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Ablation of the Ferroptosis Inhibitor Glutathione Peroxidase 4 in Neurons Results in Rapid Motor Neuron Degeneration and Paralysis

The Journal of biological chemistry, 2015-11, Vol.290 (47), p.28097-28106

2015

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Ablation of ferroptosis regulator glutathione peroxidase 4 in forebrain neurons promotes cognitive impairment and neurodegeneration

Redox biology, 2017-08, Vol.12, p.8-17

2017

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Overexpression of ferroptosis defense enzyme Gpx4 retards motor neuron disease of SOD1G93A mice

Scientific reports, 2021-06, Vol.11 (1), p.12890-12890, Article 12890

2021

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Gpx4 ablation in adult mice results in a lethal phenotype accompanied by neuronal loss in brain

Free radical biology & medicine, 2012-05, Vol.52 (9), p.1820-1827

2012

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Lipid peroxidation up-regulates BACE1 expression in vivo: a possible early event of amyloidogenesis in Alzheimer's disease

Journal of neurochemistry, 2008-10, Vol.107 (1), p.197-207

2008

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Reduction of mitochondrial H2O2 by overexpressing peroxiredoxin 3 improves glucose tolerance in mice

Aging cell, 2008-12, Vol.7 (6), p.866-878

2008

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NLRP3 inflammasome activation by mitochondrial reactive oxygen species plays a key role in long-term cognitive impairment induced by paraquat exposure

Neurobiology of aging, 2015-09, Vol.36 (9), p.2533-2543

2015

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Enhanced defense against ferroptosis ameliorates cognitive impairment and reduces neurodegeneration in 5xFAD mice

Free radical biology & medicine, 2022-02, Vol.180, p.1-12

2022

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Tea Catechins Protect against Lead-Induced ROS Formation, Mitochondrial Dysfunction, and Calcium Dysregulation in PC12 Cells

Chemical research in toxicology, 2003-09, Vol.16 (9), p.1155-1161

2003

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Enhanced defense against mitochondrial hydrogen peroxide attenuates age-associated cognition decline

Neurobiology of aging, 2014-11, Vol.35 (11), p.2552-2561

2014

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The Gpx4NIKO Mouse Is a Versatile Model for Testing Interventions Targeting Ferroptotic Cell Death of Spinal Motor Neurons

Neurotoxicity research, 2022-04, Vol.40 (2), p.373-383

2022

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Ablation of Gpx4 in Forebrain Neurons Leads to Ferroptotic Cell Death and Cognitive Impairment

Free radical biology & medicine, 2016-11, Vol.100, p.S161

2016

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382 - Ablation of Gpx4 in Forebrain Neurons Leads to Ferroptotic Cell Death and Cognitive Impairment

Free radical biology & medicine, 2016-11, Vol.100, p.S161-S161

2016

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Ablation of Gpx4 in Neurons Results in Rapid Motor Neuron Degeneration and Paralysis

Free radical biology & medicine, 2015-10, Vol.87, p.S34

2015

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Bio-coloration and Antibacterial Function of Wool grafted with pomegranate peel polyphenols catalyzed by laccase

Materials today communications, 2024-08, Vol.40, p.109910, Article 109910

2024

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Effect of Tea Catechins on the Change of Glutathione Levels Caused by Pb++ in PC12 Cells

Chemical research in toxicology, 2004-07, Vol.17 (7), p.922-928

2004

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Cognitive impairment and increased Aβ levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H2O2

Neurobiology of aging, 2012-02, Vol.33 (2), p.432.e15-432.e26

2012

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Overexpression of Glutaredoxin 2 Improves Mitochondrial Function and Prevents Memory Deficits in a Mouse Model of Alzheimer's Disease

Free radical biology & medicine, 2013-11, Vol.65, p.S43-S44

2013

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55 - Ablation of Gpx4 in Neurons Results in Rapid Motor Neuron Degeneration and Paralysis

Free radical biology & medicine, 2015-10, Vol.87, p.S34-S34

2015

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PSS74 - Overexpression of Glutaredoxin 2 Improves Mitochondrial Function and Prevents Memory Deficits in a Mouse Model of Alzheimer's Disease

Free radical biology & medicine, 2013-11, Vol.65, p.S43-S44

2013

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Tea Catechins Protect against Lead-Induced Cytotoxicity, Lipid Peroxidation, and Membrane Fluidity in HepG2 Cells

Toxicological sciences, 2002-09, Vol.69 (1), p.149-156

2002

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Reactive Oxygen Species From Mitochondria Play a Key Role in Exacerbating Cognitive Impairment and Amyloidogenesis Induced by Pesticide Exposure

Free radical biology & medicine, 2011-11, Vol.51, p.S69-S69

2011

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Cognitive impairment and increased AI super(2) levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H sub(2O) sub(2)

Neurobiology of aging, 2012-02, Vol.33 (2), p.432.e15-432.e26

2012

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Cognitive impairment and increased Aβ levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H2 O2

Neurobiology of aging, 2012, Vol.33 (2), p.432.e15-432.e26

2012

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Lipid peroxidation upregulates BACE1 expression in vivo: a possible early event of amyloidogenesis in Alzheimer’s disease

Journal of neurochemistry, 2008-08, Vol.107 (1), p.197-207

2008

Volltextzugriff (PDF)

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Ablation of the Ferroptosis Inhibitor Glutathione Peroxidase 4 in Neurons Results in Rapid Motor Neuron Degeneration and Paralysis

Ablation of ferroptosis regulator glutathione peroxidase 4 in forebrain neurons promotes cognitive impairment and neurodegeneration

Overexpression of ferroptosis defense enzyme Gpx4 retards motor neuron disease of SOD1G93A mice

Gpx4 ablation in adult mice results in a lethal phenotype accompanied by neuronal loss in brain

Lipid peroxidation up-regulates BACE1 expression in vivo: a possible early event of amyloidogenesis in Alzheimer's disease

Reduction of mitochondrial H2O2 by overexpressing peroxiredoxin 3 improves glucose tolerance in mice

NLRP3 inflammasome activation by mitochondrial reactive oxygen species plays a key role in long-term cognitive impairment induced by paraquat exposure

Enhanced defense against ferroptosis ameliorates cognitive impairment and reduces neurodegeneration in 5xFAD mice

Tea Catechins Protect against Lead-Induced ROS Formation, Mitochondrial Dysfunction, and Calcium Dysregulation in PC12 Cells

Enhanced defense against mitochondrial hydrogen peroxide attenuates age-associated cognition decline

The Gpx4NIKO Mouse Is a Versatile Model for Testing Interventions Targeting Ferroptotic Cell Death of Spinal Motor Neurons

Ablation of Gpx4 in Forebrain Neurons Leads to Ferroptotic Cell Death and Cognitive Impairment

382 - Ablation of Gpx4 in Forebrain Neurons Leads to Ferroptotic Cell Death and Cognitive Impairment

Ablation of Gpx4 in Neurons Results in Rapid Motor Neuron Degeneration and Paralysis

Bio-coloration and Antibacterial Function of Wool grafted with pomegranate peel polyphenols catalyzed by laccase

Effect of Tea Catechins on the Change of Glutathione Levels Caused by Pb++ in PC12 Cells

Cognitive impairment and increased Aβ levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H2O2

Overexpression of Glutaredoxin 2 Improves Mitochondrial Function and Prevents Memory Deficits in a Mouse Model of Alzheimer's Disease

55 - Ablation of Gpx4 in Neurons Results in Rapid Motor Neuron Degeneration and Paralysis

PSS74 - Overexpression of Glutaredoxin 2 Improves Mitochondrial Function and Prevents Memory Deficits in a Mouse Model of Alzheimer's Disease

Tea Catechins Protect against Lead-Induced Cytotoxicity, Lipid Peroxidation, and Membrane Fluidity in HepG2 Cells

Reactive Oxygen Species From Mitochondria Play a Key Role in Exacerbating Cognitive Impairment and Amyloidogenesis Induced by Pesticide Exposure

Cognitive impairment and increased AI super(2) levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H sub(2O) sub(2)

Cognitive impairment and increased Aβ levels induced by paraquat exposure are attenuated by enhanced removal of mitochondrial H2 O2

Lipid peroxidation upregulates BACE1 expression in vivo: a possible early event of amyloidogenesis in Alzheimer’s disease

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