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Circadian Regulation of Cardiac Physiology: Rhythms That Keep the Heart Beating

Annual review of physiology, 2020-02, Vol.82 (1), p.79-101

2020

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First characterization of glucose flux through the hexosamine biosynthesis pathway (HBP) in ex vivo mouse heart

The Journal of biological chemistry, 2020-02, Vol.295 (7), p.2018-2033

2020

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Role of O-linked N-acetylglucosamine (O-GlcNAc) modification of proteins in diabetic cardiovascular complications

Current opinion in pharmacology, 2021-04, Vol.57, p.1-12

2021

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Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal

Biochemical journal, 2009-10, Vol.424 (1), p.99-107

2009

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Protein glycosylation in cardiovascular health and disease

Nature reviews cardiology, 2024-08, Vol.21 (8), p.525-544

2024

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O-GlcNAcylation and cardiovascular disease

Biochemical Society transactions, 2017-04, Vol.45 (2), p.545-553

2017

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Forskolin reverses the O-GlcNAcylation dependent decrease in GABAAR current amplitude at hippocampal synapses possibly at a neurosteroid site on GABAARs

Scientific reports, 2024-07, Vol.14 (1), p.17461-18, Article 17461

2024

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Protein O-GlcNAcylation and Cardiovascular (Patho)physiology

The Journal of biological chemistry, 2014-12, Vol.289 (50), p.34449-34456

2014

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Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2

American Journal of Physiology: Cell Physiology, 2008-06, Vol.294 (6), p.C1509-C1520

2008

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Regulation of cardiac O-GlcNAcylation: More than just nutrient availability

Biochimica et biophysica acta. Molecular basis of disease, 2020-05, Vol.1866 (5), p.165712-165712, Article 165712

2020

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STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology

American journal of physiology. Heart and circulatory physiology, 2013-08, Vol.305 (4), p.H446-H458

2013

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O-GlcNAcylation and neurodegeneration

Brain research bulletin, 2017-07, Vol.133, p.80-87

2017

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Fine-tuning the cardiac O-GlcNAcylation regulatory enzymes governs the functional and structural phenotype of the diabetic heart

Cardiovascular research, 2022-01, Vol.118 (1), p.212-225

2022

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Assessing bioenergetic function in response to oxidative stress by metabolic profiling

Free radical biology & medicine, 2011-11, Vol.51 (9), p.1621-1635

2011

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Activation of AKT by O-Linked N-Acetylglucosamine Induces Vascular Calcification in Diabetes Mellitus

Circulation research, 2014-03, Vol.114 (7), p.1094-1102

2014

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O-GlcNAc modification of NFκB p65 inhibits TNF-α-induced inflammatory mediator expression in rat aortic smooth muscle cells

PloS one, 2011, Vol.6 (8), p.e24021-e24021

2011

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Reprint of: Role of O-linked N-acetylglucosamine (O-GlcNAc) modification of proteins in diabetic cardiovascular complications

Current opinion in pharmacology, 2020-10, Vol.54, p.209-220

2020

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Role of O -Linked N -Acetylglucosamine Protein Modification in Cellular (Patho)Physiology

Physiological reviews, 2021-04, Vol.101 (2), p.427-493

2021

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Cardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heart

Life sciences (1973), 2013-03, Vol.92 (11), p.648-656

2013

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Regulation of autophagy by protein post-translational modification

Laboratory investigation, 2015-01, Vol.95 (1), p.14-25

2015

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Post-translational protein modification by O-linked N-acetyl-glucosamine: Its role in mediating the adverse effects of diabetes on the heart

Life sciences (1973), 2013-03, Vol.92 (11), p.621-627

2013

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Best practices in assessing cardiac protein O -GlcNAcylation by immunoblot

American journal of physiology. Heart and circulatory physiology, 2023-10, Vol.325 (4), p.H601-H616

2023

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Modification of STIM1 by O-linked N-Acetylglucosamine (O-GlcNAc) Attenuates Store-operated Calcium Entry in Neonatal Cardiomyocytes

The Journal of biological chemistry, 2012-11, Vol.287 (46), p.39094-39106

2012

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O-GlcNAc regulation of autophagy and α-synuclein homeostasis; implications for Parkinson's disease

Molecular brain, 2017-07, Vol.10 (1), p.32-14, Article 32

2017

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Cardiomyocyte-Specific BMAL1 Plays Critical Roles in Metabolism, Signaling, and Maintenance of Contractile Function of the Heart

Journal of biological rhythms, 2014-08, Vol.29 (4), p.257-276

2014

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Circadian Regulation of Cardiac Physiology: Rhythms That Keep the Heart Beating

First characterization of glucose flux through the hexosamine biosynthesis pathway (HBP) in ex vivo mouse heart

Role of O-linked N-acetylglucosamine (O-GlcNAc) modification of proteins in diabetic cardiovascular complications

Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal

Protein glycosylation in cardiovascular health and disease

O-GlcNAcylation and cardiovascular disease

Forskolin reverses the O-GlcNAcylation dependent decrease in GABAAR current amplitude at hippocampal synapses possibly at a neurosteroid site on GABAARs

Protein O-GlcNAcylation and Cardiovascular (Patho)physiology

Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2

Regulation of cardiac O-GlcNAcylation: More than just nutrient availability

STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology

O-GlcNAcylation and neurodegeneration

Fine-tuning the cardiac O-GlcNAcylation regulatory enzymes governs the functional and structural phenotype of the diabetic heart

Assessing bioenergetic function in response to oxidative stress by metabolic profiling

Activation of AKT by O-Linked N-Acetylglucosamine Induces Vascular Calcification in Diabetes Mellitus

O-GlcNAc modification of NFκB p65 inhibits TNF-α-induced inflammatory mediator expression in rat aortic smooth muscle cells

Reprint of: Role of O-linked N-acetylglucosamine (O-GlcNAc) modification of proteins in diabetic cardiovascular complications

Role of O -Linked N -Acetylglucosamine Protein Modification in Cellular (Patho)Physiology

Cardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heart

Regulation of autophagy by protein post-translational modification

Post-translational protein modification by O-linked N-acetyl-glucosamine: Its role in mediating the adverse effects of diabetes on the heart

Best practices in assessing cardiac protein O -GlcNAcylation by immunoblot

Modification of STIM1 by O-linked N-Acetylglucosamine (O-GlcNAc) Attenuates Store-operated Calcium Entry in Neonatal Cardiomyocytes

O-GlcNAc regulation of autophagy and α-synuclein homeostasis; implications for Parkinson's disease

Cardiomyocyte-Specific BMAL1 Plays Critical Roles in Metabolism, Signaling, and Maintenance of Contractile Function of the Heart

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