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The transcription factor XBP1s restores hippocampal synaptic plasticity and memory by control of the Kalirin-7 pathway in Alzheimer model

Molecular psychiatry, 2017-11, Vol.22 (11), p.1562-1575

2017

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The Disintegrin ADAM9 Indirectly Contributes to the Physiological Processing of Cellular Prion by Modulating ADAM10 Activity

The Journal of biological chemistry, 2005-12, Vol.280 (49), p.40624-40631

2005

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Influence of Genetic Background on Apathy-Like Behavior in Triple Transgenic AD Mice

Current Alzheimer research, 2016-08, Vol.13 (8), p.942

2016

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Glioma tumor grade correlates with parkin depletion in mutant p53-linked tumors and results from loss of function of p53 transcriptional activity

Oncogene, 2014-04, Vol.33 (14), p.1764-1775

2014

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Presenilin-dependent gamma-secretase-mediated control of p53-associated cell death in Alzheimer's disease

The Journal of neuroscience, 2006-06, Vol.26 (23), p.6377-6385

2006

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Sox2 functionally interacts with βAPP, the βAPP intracellular domain and ADAM10 at a transcriptional level in human cells

Neuroscience, 2016-01, Vol.312, p.153-164

2016

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Loss of function of DJ-1 triggered by Parkinson's disease-associated mutation is due to proteolytic resistance to caspase-6

Cell death and differentiation, 2010-01, Vol.17 (1), p.158-169

2010

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Cellular prion and its catabolites in the brain: production and function

Current molecular medicine, 2012-03, Vol.12 (3), p.304

2012

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The caspase 6 derived N-terminal fragment of DJ-1 promotes apoptosis via increased ROS production

Cell death and differentiation, 2012-11, Vol.19 (11), p.1769-1778

2012

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Interplay between Parkin and p53 Governs a Physiological Homeostasis That Is Disrupted in Parkinson's Disease and Cerebral Cancer

Neuro-degenerative diseases, 2014-01, Vol.13 (2-3), p.118-121

2014

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The multiple paradoxes of presenilins

Journal of neurochemistry, 2001-03, Vol.76 (6), p.1621-1627

Checler, F.

2001

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Galantamine-based hybrid molecules with acetylcholinesterase, butyrylcholinesterase and γ-secretase inhibition activities

Current Alzheimer research, 2012-06, Vol.9 (5), p.600

2012

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Wild-type but Not Parkinson's Disease-related Ala-53 → Thr Mutant α-Synuclein Protects Neuronal Cells from Apoptotic Stimuli

The Journal of biological chemistry, 2000-08, Vol.275 (31), p.24065-24069

2000

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Prominent Cerebral Amyloid Angiopathy in Transgenic Mice Overexpressing the London Mutant of Human APP in Neurons

The American journal of pathology, 2000-10, Vol.157 (4), p.1283-1298

2000

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p53, a Pivotal Effector of a Functional Cross-Talk Linking Presenilins and Pen-2

Neuro-degenerative diseases, 2012-01, Vol.10 (1-4), p.52-55

2012

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The caspase 6 derived N-terminal fragment of DJ-1 promotes apoptosis via increased ROS production

Cell death and differentiation, 2012-11, Vol.19 (11), p.1769-1778

2012

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Unusual Phenotypic Alteration of β Amyloid Precursor Protein (β APP) Maturation by a New Val-715 → Met β APP-770 Mutation Responsible for Probable Early-Onset Alzheimer's Disease

Proceedings of the National Academy of Sciences - PNAS, 1999-03, Vol.96 (7), p.4119-4124

1999

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Parkin: Much More than a Simple Ubiquitin Ligase

Neuro-degenerative diseases, 2012-01, Vol.10 (1-4), p.49-51

2012

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Caspase-3-derived C-terminal Product of Synphilin-1 Displays Antiapoptotic Function via Modulation of the p53-dependent Cell Death Pathway

The Journal of biological chemistry, 2006-04, Vol.281 (17), p.11515-11522

2006

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Neprilysin activity and expression are controlled by nicastrin

Journal of neurochemistry, 2006-05, Vol.97 (4), p.1052-1056

2006

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α-Secretase-Derived Cleavage of Cellular Prion Yields Biologically Active Catabolites with Distinct Functions

Neuro-degenerative diseases, 2012-01, Vol.10 (1-4), p.294-297

2012

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Primary Cultured Neurons Devoid of Cellular Prion Display Lower Responsiveness to Staurosporine through the Control of p53 at Both Transcriptional and Post-transcriptional Levels

The Journal of biological chemistry, 2004-01, Vol.279 (1), p.612-618

2004

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Processing of the β‐Amyloid Precursor Protein and Its Regulation in Alzheimer's Disease

Journal of neurochemistry, 1995-10, Vol.65 (4), p.1431-1444

Checler, Frédéric

1995

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Cerebrospinal Aβ11-x and 17-x levels as indicators of mild cognitive impairment and patients' stratification in Alzheimer's disease

Translational psychiatry, 2013-07, Vol.3 (7), p.e281-e281

2013

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α-Synuclein and the Parkinson's disease-related mutant Ala53Thr-α-synuclein do not undergo proteasomal degradation in HEK293 and neuronal cells

Neuroscience letters, 2000-05, Vol.285 (2), p.79-82

2000

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The transcription factor XBP1s restores hippocampal synaptic plasticity and memory by control of the Kalirin-7 pathway in Alzheimer model

The Disintegrin ADAM9 Indirectly Contributes to the Physiological Processing of Cellular Prion by Modulating ADAM10 Activity

Influence of Genetic Background on Apathy-Like Behavior in Triple Transgenic AD Mice

Glioma tumor grade correlates with parkin depletion in mutant p53-linked tumors and results from loss of function of p53 transcriptional activity

Presenilin-dependent gamma-secretase-mediated control of p53-associated cell death in Alzheimer's disease

Sox2 functionally interacts with βAPP, the βAPP intracellular domain and ADAM10 at a transcriptional level in human cells

Loss of function of DJ-1 triggered by Parkinson's disease-associated mutation is due to proteolytic resistance to caspase-6

Cellular prion and its catabolites in the brain: production and function

The caspase 6 derived N-terminal fragment of DJ-1 promotes apoptosis via increased ROS production

Interplay between Parkin and p53 Governs a Physiological Homeostasis That Is Disrupted in Parkinson's Disease and Cerebral Cancer

The multiple paradoxes of presenilins

Galantamine-based hybrid molecules with acetylcholinesterase, butyrylcholinesterase and γ-secretase inhibition activities

Wild-type but Not Parkinson's Disease-related Ala-53 → Thr Mutant α-Synuclein Protects Neuronal Cells from Apoptotic Stimuli

Prominent Cerebral Amyloid Angiopathy in Transgenic Mice Overexpressing the London Mutant of Human APP in Neurons

p53, a Pivotal Effector of a Functional Cross-Talk Linking Presenilins and Pen-2

The caspase 6 derived N-terminal fragment of DJ-1 promotes apoptosis via increased ROS production

Unusual Phenotypic Alteration of β Amyloid Precursor Protein (β APP) Maturation by a New Val-715 → Met β APP-770 Mutation Responsible for Probable Early-Onset Alzheimer's Disease

Parkin: Much More than a Simple Ubiquitin Ligase

Caspase-3-derived C-terminal Product of Synphilin-1 Displays Antiapoptotic Function via Modulation of the p53-dependent Cell Death Pathway

Neprilysin activity and expression are controlled by nicastrin

α-Secretase-Derived Cleavage of Cellular Prion Yields Biologically Active Catabolites with Distinct Functions

Primary Cultured Neurons Devoid of Cellular Prion Display Lower Responsiveness to Staurosporine through the Control of p53 at Both Transcriptional and Post-transcriptional Levels

Processing of the β‐Amyloid Precursor Protein and Its Regulation in Alzheimer's Disease

Cerebrospinal Aβ11-x and 17-x levels as indicators of mild cognitive impairment and patients' stratification in Alzheimer's disease

α-Synuclein and the Parkinson's disease-related mutant Ala53Thr-α-synuclein do not undergo proteasomal degradation in HEK293 and neuronal cells

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