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Gut Dysbiosis and Immune System in Atherosclerotic Cardiovascular Disease (ACVD)

Microorganisms (Basel), 2022-01, Vol.10 (1), p.108

2022

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Alterations of Mitochondrial Network by Cigarette Smoking and E-Cigarette Vaping

Cells (Basel, Switzerland), 2022-05, Vol.11 (10), p.1688

2022

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Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5‐trisphosphate receptors and Ca2+ release

The Journal of physiology, 2016-06, Vol.594 (11), p.2985-3004

2016

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The Antimalarial Chloroquine Reduces the Burden of Persistent Atrial Fibrillation

Frontiers in pharmacology, 2019-11, Vol.10, p.1392-1392

2019

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Major cardiac concerns in therapy and vaccinations for COVID-19

Metabolism open, 2021-09, Vol.11, p.100102-100102, Article 100102

2021

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All-Trans Retinoic Acid Increases DRP1 Levels and Promotes Mitochondrial Fission

Cells (Basel, Switzerland), 2021-05, Vol.10 (5), p.1202

2021

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IKACh is constitutively active via PKC epsilon in aging mediated atrial fibrillation

iScience, 2022-11, Vol.25 (11), p.105442-105442, Article 105442

2022

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Signaling Pathway for Endothelin-1- and Phenylephrine-Induced cAMP Response Element Binding Protein Activation in Rat Ventricular Myocytes: Role of Inositol 1,4,5-Trisphosphate Receptors and CaMKII

Cellular physiology and biochemistry, 2017-01, Vol.41 (1), p.399-412

2017

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Abstract 16786: ADP Ribosylation Factor 6 Modulates the Cell Surface Expression of the Acetylcholine Activated Inward Rectifier Potassium Current

Circulation (New York, N.Y.), 2018-11, Vol.138 (Suppl_1 Suppl 1), p.A16786-A16786

2018

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BS-454195-4 MITOCHONDRIAL REMODELING MEDIATES CARDIAC ELECTROPHYSIOLOGICAL INJURY IN INHALATION EXPOSURE TO FLAVORED ELECTRONIC NICOTINE DELIVERY SYSTEMS

Heart rhythm, 2023-05, Vol.20 (5), p.S8-S8

2023

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PO-01-124 THE CALCIUM HANDLING MACHINERY IS REMODELED IN FRIEDREICH’S ATAXIA

Heart rhythm, 2023-05, Vol.20 (5), p.S140-S140

2023

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The Arf6/PIP5K pathway activates IKACh in cigarette smoke mediated atrial fibrillation

Cellular signalling, 2022-12, Vol.100, p.110475-110475, Article 110475

2022

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Abstract 14241: Cigarette Smoke Causes Atrial Fibrillation via PiP2 Signaling

Circulation (New York, N.Y.), 2021-11, Vol.144 (Suppl_1)

2021

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The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich’s ataxia cardiomyopathy

The international journal of biochemistry & cell biology, 2022-02, Vol.143, p.106137-106137, Article 106137

2022

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Bioengineered peptibodies as blockers of ion channels

Proceedings of the National Academy of Sciences - PNAS, 2022-12, Vol.119 (50), p.e2212564119

2022

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Abstract 16680: Bioengineered Peptibodies as Novel Therapeutic Potassium Channel Blockers

Circulation (New York, N.Y.), 2018-11, Vol.138 (Suppl_1 Suppl 1), p.A16680-A16680

2018

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Structural basis for the antiarrhythmic blockade of a potassium channel with a small molecule

The FASEB journal, 2018-04, Vol.32 (4), p.1778-1793

2018

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Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5-trisphosphate receptors and Ca(2+) release

The Journal of physiology, 2016-06, Vol.594 (11), p.2985-3004

2016

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Elevated potassium outward currents in hyperoxia treated atrial cardiomyocytes

Journal of cellular physiology, 2018-05, Vol.233 (5), p.4317-4326

2018

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I KACh is constitutively active via PKC epsilon in aging mediated atrial fibrillation

iScience, 2022-11, Vol.25 (11), p.105442

2022

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Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5-trisphosphate receptors and Ca super(2+) release

The Journal of physiology, 2016-06, Vol.594 (11), p.2985-3004

2016

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Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5‐trisphosphate receptors and Ca 2+ release

The Journal of physiology, 2016-06, Vol.594 (11), p.2985-3004

2016

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Enhancement of contraction and L-type Ca2+ current by murrayafoline-A via protein kinase C in rat ventricular myocytes

European journal of pharmacology, 2016-08, Vol.784, p.33-41

2016

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Enhancement of contraction and L-type Ca(2+) current by murrayafoline-A via protein kinase C in rat ventricular myocytes

European journal of pharmacology, 2016-08, Vol.784, p.33-41

2016

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Alterations of contractions and L-type Ca2+ currents by murrayafoline-A in rat ventricular myocytes

European journal of pharmacology, 2014-10, Vol.740, p.81-87

2014

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Gut Dysbiosis and Immune System in Atherosclerotic Cardiovascular Disease (ACVD)

Alterations of Mitochondrial Network by Cigarette Smoking and E-Cigarette Vaping

Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5‐trisphosphate receptors and Ca2+ release

The Antimalarial Chloroquine Reduces the Burden of Persistent Atrial Fibrillation

Major cardiac concerns in therapy and vaccinations for COVID-19

All-Trans Retinoic Acid Increases DRP1 Levels and Promotes Mitochondrial Fission

IKACh is constitutively active via PKC epsilon in aging mediated atrial fibrillation

Signaling Pathway for Endothelin-1- and Phenylephrine-Induced cAMP Response Element Binding Protein Activation in Rat Ventricular Myocytes: Role of Inositol 1,4,5-Trisphosphate Receptors and CaMKII

Abstract 16786: ADP Ribosylation Factor 6 Modulates the Cell Surface Expression of the Acetylcholine Activated Inward Rectifier Potassium Current

BS-454195-4 MITOCHONDRIAL REMODELING MEDIATES CARDIAC ELECTROPHYSIOLOGICAL INJURY IN INHALATION EXPOSURE TO FLAVORED ELECTRONIC NICOTINE DELIVERY SYSTEMS

PO-01-124 THE CALCIUM HANDLING MACHINERY IS REMODELED IN FRIEDREICH’S ATAXIA

The Arf6/PIP5K pathway activates IKACh in cigarette smoke mediated atrial fibrillation

Abstract 14241: Cigarette Smoke Causes Atrial Fibrillation via PiP2 Signaling

The dynamin-related protein 1 is decreased and the mitochondrial network is altered in Friedreich’s ataxia cardiomyopathy

Bioengineered peptibodies as blockers of ion channels

Abstract 16680: Bioengineered Peptibodies as Novel Therapeutic Potassium Channel Blockers

Structural basis for the antiarrhythmic blockade of a potassium channel with a small molecule

Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5-trisphosphate receptors and Ca(2+) release

Elevated potassium outward currents in hyperoxia treated atrial cardiomyocytes

I KACh is constitutively active via PKC epsilon in aging mediated atrial fibrillation

Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5-trisphosphate receptors and Ca super(2+) release

Shear stress activates monovalent cation channel transient receptor potential melastatin subfamily 4 in rat atrial myocytes via type 2 inositol 1,4,5‐trisphosphate receptors and Ca 2+ release

Enhancement of contraction and L-type Ca2+ current by murrayafoline-A via protein kinase C in rat ventricular myocytes

Enhancement of contraction and L-type Ca(2+) current by murrayafoline-A via protein kinase C in rat ventricular myocytes

Alterations of contractions and L-type Ca2+ currents by murrayafoline-A in rat ventricular myocytes

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