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Dissecting Regulators of Aging and Age-Related Macular Degeneration in the Retinal Pigment Epithelium

Oxidative medicine and cellular longevity, 2022-11, Vol.2022, p.1-19

2022

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SS-31, a Mitochondria-Targeting Peptide, Ameliorates Kidney Disease

Oxidative medicine and cellular longevity, 2022, Vol.2022, p.1295509-13

2022

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Mahogunin ring finger-1 (MGRN1) Suppresses Chaperone-Associated Misfolded Protein Aggregation and Toxicity

Scientific reports, 2013-06, Vol.3 (1), p.1972-1972, Article 1972

2013

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Molecular, functional, and pathological aspects of TDP-43 fragmentation

iScience, 2021-05, Vol.24 (5), p.102459-102459, Article 102459

2021

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Amyloid fibril proteomics of AD brains reveals modifiers of aggregation and toxicity

Molecular neurodegeneration, 2023-09, Vol.18 (1), p.61-22, Article 61

2023

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TDP-35, a truncated fragment of TDP-43, induces dose-dependent toxicity and apoptosis in flies

Neural regeneration research, 2022-11, Vol.17 (11), p.2441-2442

2022

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Autophagy Coupling Interplay: Can Improve Cellular Repair and Aging?

Molecular neurobiology, 2014-06, Vol.49 (3), p.1270-1281

2014

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Misfolded Proteins Recognition Strategies of E3 Ubiquitin Ligases and Neurodegenerative Diseases

Molecular neurobiology, 2013-02, Vol.47 (1), p.302-312

2013

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E3 Ubiquitin Ligases in Protein Quality Control Mechanism

Molecular neurobiology, 2012-06, Vol.45 (3), p.571-585

2012

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Mahogunin ring finger 1 confers cytoprotection against mutant SOD1 aggresomes and is defective in an ALS mouse model

Neurobiology of disease, 2016-02, Vol.86, p.16-28

2016

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secHsp70 as a tool to approach amyloid-β42 and other extracellular amyloids

Fly (Austin, Tex.), 2017-07, Vol.11 (3), p.179-184

2017

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Identification of potential pathways and biomarkers linked to progression in ALS

Annals of clinical and translational neurology, 2023-02, Vol.10 (2), p.150-165

2023

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Mahogunin ring finger-1 (MGRN1) Suppresses Chaperone-Associated Misfolded Protein Aggregation and Toxicity

Scientific reports, 2013-06, Vol.3 (1), p.1972

2013

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Protein Quality Control System in Neurodegeneration: A Healing Company Hard to Beat but Failure is Fatal

Molecular neurobiology, 2013-08, Vol.48 (1), p.141-156

2013

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Ubiquitin ligase ITCH recruitment suppresses the aggregation and cellular toxicity of cytoplasmic misfolded proteins

Scientific reports, 2014-05, Vol.4 (1), p.5077, Article 5077

2014

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Misfolded Proteins Recognition Strategies of E3 Ubiquitin Ligases and Neurodegenerative Diseases

Molecular neurobiology, 2013-02, Vol.47 (1), p.302-312

2013

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Selective multifaceted E3 ubiquitin ligases barricade extreme defense: Potential therapeutic targets for neurodegeneration and ageing

Ageing research reviews, 2015-11, Vol.24 (Pt B), p.138-159

2015

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Ibuprofen Induces Mitochondrial-Mediated Apoptosis Through Proteasomal Dysfunction

Molecular neurobiology, 2016-12, Vol.53 (10), p.6968-6981

2016

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Mahogunin Ring Finger-1 (MGRN1), a Multifaceted Ubiquitin Ligase: Recent Unraveling of Neurobiological Mechanisms

Molecular neurobiology, 2016-09, Vol.53 (7), p.4484-4496

2016

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Mahogunin ring finger 1 suppresses misfolded polyglutamine aggregation and cytotoxicity

Biochimica et biophysica acta, 2014-09, Vol.1842 (9), p.1472-1484

2014

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Gp78 involvement in cellular proliferation: Can act as a promising modulator for cell cycle regulatory proteins?

Journal of cellular physiology, 2018-10, Vol.233 (10), p.6352-6368

2018

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Proteasomal Dysfunction Induced By Diclofenac Engenders Apoptosis Through Mitochondrial Pathway

Journal of cellular biochemistry, 2017-05, Vol.118 (5), p.1014-1027

2017

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E6-AP association promotes SOD1 aggresomes degradation and suppresses toxicity

Neurobiology of aging, 2013-04, Vol.34 (4), p.1310.e11-1310.e23

2013

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Infection and chronic disease activate a systemic brain-muscle signaling axis

Science immunology, 2024-07, Vol.9 (97), p.eadm7908

2024

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Nuclear import receptors are recruited by FG-nucleoporins to rescue hallmarks of TDP-43 proteinopathy

Molecular neurodegeneration, 2022-12, Vol.17 (1), p.80-27, Article 80

2022

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Dissecting Regulators of Aging and Age-Related Macular Degeneration in the Retinal Pigment Epithelium

SS-31, a Mitochondria-Targeting Peptide, Ameliorates Kidney Disease

Mahogunin ring finger-1 (MGRN1) Suppresses Chaperone-Associated Misfolded Protein Aggregation and Toxicity

Molecular, functional, and pathological aspects of TDP-43 fragmentation

Amyloid fibril proteomics of AD brains reveals modifiers of aggregation and toxicity

TDP-35, a truncated fragment of TDP-43, induces dose-dependent toxicity and apoptosis in flies

Autophagy Coupling Interplay: Can Improve Cellular Repair and Aging?

Misfolded Proteins Recognition Strategies of E3 Ubiquitin Ligases and Neurodegenerative Diseases

E3 Ubiquitin Ligases in Protein Quality Control Mechanism

Mahogunin ring finger 1 confers cytoprotection against mutant SOD1 aggresomes and is defective in an ALS mouse model

secHsp70 as a tool to approach amyloid-β42 and other extracellular amyloids

Identification of potential pathways and biomarkers linked to progression in ALS

Mahogunin ring finger-1 (MGRN1) Suppresses Chaperone-Associated Misfolded Protein Aggregation and Toxicity

Protein Quality Control System in Neurodegeneration: A Healing Company Hard to Beat but Failure is Fatal

Ubiquitin ligase ITCH recruitment suppresses the aggregation and cellular toxicity of cytoplasmic misfolded proteins

Misfolded Proteins Recognition Strategies of E3 Ubiquitin Ligases and Neurodegenerative Diseases

Selective multifaceted E3 ubiquitin ligases barricade extreme defense: Potential therapeutic targets for neurodegeneration and ageing

Ibuprofen Induces Mitochondrial-Mediated Apoptosis Through Proteasomal Dysfunction

Mahogunin Ring Finger-1 (MGRN1), a Multifaceted Ubiquitin Ligase: Recent Unraveling of Neurobiological Mechanisms

Mahogunin ring finger 1 suppresses misfolded polyglutamine aggregation and cytotoxicity

Gp78 involvement in cellular proliferation: Can act as a promising modulator for cell cycle regulatory proteins?

Proteasomal Dysfunction Induced By Diclofenac Engenders Apoptosis Through Mitochondrial Pathway

E6-AP association promotes SOD1 aggresomes degradation and suppresses toxicity

Infection and chronic disease activate a systemic brain-muscle signaling axis

Nuclear import receptors are recruited by FG-nucleoporins to rescue hallmarks of TDP-43 proteinopathy

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