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Mining the Na v 1.7 interactome: Opportunities for chronic pain therapeutics

Biochemical pharmacology, 2019-05, Vol.163, p.9

2019

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Long-lasting antinociceptive effects of green light in acute and chronic pain in rats

Pain (Amsterdam), 2017-02, Vol.158 (2), p.347-360

2017

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CRMP2 and voltage-gated ion channels: potential roles in neuropathic pain

Neuronal signaling, 2018-03, Vol.2 (1)

2018

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Gene-agnostic approaches to treating inherited retinal degenerations

Frontiers in cell and developmental biology, 2023-04, Vol.11, p.1177838-1177838

2023

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Corneal endothelial transplantation from bench to bedside: A review of animal models and their translational value for therapeutic development

Experimental eye research, 2022-11, Vol.224, p.109241-109241, Article 109241

2022

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AAV Gene Augmentation of Truncated Complement Factor H Differentially Rescues Ocular Complement Dysregulation in a Mouse Model

Investigative ophthalmology & visual science, 2023-07, Vol.64 (10), p.25

2023

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Robust cone-mediated signaling persists late into rod photoreceptor degeneration

eLife, 2022-08, Vol.11

2022

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Conotoxin contulakin-G engages a neurotensin receptor 2 /R-type calcium channel (Cav2.3) pathway to mediate spinal antinociception

Pain (Amsterdam), 2022-09, Vol.163 (9), p.1751-1762

2022

Volltextzugriff (PDF)

CRISPR/Cas9 editing of Nf1 gene identifies CRMP2 as a therapeutic target in neurofibromatosis type 1-related pain that is reversed by (S)-Lacosamide

Pain (Amsterdam), 2017-12, Vol.158 (12), p.2301-2319

2017

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Dissecting the role of the CRMP2-neurofibromin complex on pain behaviors

Pain (Amsterdam), 2017-11, Vol.158 (11), p.2203-2221

2017

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Studies on CRMP2 SUMOylation-deficient transgenic mice identify sex-specific Nav1.7 regulation in the pathogenesis of chronic neuropathic pain

Pain (Amsterdam), 2020-11, Vol.161 (11), p.2629-2651

2020

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Selective targeting of NaV1.7 via inhibition of the CRMP2-Ubc9 interaction reduces pain in rodents

Science translational medicine, 2021-11, Vol.13 (619), p.eabh1314

2021

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Betulinic acid, derived from the desert lavender Hyptis emoryi, attenuates paclitaxel-, HIV-, and nerve injury-associated peripheral sensory neuropathy via block of N- and T-type calcium channels

Pain (Amsterdam), 2019-01, Vol.160 (1), p.117-135

2019

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Mining the Nav1.7 interactome: Opportunities for chronic pain therapeutics

Biochemical pharmacology, 2019-05, Vol.163, p.9-20

2019

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(S)-lacosamide inhibition of CRMP2 phosphorylation reduces postoperative and neuropathic pain behaviors through distinct classes of sensory neurons identified by constellation pharmacology

Pain (Amsterdam), 2016-07, Vol.157 (7), p.1448-1463

2016

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Targeting the CaVα-CaVβ interaction yields an antagonist of the N-type CaV2.2 channel with broad antinociceptive efficacy

Pain (Amsterdam), 2019-07, Vol.160 (7), p.1644-1661

2019

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Tetramethylpyrazine Reduces Epileptogenesis Progression in Electrical Kindling Models by Modulating Hippocampal Excitatory Neurotransmission

ACS chemical neuroscience, 2019-12, Vol.10 (12), p.4854-4863

2019

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Development and Characterization of An Injury-free Model of Functional Pain in Rats by Exposure to Red Light

The journal of pain, 2019-11, Vol.20 (11), p.1293-1306

2019

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Sustained relief of ongoing experimental neuropathic pain by a CRMP2 peptide aptamer with low abuse potential

Pain (Amsterdam), 2016-09, Vol.157 (9), p.2124-2140

2016

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Erratum to ‛Development and Characterization of An Injury-free Model of Functional Pain in Rats by Exposure to Red Light’: The Journal of Pain 20 (2019) 1293-1306

The journal of pain, 2019-12, Vol.20 (12), p.1509-1509

2019

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Chemotherapy can induce weight normalization of morbidly obese mice despite undiminished ingestion of high fat diet

Oncotarget, 2017-01, Vol.8 (3), p.5426-5438

2017

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(S)-Lacosamide Binding to Collapsin Response Mediator Protein 2 (CRMP2) Regulates CaV2.2 Activity by Subverting Its Phosphorylation by Cdk5

Molecular neurobiology, 2016-04, Vol.53 (3), p.1959-1976

2016

Volltextzugriff (PDF)

Microglia at sites of atrophy restrict the progression of retinal degeneration via galectin-3 and Trem2

The Journal of experimental medicine, 2024-03, Vol.221 (3)

2024

Volltextzugriff (PDF)

Erratum to ‛Development and Characterization of An Injury-free Model of Functional Pain in Rats by Exposure to Red Light

The journal of pain, 2019-12, Vol.20 (12), p.1509

2019

Volltextzugriff (PDF)

Choriocapillaris

Reference Module in Neuroscience and Biobehavioral Psychology, 2017

2017

Volltextzugriff (PDF)

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Mining the Na v 1.7 interactome: Opportunities for chronic pain therapeutics

Long-lasting antinociceptive effects of green light in acute and chronic pain in rats

CRMP2 and voltage-gated ion channels: potential roles in neuropathic pain

Gene-agnostic approaches to treating inherited retinal degenerations

Corneal endothelial transplantation from bench to bedside: A review of animal models and their translational value for therapeutic development

AAV Gene Augmentation of Truncated Complement Factor H Differentially Rescues Ocular Complement Dysregulation in a Mouse Model

Robust cone-mediated signaling persists late into rod photoreceptor degeneration

Conotoxin contulakin-G engages a neurotensin receptor 2 /R-type calcium channel (Cav2.3) pathway to mediate spinal antinociception

CRISPR/Cas9 editing of Nf1 gene identifies CRMP2 as a therapeutic target in neurofibromatosis type 1-related pain that is reversed by (S)-Lacosamide

Dissecting the role of the CRMP2-neurofibromin complex on pain behaviors

Studies on CRMP2 SUMOylation-deficient transgenic mice identify sex-specific Nav1.7 regulation in the pathogenesis of chronic neuropathic pain

Selective targeting of NaV1.7 via inhibition of the CRMP2-Ubc9 interaction reduces pain in rodents

Betulinic acid, derived from the desert lavender Hyptis emoryi, attenuates paclitaxel-, HIV-, and nerve injury-associated peripheral sensory neuropathy via block of N- and T-type calcium channels

Mining the Nav1.7 interactome: Opportunities for chronic pain therapeutics

(S)-lacosamide inhibition of CRMP2 phosphorylation reduces postoperative and neuropathic pain behaviors through distinct classes of sensory neurons identified by constellation pharmacology

Targeting the CaVα-CaVβ interaction yields an antagonist of the N-type CaV2.2 channel with broad antinociceptive efficacy

Tetramethylpyrazine Reduces Epileptogenesis Progression in Electrical Kindling Models by Modulating Hippocampal Excitatory Neurotransmission

Development and Characterization of An Injury-free Model of Functional Pain in Rats by Exposure to Red Light

Sustained relief of ongoing experimental neuropathic pain by a CRMP2 peptide aptamer with low abuse potential

Erratum to ‛Development and Characterization of An Injury-free Model of Functional Pain in Rats by Exposure to Red Light’: The Journal of Pain 20 (2019) 1293-1306

Chemotherapy can induce weight normalization of morbidly obese mice despite undiminished ingestion of high fat diet

(S)-Lacosamide Binding to Collapsin Response Mediator Protein 2 (CRMP2) Regulates CaV2.2 Activity by Subverting Its Phosphorylation by Cdk5

Microglia at sites of atrophy restrict the progression of retinal degeneration via galectin-3 and Trem2

Erratum to ‛Development and Characterization of An Injury-free Model of Functional Pain in Rats by Exposure to Red Light

Choriocapillaris

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