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Diverse mechanisms of AKT pathway activation in human malignancy

Current cancer drug targets, 2013-03, Vol.13 (3), p.234

2013

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Germline BAP1 mutations predispose to malignant mesothelioma

Nature genetics, 2011-10, Vol.43 (10), p.1022-1025

2011

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Bap1 Is a Bona Fide Tumor Suppressor: Genetic Evidence from Mouse Models Carrying Heterozygous Germline Bap1 Mutations

Cancer research (Chicago, Ill.), 2016-05, Vol.76 (9), p.2836-2844

2016

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Germline Mutation of Bap1 Accelerates Development of Asbestos-Induced Malignant Mesothelioma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4388-4397

2014

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Deregulated Akt3 activity promotes development of malignant melanoma

Cancer research (Chicago, Ill.), 2004-10, Vol.64 (19), p.7002-7010

2004

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RAD001 Inhibits Human Ovarian Cancer Cell Proliferation, Enhances Cisplatin-Induced Apoptosis, and Prolongs Survival in an Ovarian Cancer Model

Clinical cancer research, 2007-07, Vol.13 (14), p.4261-4270

2007

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Upregulation of DLX5 Promotes Ovarian Cancer Cell Proliferation by Enhancing IRS-2-AKT Signaling

Cancer research (Chicago, Ill.), 2010-11, Vol.70 (22), p.9197-9206

2010

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Onconase responsive genes in human mesothelioma cells: implications for an RNA damaging therapeutic agent

BMC cancer, 2010-02, Vol.10 (1), p.34-34, Article 34

2010

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Regulation of B-Raf Kinase Activity by Tuberin and Rheb Is Mammalian Target of Rapamycin (mTOR)-independent

The Journal of biological chemistry, 2004-07, Vol.279 (29), p.29930-29937

2004

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BAP1, a tumor suppressor gene driving malignant mesothelioma

Translational lung cancer research, 2017-06, Vol.6 (3), p.270-278

2017

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Germline BAP1 Mutational Landscape of Asbestos-Exposed Malignant Mesothelioma Patients with Family History of Cancer

Cancer research (Chicago, Ill.), 2016-01, Vol.76 (2), p.206-215

2016

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Akt3 and Mutant V600EB-Raf Cooperate to Promote Early Melanoma Development

Cancer research (Chicago, Ill.), 2008-05, Vol.68 (9), p.3429-3439

2008

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Novel LRRK2 mutations and other rare, non-BAP1-related candidate tumor predisposition gene variants in high-risk cancer families with mesothelioma and other tumors

Human molecular genetics, 2021-08, Vol.30 (18), p.1750-1761

2021

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Somatic Epigenetic Silencing of RIPK3 Inactivates Necroptosis and Contributes to Chemoresistance in Malignant Mesothelioma

Clinical cancer research, 2021-02, Vol.27 (4), p.1200-1213

2021

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Inactivation of Bap1 Cooperates with Losses of Nf2 and Cdkn2a to Drive the Development of Pleural Malignant Mesothelioma in Conditional Mouse Models

Cancer research (Chicago, Ill.), 2019-08, Vol.79 (16), p.4113-4123

2019

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Germline BAP1 mutation in a family with high incidence of multiple primary cancers and a potential gene–environment interaction

Cancer letters, 2015-12, Vol.369 (2), p.261-265

2015

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Abstract 1232: Familial mesothelioma: Beyond BAP1

Cancer research (Chicago, Ill.), 2018-07, Vol.78 (13_Supplement), p.1232-1232

2018

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Inactivation of p21-Activated Kinase 2 (Pak2) Inhibits the Development of Nf2-Deficient Tumors by Restricting Downstream Hedgehog and Wnt Signaling

Molecular cancer research, 2022-05, Vol.20 (5), p.699-711

2022

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Familial and Somatic BAP1 Mutations Inactivate ASXL1/2-Mediated Allosteric Regulation of BAP1 Deubiquitinase by Targeting Multiple Independent Domains

Cancer research (Chicago, Ill.), 2018-03, Vol.78 (5), p.1200-1213

2018

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Inflammation-Related IL1β/IL1R Signaling Promotes the Development of Asbestos-Induced Malignant Mesothelioma

Cancer prevention research (Philadelphia, Pa.), 2016-05, Vol.9 (5), p.406-414

2016

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PD46-01 BAP1 DE-UBIQUITINASE IS ALLOSTERICALLY REGULATED BY ASXL1/2: SOMATIC BAP1 MUTATION IN RCC AND OTHER TUMORS INACTIVE THIS REGULATION BY TARGETING MULTIPLE INDEPENDENT DOMAINS

The Journal of urology, 2018-04, Vol.199 (4), p.e891-e891

2018

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Kinetic Characterization of ASXL1/2-Mediated Allosteric Regulation of the BAP1 Deubiquitinase

Molecular cancer research, 2021-07, Vol.19 (7), p.1099-1112

2021

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Further evidence for germline BAP1 mutations predisposing to melanoma and malignant mesothelioma

Cancer genetics, 2013-05, Vol.206 (5), p.206-210

2013

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Low Exposures to Amphibole or Serpentine Asbestos in Germline Bap1-mutant Mice Induce Mesothelioma Characterized by an Immunosuppressive Tumor Microenvironment

Cancer research communications, 2024-04, Vol.4 (4), p.1004-1015

2024

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The promyelocytic leukemia zinc-finger gene, PLZF, is frequently downregulated in malignant mesothelioma cells and contributes to cell survival

Oncogene, 2010-03, Vol.29 (11), p.1633-1640

2010

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Diverse mechanisms of AKT pathway activation in human malignancy

Germline BAP1 mutations predispose to malignant mesothelioma

Bap1 Is a Bona Fide Tumor Suppressor: Genetic Evidence from Mouse Models Carrying Heterozygous Germline Bap1 Mutations

Germline Mutation of Bap1 Accelerates Development of Asbestos-Induced Malignant Mesothelioma

Deregulated Akt3 activity promotes development of malignant melanoma

RAD001 Inhibits Human Ovarian Cancer Cell Proliferation, Enhances Cisplatin-Induced Apoptosis, and Prolongs Survival in an Ovarian Cancer Model

Upregulation of DLX5 Promotes Ovarian Cancer Cell Proliferation by Enhancing IRS-2-AKT Signaling

Onconase responsive genes in human mesothelioma cells: implications for an RNA damaging therapeutic agent

Regulation of B-Raf Kinase Activity by Tuberin and Rheb Is Mammalian Target of Rapamycin (mTOR)-independent

BAP1, a tumor suppressor gene driving malignant mesothelioma

Germline BAP1 Mutational Landscape of Asbestos-Exposed Malignant Mesothelioma Patients with Family History of Cancer

Akt3 and Mutant V600EB-Raf Cooperate to Promote Early Melanoma Development

Novel LRRK2 mutations and other rare, non-BAP1-related candidate tumor predisposition gene variants in high-risk cancer families with mesothelioma and other tumors

Somatic Epigenetic Silencing of RIPK3 Inactivates Necroptosis and Contributes to Chemoresistance in Malignant Mesothelioma

Inactivation of Bap1 Cooperates with Losses of Nf2 and Cdkn2a to Drive the Development of Pleural Malignant Mesothelioma in Conditional Mouse Models

Germline BAP1 mutation in a family with high incidence of multiple primary cancers and a potential gene–environment interaction

Abstract 1232: Familial mesothelioma: Beyond BAP1

Inactivation of p21-Activated Kinase 2 (Pak2) Inhibits the Development of Nf2-Deficient Tumors by Restricting Downstream Hedgehog and Wnt Signaling

Familial and Somatic BAP1 Mutations Inactivate ASXL1/2-Mediated Allosteric Regulation of BAP1 Deubiquitinase by Targeting Multiple Independent Domains

Inflammation-Related IL1β/IL1R Signaling Promotes the Development of Asbestos-Induced Malignant Mesothelioma

PD46-01 BAP1 DE-UBIQUITINASE IS ALLOSTERICALLY REGULATED BY ASXL1/2: SOMATIC BAP1 MUTATION IN RCC AND OTHER TUMORS INACTIVE THIS REGULATION BY TARGETING MULTIPLE INDEPENDENT DOMAINS

Kinetic Characterization of ASXL1/2-Mediated Allosteric Regulation of the BAP1 Deubiquitinase

Further evidence for germline BAP1 mutations predisposing to melanoma and malignant mesothelioma

Low Exposures to Amphibole or Serpentine Asbestos in Germline Bap1-mutant Mice Induce Mesothelioma Characterized by an Immunosuppressive Tumor Microenvironment

The promyelocytic leukemia zinc-finger gene, PLZF, is frequently downregulated in malignant mesothelioma cells and contributes to cell survival

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