UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Resistance of Akt kinases to dephosphorylation through ATP-dependent conformational plasticity

Proceedings of the National Academy of Sciences - PNAS, 2011-11, Vol.108 (46), p.E1120-E1127

2011

Volltextzugriff (PDF)

AKT/PKB AND OTHER D3 PHOSPHOINOSITIDE-REGULATED KINASES: Kinase Activation by Phosphoinositide-Dependent Phosphorylation

Annual review of biochemistry, 1999-01, Vol.68 (1), p.965-1014

1999

Volltextzugriff (PDF)

miR-146a targets Fos expression in human cardiac cells

Disease models & mechanisms, 2015-09, Vol.8 (9), p.1081-1091

2015

Volltextzugriff (PDF)

Akt kinase C-terminal modifications control activation loop dephosphorylation and enhance insulin response

Biochemical journal, 2015-10, Vol.471 (1), p.37-51

2015

Volltextzugriff (PDF)

A Truncated Fragment of Src Protein Kinase Generated by Calpain-mediated Cleavage Is a Mediator of Neuronal Death in Excitotoxicity

The Journal of biological chemistry, 2013-04, Vol.288 (14), p.9696-9709

2013

Volltextzugriff (PDF)

The interplay between NF-kappaB and E2F1 coordinately regulates inflammation and metabolism in human cardiac cells

PloS one, 2011-05, Vol.6 (5), p.e19724-e19724

2011

Volltextzugriff (PDF)

Akt activation by growth factors is a multiple-step process : the role of the PH domain

Oncogene, 1998-07, Vol.17 (3), p.313-325

1998

Volltextzugriff (PDF)

Induced overexpression of Na+/Ca2+ exchanger transgene: altered myocyte contractility, [Ca2+]i transients, SR Ca2+ contents, and action potential duration

American journal of physiology. Heart and circulatory physiology, 2009-08, Vol.297 (2), p.H590-H601

2009

Volltextzugriff (PDF)

Phosphatidylinositol 3-kinase is required for integrin-stimulated AKT and Raf-1/mitogen-activated protein kinase pathway activation

Molecular and Cellular Biology, 1997-08, Vol.17 (8), p.4406-4418

1997

Volltextzugriff (PDF)

Transduction of Interleukin-2 Antiapoptotic and Proliferative Signals via Akt Protein Kinase

Proceedings of the National Academy of Sciences - PNAS, 1997-04, Vol.94 (8), p.3627-3632

1997

Volltextzugriff (PDF)

The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinase

Cell, 1995-06, Vol.81 (5), p.727-736

1995

Volltextzugriff (PDF)

Cardioprotection of controlled and cardiac-specific over-expression of A(2A)-adenosine receptor in the pressure overload

PloS one, 2012, Vol.7 (7), p.e39919-e39919

2012

Volltextzugriff (PDF)

Autoregulation of kinase dephosphorylation by ATP binding in AGC protein kinases

Cell cycle (Georgetown, Tex.), 2012-02, Vol.11 (3), p.475-478

2012

Volltextzugriff (PDF)

The p65 subunit of NF-κB binds to PGC-1α, linking inflammation and metabolic disturbances in cardiac cells

Cardiovascular research, 2010-08, Vol.87 (3), p.449-458

2010

Volltextzugriff (PDF)

A tripartite cooperative mechanism confers resistance of the protein kinase A catalytic subunit to dephosphorylation

The Journal of biological chemistry, 2020-03, Vol.295 (10), p.3316-3329

2020

Volltextzugriff (PDF)

Targeting the epidermal growth factor receptor in cancer: apoptosis takes center stage

Cancer research (Chicago, Ill.), 2003-01, Vol.63 (1), p.1-5

2003

Volltextzugriff (PDF)

The Gfi-1 Protooncoprotein Represses Bax Expression and Inhibits T-Cell Death

Proceedings of the National Academy of Sciences - PNAS, 1996-12, Vol.93 (25), p.14569-14573

1996

Volltextzugriff (PDF)

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange

American journal of physiology. Heart and circulatory physiology, 2011-03, Vol.300 (3), p.H859-H868

2011

Volltextzugriff (PDF)

Myocardial injury after ischemia-reperfusion in mice deficient in Akt2 is associated with increased cardiac macrophage density

American journal of physiology. Heart and circulatory physiology, 2011-11, Vol.301 (5), p.H1932-H1940

2011

Volltextzugriff (PDF)

Tumor necrosis factor receptors 1 and 2 differentially regulate survival, cardiac dysfunction, and remodeling in transgenic mice with tumor necrosis factor-α-induced cardiomyopathy

Circulation (New York, N.Y.), 2004-04, Vol.109 (15), p.1892-1897

2004

Volltextzugriff (PDF)

Cardioprotection of Controlled and Cardiac-Specific Over-Expression of A2A-Adenosine Receptor in the Pressure Overload

PloS one, 2012-07, Vol.7 (7), p.e39919

2012

Volltextzugriff (PDF)

Constitutive overexpression of phosphomimetic phospholemman S68E mutant results in arrhythmias, early mortality, and heart failure: potential involvement of Na+/Ca2+ exchanger

American journal of physiology. Heart and circulatory physiology, 2012-02, Vol.302 (3), p.H770-H781

2012

Volltextzugriff (PDF)

Small GTPases and tyrosine kinases coregulate a molecular switch in the phosphoinositide 3-kinase regulatory subunit

Cancer cell, 2002-03, Vol.1 (2), p.181-191

2002

Volltextzugriff (PDF)

Induced overexpression of Na(+)/Ca(2+) exchanger does not aggravate myocardial dysfunction induced by transverse aortic constriction

Journal of cardiac failure, 2013-01, Vol.19 (1), p.60-70

2013

Volltextzugriff (PDF)

1 – 25 von 170

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(165)
Mit Volltextzugriff
(160)

Publikationsform

Sprache

Englisch
(170)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Resistance of Akt kinases to dephosphorylation through ATP-dependent conformational plasticity

AKT/PKB AND OTHER D3 PHOSPHOINOSITIDE-REGULATED KINASES: Kinase Activation by Phosphoinositide-Dependent Phosphorylation

miR-146a targets Fos expression in human cardiac cells

Akt kinase C-terminal modifications control activation loop dephosphorylation and enhance insulin response

A Truncated Fragment of Src Protein Kinase Generated by Calpain-mediated Cleavage Is a Mediator of Neuronal Death in Excitotoxicity

The interplay between NF-kappaB and E2F1 coordinately regulates inflammation and metabolism in human cardiac cells

Akt activation by growth factors is a multiple-step process : the role of the PH domain

Induced overexpression of Na+/Ca2+ exchanger transgene: altered myocyte contractility, [Ca2+]i transients, SR Ca2+ contents, and action potential duration

Phosphatidylinositol 3-kinase is required for integrin-stimulated AKT and Raf-1/mitogen-activated protein kinase pathway activation

Transduction of Interleukin-2 Antiapoptotic and Proliferative Signals via Akt Protein Kinase

The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinase

Cardioprotection of controlled and cardiac-specific over-expression of A(2A)-adenosine receptor in the pressure overload

Autoregulation of kinase dephosphorylation by ATP binding in AGC protein kinases

Left Ventricular Dysfunction in Murine Models of Heart Failure and in Failing Human Heart is Associated With a Selective Decrease in the Expression of Caveolin-3

The p65 subunit of NF-κB binds to PGC-1α, linking inflammation and metabolic disturbances in cardiac cells

A tripartite cooperative mechanism confers resistance of the protein kinase A catalytic subunit to dephosphorylation

Targeting the epidermal growth factor receptor in cancer: apoptosis takes center stage

The Gfi-1 Protooncoprotein Represses Bax Expression and Inhibits T-Cell Death

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange

Myocardial injury after ischemia-reperfusion in mice deficient in Akt2 is associated with increased cardiac macrophage density

Tumor necrosis factor receptors 1 and 2 differentially regulate survival, cardiac dysfunction, and remodeling in transgenic mice with tumor necrosis factor-α-induced cardiomyopathy

Cardioprotection of Controlled and Cardiac-Specific Over-Expression of A2A-Adenosine Receptor in the Pressure Overload

Constitutive overexpression of phosphomimetic phospholemman S68E mutant results in arrhythmias, early mortality, and heart failure: potential involvement of Na+/Ca2+ exchanger

Small GTPases and tyrosine kinases coregulate a molecular switch in the phosphoinositide 3-kinase regulatory subunit

Induced overexpression of Na(+)/Ca(2+) exchanger does not aggravate myocardial dysfunction induced by transverse aortic constriction

Suchergebnisse filtern