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AP-1 Transcription Factors as Regulators of Immune Responses in Cancer

Cancers, 2019-07, Vol.11 (7), p.1037

2019

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Down‐regulation of the cyclin‐dependent kinase inhibitor p57 is mediated by Jab1/Csn5 in hepatocarcinogenesis

Hepatology (Baltimore, Md.), 2016-03, Vol.63 (3), p.898-913

2016

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Sustained Activation of JNK/p38 MAPK Pathways in Response to Cisplatin Leads to Fas Ligand Induction and Cell Death in Ovarian Carcinoma Cells

The Journal of biological chemistry, 2003-05, Vol.278 (21), p.19245-19256

2003

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Constitutive control of AKT1 gene expression by JUNB/CJUN in ALK+ anaplastic large-cell lymphoma: a novel crosstalk mechanism

Leukemia, 2015-11, Vol.29 (11), p.2162-2172

2015

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Essential roles of Jab1 in cell survival, spontaneous DNA damage and DNA repair

Oncogene, 2010-11, Vol.29 (46), p.6125-6137

2010

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Withdrawal of Survival Factors Results in Activation of the JNK Pathway in Neuronal Cells Leading to Fas Ligand Induction and Cell Death

Molecular and Cellular Biology, 1999-01, Vol.19 (1), p.751-763

1999

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YAP1 Promotes Tumor Invasion and Metastasis in Nasopharyngeal Carcinoma with Hepatitis B Virus Infection

OncoTargets and therapy, 2020-01, Vol.13, p.5629-5642

2020

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The oncogenic JUNB/CD30 axis contributes to cell cycle deregulation in ALK+ anaplastic large cell lymphoma

British journal of haematology, 2014-11, Vol.167 (4), p.514-523

2014

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Selective activation of the JNK signaling cascade and c-Jun transcriptional activity by the small GTPases Rac and Cdc42Hs

Cell, 1995-06, Vol.81 (7), p.1147-1157

1995

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Mechanisms for Lysophosphatidic Acid-induced Cytokine Production in Ovarian Cancer Cells

The Journal of biological chemistry, 2004-03, Vol.279 (10), p.9653-9661

2004

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N-(4-hydroxyphenyl)retinamide-induced apoptosis triggered by reactive oxygen species is mediated by activation of MAPKs in head and neck squamous carcinoma cells

Oncogene, 2006-05, Vol.25 (19), p.2785-2794

2006

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Abstract P5-08-02: New target in the resistant mechanism to trastuzumab

Cancer research (Chicago, Ill.), 2013-12, Vol.73 (24_Supplement), p.P5-P5-08-02

2013

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Lasting N-Terminal Phosphorylation of c-Jun and Activation of c-Jun N-Terminal Kinases after Neuronal Injury

The Journal of neuroscience, 1998-07, Vol.18 (14), p.5124-5135

1998

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A new group of conserved coactivators that increase the specificity of AP-1 transcription factors

Nature (London), 1996-10, Vol.383 (6599), p.453-457

1996

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Induction of c‐fos expression through JNK‐mediated TCF/Elk‐1 phosphorylation

The EMBO journal, 1995-12, Vol.14 (23), p.5957-5964

1995

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Identification of a Dual Specificity Kinase That Activates the Jun Kinases and p38-Mpk2

Science (American Association for the Advancement of Science), 1995-04, Vol.268 (5208), p.286-290

1995

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Jun activation domain-binding protein 1 expression in breast cancer inversely correlates with the cell cycle inhibitor p27(Kip1)

Cancer research (Chicago, Ill.), 2003-06, Vol.63 (11), p.2977-2981

2003

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Macrophage migration inhibitory factor expression is increased in pituitary adenoma cell nuclei

Journal of endocrinology, 2003-01, Vol.176 (1), p.103-110

2003

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Activation of cAMP and mitogen responsive genes relies on a common nuclear factor

Nature (London), 1994-07, Vol.370 (6486), p.226-229

1994

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JunB expression is a common feature of CD30+ lymphomas and lymphomatoid papulosis

Modern pathology, 2005-10, Vol.18 (10), p.1365-1370

2005

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2-acetylaminofluorene up-regulates rat mdr1b expression through generating reactive oxygen species that activate NF-kappa B pathway

The Journal of biological chemistry, 2001-01, Vol.276 (1), p.413-420

2001

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Expression of phosphorylated p27(Kip1) protein and Jun activation domain-binding protein 1 in human pituitary tumors

The journal of clinical endocrinology and metabolism, 2002-06, Vol.87 (6), p.2635-2643

2002

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Jun activation domain binding protein 1 expression is associated with low p27(Kip1)levels in node-negative breast cancer

Clinical cancer research, 2003-11, Vol.9 (15), p.5652-5659

2003

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Expression of p27(Kip1) and c-Jun activation binding protein 1 are inversely correlated in systemic anaplastic large cell lymphoma

Clinical cancer research, 2003-03, Vol.9 (3), p.1121-1128

2003

Volltextzugriff (PDF)

All-trans-Retinoic Acid Inhibits Jun N-Terminal Kinase by Increasing Dual-Specificity Phosphatase Activity

Molecular and Cellular Biology, 1999-03, Vol.19 (3), p.1973-1980

1999

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AP-1 Transcription Factors as Regulators of Immune Responses in Cancer

Down‐regulation of the cyclin‐dependent kinase inhibitor p57 is mediated by Jab1/Csn5 in hepatocarcinogenesis

Sustained Activation of JNK/p38 MAPK Pathways in Response to Cisplatin Leads to Fas Ligand Induction and Cell Death in Ovarian Carcinoma Cells

Constitutive control of AKT1 gene expression by JUNB/CJUN in ALK+ anaplastic large-cell lymphoma: a novel crosstalk mechanism

Essential roles of Jab1 in cell survival, spontaneous DNA damage and DNA repair

Withdrawal of Survival Factors Results in Activation of the JNK Pathway in Neuronal Cells Leading to Fas Ligand Induction and Cell Death

YAP1 Promotes Tumor Invasion and Metastasis in Nasopharyngeal Carcinoma with Hepatitis B Virus Infection

The oncogenic JUNB/CD30 axis contributes to cell cycle deregulation in ALK+ anaplastic large cell lymphoma

Selective activation of the JNK signaling cascade and c-Jun transcriptional activity by the small GTPases Rac and Cdc42Hs

Mechanisms for Lysophosphatidic Acid-induced Cytokine Production in Ovarian Cancer Cells

N-(4-hydroxyphenyl)retinamide-induced apoptosis triggered by reactive oxygen species is mediated by activation of MAPKs in head and neck squamous carcinoma cells

Abstract P5-08-02: New target in the resistant mechanism to trastuzumab

Lasting N-Terminal Phosphorylation of c-Jun and Activation of c-Jun N-Terminal Kinases after Neuronal Injury

A new group of conserved coactivators that increase the specificity of AP-1 transcription factors

Induction of c‐fos expression through JNK‐mediated TCF/Elk‐1 phosphorylation

Identification of a Dual Specificity Kinase That Activates the Jun Kinases and p38-Mpk2

Jun activation domain-binding protein 1 expression in breast cancer inversely correlates with the cell cycle inhibitor p27(Kip1)

Macrophage migration inhibitory factor expression is increased in pituitary adenoma cell nuclei

Activation of cAMP and mitogen responsive genes relies on a common nuclear factor

JunB expression is a common feature of CD30+ lymphomas and lymphomatoid papulosis

2-acetylaminofluorene up-regulates rat mdr1b expression through generating reactive oxygen species that activate NF-kappa B pathway

Expression of phosphorylated p27(Kip1) protein and Jun activation domain-binding protein 1 in human pituitary tumors

Jun activation domain binding protein 1 expression is associated with low p27(Kip1)levels in node-negative breast cancer

Expression of p27(Kip1) and c-Jun activation binding protein 1 are inversely correlated in systemic anaplastic large cell lymphoma

All-trans-Retinoic Acid Inhibits Jun N-Terminal Kinase by Increasing Dual-Specificity Phosphatase Activity

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