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Cellular mechanisms of injury after major trauma

British journal of surgery, 2009-10, Vol.96 (10), p.1097-1098

2009

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Mechanism of hepatocellular dysfunction during hyperdynamic sepsis

American journal of physiology. Regulatory, integrative and comparative physiology, 1996-05, Vol.270 (5), p.927-R938

1996

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Effect of gender and sex hormones on immune responses following shock

Shock (Augusta, Ga.), 2000-08, Vol.14 (2), p.81-90

2000

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Organ dysfunction following hemorrhage and sepsis: mechanisms and therapeutic approaches (Review)

International journal of molecular medicine, 1999-12, Vol.4 (6), p.575

1999

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Differential Induction of Apoptosis in Lymphoid Tissues During Sepsis: Variation in Onset, Frequency, and the Nature of the Mediators

Blood, 1996-05, Vol.87 (10), p.4261-4275

1996

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Estradiol administration after trauma-hemorrhage improves cardiovascular and hepatocellular functions in male animals

Annals of surgery, 2000-11, Vol.232 (5), p.673-679

2000

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Females in proestrus state maintain splenic immune functions and tolerate sepsis better than males

Critical care medicine, 1997-01, Vol.25 (1), p.106-110

1997

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Alterations in tissue oxygen consumption and extraction after trauma and hemorrhagic shock

Critical care medicine, 2000-08, Vol.28 (8), p.2837-2842

2000

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Castration prevents suppression of MHC class II (Ia) expression on macrophages after trauma-hemorrhage

Journal of applied physiology (1985), 2006-08, Vol.101 (2), p.448-453

2006

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Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxation

American journal of physiology. Heart and circulatory physiology, 1994-06, Vol.266 (6), p.H2535-H2541

1994

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Kupffer Cells Are Responsible for Producing Inflammatory Cytokines and Hepatocellular Dysfunction during Early Sepsis

The Journal of surgical research, 1999-05, Vol.83 (2), p.151-157

1999

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Differential alterations in plasma IL-6 and TNF levels after trauma and hemorrhage

American journal of physiology. Regulatory, integrative and comparative physiology, 1991-01, Vol.260 (1), p.167-R171

1991

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Hypoxemia in the absence of blood loss or significant hypotension causes inflammatory cytokine release

American journal of physiology. Regulatory, integrative and comparative physiology, 1995-07, Vol.269 (1), p.160-R166

1995

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Severe depression of host immune functions following closed-bone fracture, soft-tissue trauma, and hemorrhagic shock

Critical care medicine, 1998-08, Vol.26 (8), p.1372-1378

1998

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Insight into the mechanism by which estradiol improves organ functions after trauma-hemorrhage

Surgery, 2000-08, Vol.128 (2), p.246-252

2000

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Prolactin administration following hemorrhagic shock improves macrophage cytokine release capacity and decreases mortality from subsequent sepsis

The Journal of immunology (1950), 1996-12, Vol.157 (12), p.5748-5754

1996

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Effects of hydroxyethyl starch after trauma-hemorrhagic shock: Restoration of macrophage integrity and prevention of increased circulating interleukin-6 levels

Critical care medicine, 1995-05, Vol.23 (5), p.806-814

1995

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Endothelial cell dysfunction occurs very early following trauma-hemorrhage and persists despite fluid resuscitation

American journal of physiology. Heart and circulatory physiology, 1993-09, Vol.265 (3), p.H973-H979

1993

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Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsis

American journal of physiology. Regulatory, integrative and comparative physiology, 1995-11, Vol.269 (5), p.988-R994

1995

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Increased Gut Permeability after Hemorrhage Is Associated with Upregulation of Local and Systemic IL-6

The Journal of surgical research, 1998-09, Vol.79 (1), p.39-46

1998

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The complex pattern of cytokines in sepsis : association between prostaglandins, cachectin, and interleukins

Annals of surgery, 1991-08, Vol.214 (2), p.141-148

1991

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Glycine attenuates hepatocellular depression during early sepsis and reduces sepsis-induced mortality

Critical care medicine, 2001-06, Vol.29 (6), p.1201-1206

2001

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Effects of trauma, duration of hypotension, and resuscitation regimen on cellular immunity after hemorrhagic shock

Critical care medicine, 1994-07, Vol.22 (7), p.1076-1083

1994

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Trauma-induced suppression of antigen presentation and expression of major histocompatibility class II antigen complex in leukocytes

Shock (Augusta, Ga.), 1996-02, Vol.5 (2), p.79-90

1996

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Increased mucosal B-lymphocyte apoptosis during polymicrobial sepsis is a fas ligand but not an endotoxin-mediated process

Blood, 1998-02, Vol.91 (4), p.1362-1372

1998

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Cellular mechanisms of injury after major trauma

Mechanism of hepatocellular dysfunction during hyperdynamic sepsis

Effect of gender and sex hormones on immune responses following shock

Organ dysfunction following hemorrhage and sepsis: mechanisms and therapeutic approaches (Review)

Differential Induction of Apoptosis in Lymphoid Tissues During Sepsis: Variation in Onset, Frequency, and the Nature of the Mediators

Estradiol administration after trauma-hemorrhage improves cardiovascular and hepatocellular functions in male animals

Females in proestrus state maintain splenic immune functions and tolerate sepsis better than males

Alterations in tissue oxygen consumption and extraction after trauma and hemorrhagic shock

Castration prevents suppression of MHC class II (Ia) expression on macrophages after trauma-hemorrhage

Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxation

Kupffer Cells Are Responsible for Producing Inflammatory Cytokines and Hepatocellular Dysfunction during Early Sepsis

Differential alterations in plasma IL-6 and TNF levels after trauma and hemorrhage

Hypoxemia in the absence of blood loss or significant hypotension causes inflammatory cytokine release

Severe depression of host immune functions following closed-bone fracture, soft-tissue trauma, and hemorrhagic shock

Insight into the mechanism by which estradiol improves organ functions after trauma-hemorrhage

Prolactin administration following hemorrhagic shock improves macrophage cytokine release capacity and decreases mortality from subsequent sepsis

Effects of hydroxyethyl starch after trauma-hemorrhagic shock: Restoration of macrophage integrity and prevention of increased circulating interleukin-6 levels

Endothelial cell dysfunction occurs very early following trauma-hemorrhage and persists despite fluid resuscitation

Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsis

Increased Gut Permeability after Hemorrhage Is Associated with Upregulation of Local and Systemic IL-6

The complex pattern of cytokines in sepsis : association between prostaglandins, cachectin, and interleukins

Glycine attenuates hepatocellular depression during early sepsis and reduces sepsis-induced mortality

Effects of trauma, duration of hypotension, and resuscitation regimen on cellular immunity after hemorrhagic shock

Trauma-induced suppression of antigen presentation and expression of major histocompatibility class II antigen complex in leukocytes

Increased mucosal B-lymphocyte apoptosis during polymicrobial sepsis is a fas ligand but not an endotoxin-mediated process

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