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Coeliac Disease Pathogenesis: The Uncertainties of a Well-Known Immune Mediated Disorder

Frontiers in immunology, 2020-07, Vol.11, p.1374-1374

2020

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IL-1β blockade prevents cell death and mucosal damage of the small intestine in a model of sterile inflammation

Immunology letters, 2022-12, Vol.251-252, p.56-62

2022

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Duodenal bacterial proteolytic activity determines sensitivity to dietary antigen through protease-activated receptor-2

Nature communications, 2019-03, Vol.10 (1), p.1198-1198, Article 1198

2019

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Role of CXCR3/CXCL10 axis in immune cell recruitment into the small intestine in celiac disease

PloS one, 2014-02, Vol.9 (2), p.e89068-e89068

2014

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Programmed Cell Death in the Small Intestine: Implications for the Pathogenesis of Celiac Disease

International journal of molecular sciences, 2021-07, Vol.22 (14), p.7426

2021

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Coexistence of apoptosis, pyroptosis, and necroptosis pathways in celiac disease

Clinical and experimental immunology, 2023-12, Vol.214 (3), p.328-340

2023

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Broad MICA/B expression in the small bowel mucosa: a link between cellular stress and celiac disease

PloS one, 2013-09, Vol.8 (9), p.e73658-e73658

2013

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Biogeographic Variation and Functional Pathways of the Gut Microbiota in Celiac Disease

Gastroenterology (New York, N.Y. 1943), 2022-11, Vol.163 (5), p.1351-1363.e15

2022

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Intraluminal administration of poly I:C causes an enteropathy that is exacerbated by administration of oral dietary antigen

PloS one, 2014-06, Vol.9 (6), p.e99236

2014

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Intestinal Microbiota Modulates Gluten-Induced Immunopathology in Humanized Mice

The American journal of pathology, 2015-11, Vol.185 (11), p.2969-2982

2015

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IL-33 Alarmin and Its Active Proinflammatory Fragments Are Released in Small Intestine in Celiac Disease

Frontiers in immunology, 2020-10, Vol.11, p.581445-581445

2020

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p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine

Frontiers in immunology, 2019, Vol.10, p.31-31

2019

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Sterile inflammation drives multiple programmed cell death pathways in the gut

Journal of leukocyte biology, 2021-01, Vol.109 (1), p.211-221

2021

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Commentary: p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine

Frontiers in immunology, 2019-11, Vol.10, p.2792-2792

2019

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Immunomodulatory dendritic cells in intestinal lamina propria

European journal of immunology, 2005-06, Vol.35 (6), p.1831-1840

2005

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Gluten-Dependent Activation of CD4+ T Cells by MHC Class II–Expressing Epithelium

Gastroenterology (New York, N.Y. 1943), 2024-11, Vol.167 (6), p.1113-1128

2024

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Sensitization to gliadin induces moderate enteropathy and insulitis in nonobese diabetic-DQ8 mice

The Journal of immunology (1950), 2011-10, Vol.187 (8), p.4338-4346

2011

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Mechanisms of innate immune activation by gluten peptide p31-43 in mice

American journal of physiology: Gastrointestinal and liver physiology, 2016-07, Vol.311 (1), p.G40-G49

2016

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Immunomodulatory dendritic cells in intestinal lamina propria

European journal of immunology, 2005-06, Vol.35 (6), p.1831-1840

2005

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Statement of the Prolamin Working Group on the Determination of Gluten in Fermented Foods Containing Partially Hydrolyzed Gluten

Frontiers in nutrition (Lausanne), 2021-01, Vol.7, p.626712-626712

2021

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Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes

International journal of molecular sciences, 2022-02, Vol.23 (4), p.1973

2022

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Expression Pattern of Fatty Acid Binding Proteins in Celiac Disease Enteropathy

Mediators of Inflammation, 2015-01, Vol.2015 (2015), p.816-826-383

2015

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Increased Intraepithelial Vα24 Invariant NKT Cells in the Celiac Duodenum

Nutrients, 2015-10, Vol.7 (11), p.8960-8976

2015

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Role of germinal center and CD39highCD73+ B cells in the age-related tonsillar involution

Immunity & ageing, 2024-04, Vol.21 (1), p.1-24, Article 24

2024

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A galectin-specific signature in the gut delineates Crohn's disease and ulcerative colitis from other human inflammatory intestinal disorders

BioFactors (Oxford), 2016-01, Vol.42 (1), p.93-105

2016

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Coeliac Disease Pathogenesis: The Uncertainties of a Well-Known Immune Mediated Disorder

IL-1β blockade prevents cell death and mucosal damage of the small intestine in a model of sterile inflammation

Duodenal bacterial proteolytic activity determines sensitivity to dietary antigen through protease-activated receptor-2

Role of CXCR3/CXCL10 axis in immune cell recruitment into the small intestine in celiac disease

Programmed Cell Death in the Small Intestine: Implications for the Pathogenesis of Celiac Disease

Coexistence of apoptosis, pyroptosis, and necroptosis pathways in celiac disease

Broad MICA/B expression in the small bowel mucosa: a link between cellular stress and celiac disease

Biogeographic Variation and Functional Pathways of the Gut Microbiota in Celiac Disease

Intraluminal administration of poly I:C causes an enteropathy that is exacerbated by administration of oral dietary antigen

Intestinal Microbiota Modulates Gluten-Induced Immunopathology in Humanized Mice

IL-33 Alarmin and Its Active Proinflammatory Fragments Are Released in Small Intestine in Celiac Disease

p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine

Sterile inflammation drives multiple programmed cell death pathways in the gut

Commentary: p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine

Immunomodulatory dendritic cells in intestinal lamina propria

Gluten-Dependent Activation of CD4+ T Cells by MHC Class II–Expressing Epithelium

Sensitization to gliadin induces moderate enteropathy and insulitis in nonobese diabetic-DQ8 mice

Mechanisms of innate immune activation by gluten peptide p31-43 in mice

Immunomodulatory dendritic cells in intestinal lamina propria

Statement of the Prolamin Working Group on the Determination of Gluten in Fermented Foods Containing Partially Hydrolyzed Gluten

Inflammation Is Present, Persistent and More Sensitive to Proinflammatory Triggers in Celiac Disease Enterocytes

Expression Pattern of Fatty Acid Binding Proteins in Celiac Disease Enteropathy

Increased Intraepithelial Vα24 Invariant NKT Cells in the Celiac Duodenum

Role of germinal center and CD39highCD73+ B cells in the age-related tonsillar involution

A galectin-specific signature in the gut delineates Crohn's disease and ulcerative colitis from other human inflammatory intestinal disorders

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