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μ‐Opioid receptors in primary sensory neurons are essential for opioid analgesic effect on acute and inflammatory pain and opioid‐induced hyperalgesia

The Journal of physiology, 2019-03, Vol.597 (6), p.1661-1675

2019

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Increased α2δ‐1–NMDA receptor coupling potentiates glutamatergic input to spinal dorsal horn neurons in chemotherapy‐induced neuropathic pain

Journal of neurochemistry, 2019-01, Vol.148 (2), p.252-274

2019

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α2δ‐1 protein drives opioid‐induced conditioned reward and synaptic NMDA receptor hyperactivity in the nucleus accumbens

Journal of neurochemistry, 2023-01, Vol.164 (2), p.143-157

2023

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Presynaptic NMDA receptors control nociceptive transmission at the spinal cord level in neuropathic pain

Cellular and molecular life sciences : CMLS, 2019-05, Vol.76 (10), p.1889-1899

2019

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Endogenous transient receptor potential ankyrin 1 and vanilloid 1 activity potentiates glutamatergic input to spinal lamina I neurons in inflammatory pain

Journal of neurochemistry, 2019-05, Vol.149 (3), p.381-398

2019

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The α2δ-1-NMDA Receptor Complex Is Critically Involved in Neuropathic Pain Development and Gabapentin Therapeutic Actions

Cell reports (Cambridge), 2018-02, Vol.22 (9), p.2307-2321

2018

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Regulating nociceptive transmission by VGluT2‐expressing spinal dorsal horn neurons

Journal of neurochemistry, 2018-11, Vol.147 (4), p.526-540

2018

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Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension

The Journal of physiology, 2019-08, Vol.597 (16), p.4325-4340

2019

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α2δ‐1 couples to NMDA receptors in the hypothalamus to sustain sympathetic vasomotor activity in hypertension

The Journal of physiology, 2018-09, Vol.596 (17), p.4269-4283

2018

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Mitogen‐activated protein kinase signaling mediates opioid‐induced presynaptic NMDA receptor activation and analgesic tolerance

Journal of neurochemistry, 2019-01, Vol.148 (2), p.275-290

2019

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Calcineurin regulates synaptic Ca2+‐permeable AMPA receptors in hypothalamic presympathetic neurons via α2δ‐1‐mediated GluA1/GluA2 assembly

The Journal of physiology, 2024-05, Vol.602 (10), p.2179-2197

2024

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Ghrelin receptors mediate ghrelin‐induced excitation of agouti‐related protein/neuropeptide Y but not pro‐opiomelanocortin neurons

Journal of neurochemistry, 2017-08, Vol.142 (4), p.512-520

2017

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Cannabinoids suppress inflammatory and neuropathic pain by targeting α3 glycine receptors

The Journal of experimental medicine, 2012-06, Vol.209 (6), p.1121-1134

2012

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Pannexin-1 Up-regulation in the Dorsal Root Ganglion Contributes to Neuropathic Pain Development

The Journal of biological chemistry, 2015-06, Vol.290 (23), p.14647-14655

2015

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Bortezomib induces neuropathic pain through protein kinase C-mediated activation of presynaptic NMDA receptors in the spinal cord

Neuropharmacology, 2017-09, Vol.123, p.477-487

2017

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N-Methyl-d-aspartate Receptor- and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic Pain

The Journal of biological chemistry, 2012-09, Vol.287 (40), p.33853-33864

2012

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α2δ-1 Is Essential for Sympathetic Output and NMDA Receptor Activity Potentiated by Angiotensin II in the Hypothalamus

The Journal of neuroscience, 2018-07, Vol.38 (28), p.6388-6398

2018

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α2δ‐1 protein promotes synaptic expression of Ca2+ permeable–AMPA receptors by inhibiting GluA1/GluA2 heteromeric assembly in the hypothalamus in hypertension

Journal of neurochemistry, 2022-04, Vol.161 (1), p.40-52

2022

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Nerve injury increases brain-derived neurotrophic factor levels to suppress BK channel activity in primary sensory neurons

Journal of neurochemistry, 2012-06, Vol.121 (6), p.944-953

2012

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Calcineurin inhibitor induces pain hypersensitivity by potentiating pre‐ and postsynaptic NMDA receptor activity in spinal cords

The Journal of physiology, 2014-01, Vol.592 (1), p.215-227

2014

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Chloride Homeostasis Critically Regulates Synaptic NMDA Receptor Activity in Neuropathic Pain

Cell reports (Cambridge), 2016-05, Vol.15 (7), p.1376-1383

2016

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Opioid-Induced Long-Term Potentiation in the Spinal Cord Is a Presynaptic Event

The Journal of neuroscience, 2010-03, Vol.30 (12), p.4460-4466

2010

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Reduction in voltage-gated K⁺ channel activity in primary sensory neurons in painful diabetic neuropathy: role of brain-derived neurotrophic factor

Journal of neurochemistry, 2010-09, Vol.114 (5), p.1460-1475

2010

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Presynaptic N-Methyl-d-aspartate (NMDA) Receptor Activity Is Increased Through Protein Kinase C in Paclitaxel-induced Neuropathic Pain

The Journal of biological chemistry, 2016-09, Vol.291 (37), p.19364-19373

2016

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Increased Spinal Cord Na+-K+-2Cl− Cotransporter-1 (NKCC1) Activity Contributes to Impairment of Synaptic Inhibition in Paclitaxel-induced Neuropathic Pain

The Journal of biological chemistry, 2014-11, Vol.289 (45), p.31111-31120

2014

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μ‐Opioid receptors in primary sensory neurons are essential for opioid analgesic effect on acute and inflammatory pain and opioid‐induced hyperalgesia

Increased α2δ‐1–NMDA receptor coupling potentiates glutamatergic input to spinal dorsal horn neurons in chemotherapy‐induced neuropathic pain

α2δ‐1 protein drives opioid‐induced conditioned reward and synaptic NMDA receptor hyperactivity in the nucleus accumbens

Presynaptic NMDA receptors control nociceptive transmission at the spinal cord level in neuropathic pain

Endogenous transient receptor potential ankyrin 1 and vanilloid 1 activity potentiates glutamatergic input to spinal lamina I neurons in inflammatory pain

The α2δ-1-NMDA Receptor Complex Is Critically Involved in Neuropathic Pain Development and Gabapentin Therapeutic Actions

Regulating nociceptive transmission by VGluT2‐expressing spinal dorsal horn neurons

Endogenous AT1 receptor–protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension

α2δ‐1 couples to NMDA receptors in the hypothalamus to sustain sympathetic vasomotor activity in hypertension

Mitogen‐activated protein kinase signaling mediates opioid‐induced presynaptic NMDA receptor activation and analgesic tolerance

Calcineurin regulates synaptic Ca2+‐permeable AMPA receptors in hypothalamic presympathetic neurons via α2δ‐1‐mediated GluA1/GluA2 assembly

Ghrelin receptors mediate ghrelin‐induced excitation of agouti‐related protein/neuropeptide Y but not pro‐opiomelanocortin neurons

Cannabinoids suppress inflammatory and neuropathic pain by targeting α3 glycine receptors

Pannexin-1 Up-regulation in the Dorsal Root Ganglion Contributes to Neuropathic Pain Development

Bortezomib induces neuropathic pain through protein kinase C-mediated activation of presynaptic NMDA receptors in the spinal cord

N-Methyl-d-aspartate Receptor- and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic Pain

α2δ-1 Is Essential for Sympathetic Output and NMDA Receptor Activity Potentiated by Angiotensin II in the Hypothalamus

α2δ‐1 protein promotes synaptic expression of Ca2+ permeable–AMPA receptors by inhibiting GluA1/GluA2 heteromeric assembly in the hypothalamus in hypertension

Nerve injury increases brain-derived neurotrophic factor levels to suppress BK channel activity in primary sensory neurons

Calcineurin inhibitor induces pain hypersensitivity by potentiating pre‐ and postsynaptic NMDA receptor activity in spinal cords

Chloride Homeostasis Critically Regulates Synaptic NMDA Receptor Activity in Neuropathic Pain

Opioid-Induced Long-Term Potentiation in the Spinal Cord Is a Presynaptic Event

Reduction in voltage-gated K⁺ channel activity in primary sensory neurons in painful diabetic neuropathy: role of brain-derived neurotrophic factor

Presynaptic N-Methyl-d-aspartate (NMDA) Receptor Activity Is Increased Through Protein Kinase C in Paclitaxel-induced Neuropathic Pain

Increased Spinal Cord Na+-K+-2Cl− Cotransporter-1 (NKCC1) Activity Contributes to Impairment of Synaptic Inhibition in Paclitaxel-induced Neuropathic Pain

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