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CFTR dysfunction leads to defective bacterial eradication on cystic fibrosis airways

Frontiers in physiology, 2024-04, Vol.15, p.1385661-1385661

2024

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Altering intracellular pH reveals the kinetic basis of intraburst gating in the CFTR Cl− channel

The Journal of physiology, 2017-02, Vol.595 (4), p.1059-1076

2017

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Human cystic fibrosis airway epithelia have reduced Cl−conductance but not increased Na+conductance

Proceedings of the National Academy of Sciences - PNAS, 2011-06, Vol.108 (25), p.10260-10265

2011

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A defective flexible loop contributes to the processing and gating defects of the predominant cystic fibrosis‐causing mutation

The FASEB journal, 2019-04, Vol.33 (4), p.5126-5142

2019

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Protein kinase A phosphorylation potentiates cystic fibrosis transmembrane conductance regulator gating by relieving autoinhibition on the stimulatory C terminus of the regulatory domain

The Journal of biological chemistry, 2020-04, Vol.295 (14), p.4577-4590

Chen, Jeng-Haur

2020

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CFTR-deficient pigs display peripheral nervous system defects at birth

Proceedings of the National Academy of Sciences - PNAS, 2013-02, Vol.110 (8), p.3083-3088

2013

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Loss of Anion Transport without Increased Sodium Absorption Characterizes Newborn Porcine Cystic Fibrosis Airway Epithelia

Cell, 2010-12, Vol.143 (6), p.911-923

2010

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CFTR founder mutation causes protein trafficking defects in Chinese patients with cystic fibrosis

Molecular genetics & genomic medicine, 2017-01, Vol.5 (1), p.40-49

2017

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The ΔF508 mutation causes CFTR misprocessing and cystic fibrosis-like disease in pigs

Science translational medicine, 2011-03, Vol.3 (74), p.74ra24

2011

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A cocktail drug therapy for patients with cystic fibrosis?

Journal of cystic fibrosis, 2014-09, Vol.13 (5), p.489-490

Chen, Jeng-Haur

2014

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Intestinal CFTR expression alleviates meconium ileus in cystic fibrosis pigs

The Journal of clinical investigation, 2013-06, Vol.123 (6), p.2685-2693

2013

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Acute inhibition of the cystic fibrosis transmembrane conductance regulator (CFTR) Cl- channel by thyroid hormones involves multiple mechanisms

American Journal of Physiology: Cell Physiology, 2013-10, Vol.305 (8), p.C817-C828

2013

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Direct Sensing of Intracellular pH by the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Cl− Channel

The Journal of biological chemistry, 2009-12, Vol.284 (51), p.35495-35506

2009

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Inhibition of Protein Kinase CK2 Closes the CFTR Cl- Channel, but has no Effect on the Cystic Fibrosis Mutant ΔF508-CFTR

Cellular physiology and biochemistry, 2009-01, Vol.24 (5-6), p.347-360

2009

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Protein Kinase CK2, Cystic Fibrosis Transmembrane Conductance Regulator, and the ΔF508 Mutation: F508 DELETION DISRUPTS A KINASE-BINDING SITE

The Journal of biological chemistry, 2007-04, Vol.282 (14), p.10804-10813

2007

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The Physiology and Pharmacology of the CFTR Cl− Channel

Advances in Molecular and Cell Biology, 2007, Vol.38, p.109-143

2007

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The cystic fibrosis transmembrane conductance regulator Cl⁻ channel: a versatile engine for transepithelial ion transport

Sheng li hsüeh pao, 2007-08, Vol.59 (4), p.416-430

2007

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Altering intracellular pH reveals the kinetic basis of intraburst gating in the CFTR Cl super(-) channel

The Journal of physiology, 2017-02, Vol.595 (4), p.1059-1076

2017

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Direct Sensing of Intracellular pH by the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Clâ Channel

The Journal of biological chemistry, 2009-12, Vol.284 (51), p.35495

2009

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Human cystic fibrosis airway epithelia have reduced Clâ» conductance but not increased Naâº conductance

Proceedings of the National Academy of Sciences - PNAS, 2011, Vol.108 (25), p.10260-10265

2011

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Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the ΔF508 mutation. F508 deletion disrupts a kinase-binding site. VOLUME 282 (2007) PAGES 10804-10813

The Journal of biological chemistry, 2008-09, Vol.283 (36), p.25103-25103

2008

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Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the deltaF508 mutation: F508 deletion disrupts a kinase-binding site

The Journal of biological chemistry, 2008-09, Vol.283 (36), p.25103-25103

2008

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Protein Kinase CK2, Cystic Fibrosis Transmembrane Conductance Regulator, and the ÎF508 Mutation

The Journal of biological chemistry, 2007-04, Vol.282 (14), p.10804

2007

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Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the deltaF508 mutation: F508 deletion disrupts a kinase-binding site

The Journal of biological chemistry, 2007-04, Vol.282 (14), p.10804-10813

2007

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Novel role of the Ca(2+)-ATPase in NMDA-induced intracellular acidification

The American journal of physiology, 1999-10, Vol.277 (4), p.C717

1999

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CFTR dysfunction leads to defective bacterial eradication on cystic fibrosis airways

Altering intracellular pH reveals the kinetic basis of intraburst gating in the CFTR Cl− channel

Human cystic fibrosis airway epithelia have reduced Cl−conductance but not increased Na+conductance

A defective flexible loop contributes to the processing and gating defects of the predominant cystic fibrosis‐causing mutation

Protein kinase A phosphorylation potentiates cystic fibrosis transmembrane conductance regulator gating by relieving autoinhibition on the stimulatory C terminus of the regulatory domain

CFTR-deficient pigs display peripheral nervous system defects at birth

Loss of Anion Transport without Increased Sodium Absorption Characterizes Newborn Porcine Cystic Fibrosis Airway Epithelia

CFTR founder mutation causes protein trafficking defects in Chinese patients with cystic fibrosis

The ΔF508 mutation causes CFTR misprocessing and cystic fibrosis-like disease in pigs

A cocktail drug therapy for patients with cystic fibrosis?

Intestinal CFTR expression alleviates meconium ileus in cystic fibrosis pigs

Acute inhibition of the cystic fibrosis transmembrane conductance regulator (CFTR) Cl- channel by thyroid hormones involves multiple mechanisms

Direct Sensing of Intracellular pH by the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Cl− Channel

Inhibition of Protein Kinase CK2 Closes the CFTR Cl- Channel, but has no Effect on the Cystic Fibrosis Mutant ΔF508-CFTR

Protein Kinase CK2, Cystic Fibrosis Transmembrane Conductance Regulator, and the ΔF508 Mutation: F508 DELETION DISRUPTS A KINASE-BINDING SITE

The Physiology and Pharmacology of the CFTR Cl− Channel

The cystic fibrosis transmembrane conductance regulator Cl⁻ channel: a versatile engine for transepithelial ion transport

Altering intracellular pH reveals the kinetic basis of intraburst gating in the CFTR Cl super(-) channel

Direct Sensing of Intracellular pH by the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Clâ Channel

Human cystic fibrosis airway epithelia have reduced Clâ» conductance but not increased Naâº conductance

Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the ΔF508 mutation. F508 deletion disrupts a kinase-binding site. VOLUME 282 (2007) PAGES 10804-10813

Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the deltaF508 mutation: F508 deletion disrupts a kinase-binding site

Protein Kinase CK2, Cystic Fibrosis Transmembrane Conductance Regulator, and the ÎF508 Mutation

Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the deltaF508 mutation: F508 deletion disrupts a kinase-binding site

Novel role of the Ca(2+)-ATPase in NMDA-induced intracellular acidification

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