevention

Molecular carcinogenesis, 2018-08, Vol.57 (8), p.997-1007

2018

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Topically Applied Carvedilol Attenuates Solar Ultraviolet Radiation Induced Skin Carcinogenesis

Cancer prevention research (Philadelphia, Pa.), 2017-10, Vol.10 (10), p.598-606

2017

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Mitochondrial Fission and Fusion Dynamics Are Regulated by Multiple Pathways in Renal Proximal Tubule Cells Treated with High Glucose: PO0699

Journal of the American Society of Nephrology, 2021-10, Vol.32 (10S), p.250-250

2021

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The β2-Adrenergic Receptor Agonist Formoterol Restores Mitochondrial Dynamics and Energy Production in the Diabetic Renal Proximal Tubule: PO0897

Journal of the American Society of Nephrology, 2020-10, Vol.31 (10S), p.313-314

2020

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The β 2 -adrenergic receptor agonist formoterol restores mitochondrial homeostasis in glucose-induced renal proximal tubule injury through separate integrated pathways

Biochemical pharmacology, 2023-03, Vol.209, p.115436

2023

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Regulation of Mitochondrial Dynamics in the Diabetic Renal Proximal Tubule by the β2-Adrenergic Receptor Agonist Formoterol

Cleveland, Kristan H

2022

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The β‐blockers carvedilol and nebivolol inhibit neoplastic transformation through a β2‐adrenergic receptor independent mechanism

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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Abstract 2617: Protective effect of the β-blocker carvedilol on UVB-induced skin damage

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.2617-2617

2016

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Abstract 2617: Protective effect of the -blocker carvedilol on UVB-induced skin damage

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.2617-2617

2016

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β‐Adrenergic receptor‐biased β‐blockers for skin cancer prevention

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Abstract 1915: Effects of carvedilol, a β-adrenergic receptor antagonist, on UVB-induced skin cancer development and its possible mechanism of action

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.1915-1915

2015

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Metformin: Experimental and Clinical Evidence for a Potential Role in Emphysema Treatment

Carvedilol inhibits EGF-mediated JB6 P+ colony formation through a mechanism independent of adrenoceptors

Pharmacological Targeting of Mitochondria in Diabetic Kidney Disease

The β2-adrenergic receptor agonist formoterol restores mitochondrial homeostasis in glucose-induced renal proximal tubule injury through separate integrated pathways

Regulation of mitochondrial dynamics and energetics in the diabetic renal proximal tubule by the β2-adrenergic receptor agonist formoterol

Mitochondrial Fission and Fusion Dynamics are Regulated by Multiple Pathways in Renal Proximal Tubule Cells Treated with High Glucose

5-hydroxytryptamine 1F Receptor Agonist Induces Mitochondrial Biogenesis and Promotes Recovery from Spinal Cord Injury

Regulation of mitochondrial dynamics and energetics in the diabetic renal proximal tubule by the β 2 -adrenergic receptor agonist formoterol

Prevention of Skin Carcinogenesis by the Non-β-blocking R-carvedilol Enantiomer

Phosphoproteome profiling provides insight into the mechanism of action for carvedilol‐mediated cancer prevention

Topically Applied Carvedilol Attenuates Solar Ultraviolet Radiation Induced Skin Carcinogenesis

Mitochondrial Fission and Fusion Dynamics Are Regulated by Multiple Pathways in Renal Proximal Tubule Cells Treated with High Glucose: PO0699

The β2-Adrenergic Receptor Agonist Formoterol Restores Mitochondrial Dynamics and Energy Production in the Diabetic Renal Proximal Tubule: PO0897

The β 2 -adrenergic receptor agonist formoterol restores mitochondrial homeostasis in glucose-induced renal proximal tubule injury through separate integrated pathways

Regulation of Mitochondrial Dynamics in the Diabetic Renal Proximal Tubule by the β2-Adrenergic Receptor Agonist Formoterol

The β‐blockers carvedilol and nebivolol inhibit neoplastic transformation through a β2‐adrenergic receptor independent mechanism

Abstract 2617: Protective effect of the β-blocker carvedilol on UVB-induced skin damage

Abstract 2617: Protective effect of the -blocker carvedilol on UVB-induced skin damage

β‐Adrenergic receptor‐biased β‐blockers for skin cancer prevention

Abstract 1915: Effects of carvedilol, a β-adrenergic receptor antagonist, on UVB-induced skin cancer development and its possible mechanism of action

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