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The secret life of ion channels: Kv1.3 potassium channels and proliferation

American Journal of Physiology: Cell Physiology, 2018-01, Vol.314 (1), p.C27-C42

2018

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Vascular smooth muscle ion channels in essential hypertension

Frontiers in physiology, 2022-09, Vol.13, p.1016175-1016175

2022

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Liver-specific ablation of insulin-degrading enzyme causes hepatic insulin resistance and glucose intolerance, without affecting insulin clearance in mice

Metabolism, clinical and experimental, 2018-11, Vol.88, p.1-11

2018

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Kv1.3 channels can modulate cell proliferation during phenotypic switch by an ion-flux independent mechanism

Arteriosclerosis, thrombosis, and vascular biology, 2012-05, Vol.32 (5), p.1299

2012

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miR-126 contributes to the epigenetic signature of diabetic vascular smooth muscle and enhances antirestenosis effects of Kv1.3 blockers

Molecular metabolism (Germany), 2021-11, Vol.53, p.101306-101306, Article 101306

2021

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Kv1.3 channels modulate human vascular smooth muscle cells proliferation independently of mTOR signaling pathway

Pflügers Archiv, 2015-08, Vol.467 (8), p.1711-1722

2015

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Phenotypic Modulation of Cultured Primary Human Aortic Vascular Smooth Muscle Cells by Uremic Serum

Frontiers in physiology, 2018-02, Vol.9, p.89-89

2018

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Association of Circulating microRNAs with Coronary Artery Disease and Usefulness for Reclassification of Healthy Individuals: The REGICOR Study

Journal of clinical medicine, 2020-05, Vol.9 (5), p.1402

2020

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Proliferative Role of Kv11 Channels in Murine Arteries

Frontiers in physiology, 2017-07, Vol.8, p.500-500

2017

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De novo expression of Kv6.3 contributes to changes in vascular smooth muscle cell excitability in a hypertensive mice strain

The Journal of physiology, 2009-02, Vol.587 (3), p.625-640

2009

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Tungstate-targeting of BKαβ1 channels tunes ERK phosphorylation and cell proliferation in human vascular smooth muscle

PloS one, 2015, Vol.10 (2), p.e0118148-e0118148

2015

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Contribution of Kv Channels to Phenotypic Remodeling of Human Uterine Artery Smooth Muscle Cells

Circulation research, 2005-12, Vol.97 (12), p.1280-1287

2005

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Characterization of endogenous Kv1.3 channel isoforms in T cells

Biophysical journal, 2022-02, Vol.121 (3), p.385a-386a

2022

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Regulation of glucose metabolism by nitrosative stress in neural cells

Molecular Aspects of Medicine, 2004-02, Vol.25 (1), p.61-73

2004

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Pancreatic β-cell-specific deletion of insulin-degrading enzyme leads to dysregulated insulin secretion and β-cell functional immaturity

American journal of physiology: endocrinology and metabolism, 2019-11, Vol.317 (5), p.E805-E819

2019

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Expression of glucose transporter GLUT3 by endotoxin in cultured rat astrocytes: the role of nitric oxide

Journal of neurochemistry, 2001-10, Vol.79 (1), p.17-24

2001

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Purinergic signalling in mesenteric vascular smooth muscle in a mice model of essential hypertension

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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Differences in TRPC3 and TRPC6 channels assembly in mesenteric vascular smooth muscle cells in essential hypertension

The Journal of physiology, 2017-03, Vol.595 (5), p.1497-1513

2017

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Molecular Determinants of Kv1.3 Potassium Channels-induced Proliferation

The Journal of biological chemistry, 2016-02, Vol.291 (7), p.3569-3580

2016

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Voltage-Dependent Conformational Changes of KV1.3 Potassium Channels are an Essential Element for KV1.3-induced cell proliferation

Biophysical journal, 2018-02, Vol.114 (3), p.378a-378a

2018

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Voltage‐dependent conformational changes of Kv1.3 channels activate cell proliferation

Journal of cellular physiology, 2021-06, Vol.236 (6), p.4330-4347

2021

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Characterization of endogenous Kv1.3 channel isoforms in T cells

Journal of cellular physiology, 2023-05, Vol.238 (5), p.976-991

2023

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Elastin-like recombinamer-based devices releasing Kv1.3 blockers for the prevention of intimal hyperplasia: An in vitro and in vivo study

Acta biomaterialia, 2020-10, Vol.115, p.264-274

2020

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Myocardin-Dependent Kv1.5 Channel Expression Prevents Phenotypic Modulation of Human Vessels in Organ Culture

Arteriosclerosis, thrombosis, and vascular biology, 2019-12, Vol.39 (12), p.e273

2019

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Down-regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca(2+) into hypertensive arterial smooth muscle

The Journal of physiology, 2013-12, Vol.591 (24), p.6175-6191

2013

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The secret life of ion channels: Kv1.3 potassium channels and proliferation

Vascular smooth muscle ion channels in essential hypertension

Liver-specific ablation of insulin-degrading enzyme causes hepatic insulin resistance and glucose intolerance, without affecting insulin clearance in mice

Kv1.3 channels can modulate cell proliferation during phenotypic switch by an ion-flux independent mechanism

miR-126 contributes to the epigenetic signature of diabetic vascular smooth muscle and enhances antirestenosis effects of Kv1.3 blockers

Kv1.3 channels modulate human vascular smooth muscle cells proliferation independently of mTOR signaling pathway

Phenotypic Modulation of Cultured Primary Human Aortic Vascular Smooth Muscle Cells by Uremic Serum

Association of Circulating microRNAs with Coronary Artery Disease and Usefulness for Reclassification of Healthy Individuals: The REGICOR Study

Proliferative Role of Kv11 Channels in Murine Arteries

De novo expression of Kv6.3 contributes to changes in vascular smooth muscle cell excitability in a hypertensive mice strain

Tungstate-targeting of BKαβ1 channels tunes ERK phosphorylation and cell proliferation in human vascular smooth muscle

Contribution of Kv Channels to Phenotypic Remodeling of Human Uterine Artery Smooth Muscle Cells

Characterization of endogenous Kv1.3 channel isoforms in T cells

Regulation of glucose metabolism by nitrosative stress in neural cells

Pancreatic β-cell-specific deletion of insulin-degrading enzyme leads to dysregulated insulin secretion and β-cell functional immaturity

Expression of glucose transporter GLUT3 by endotoxin in cultured rat astrocytes: the role of nitric oxide

Purinergic signalling in mesenteric vascular smooth muscle in a mice model of essential hypertension

Differences in TRPC3 and TRPC6 channels assembly in mesenteric vascular smooth muscle cells in essential hypertension

Molecular Determinants of Kv1.3 Potassium Channels-induced Proliferation

Voltage-Dependent Conformational Changes of KV1.3 Potassium Channels are an Essential Element for KV1.3-induced cell proliferation

Voltage‐dependent conformational changes of Kv1.3 channels activate cell proliferation

Characterization of endogenous Kv1.3 channel isoforms in T cells

Elastin-like recombinamer-based devices releasing Kv1.3 blockers for the prevention of intimal hyperplasia: An in vitro and in vivo study

Myocardin-Dependent Kv1.5 Channel Expression Prevents Phenotypic Modulation of Human Vessels in Organ Culture

Down-regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca(2+) into hypertensive arterial smooth muscle

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